Aspartate aminotransferase Rv3722c governs aspartate-dependent nitrogen metabolism in Mycobacterium tuberculosis.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
23 04 2020
Historique:
received: 12 09 2019
accepted: 31 03 2020
entrez: 25 4 2020
pubmed: 25 4 2020
medline: 1 8 2020
Statut: epublish

Résumé

Gene rv3722c of Mycobacterium tuberculosis is essential for in vitro growth, and encodes a putative pyridoxal phosphate-binding protein of unknown function. Here we use metabolomic, genetic and structural approaches to show that Rv3722c is the primary aspartate aminotransferase of M. tuberculosis, and mediates an essential but underrecognized role in metabolism: nitrogen distribution. Rv3722c deficiency leads to virulence attenuation in macrophages and mice. Our results identify aspartate biosynthesis and nitrogen distribution as potential species-selective drug targets in M. tuberculosis.

Identifiants

pubmed: 32327655
doi: 10.1038/s41467-020-15876-8
pii: 10.1038/s41467-020-15876-8
pmc: PMC7181641
doi:

Substances chimiques

Bacterial Proteins 0
Aspartic Acid 30KYC7MIAI
Aspartate Aminotransferases EC 2.6.1.1
Nitrogen N762921K75

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1960

Subventions

Organisme : NIAID NIH HHS
ID : U19 AI107774
Pays : United States

Références

Ellens, K. W. et al. Confronting the catalytic dark matter encoded by sequenced genomes. Nucleic Acids Res. 45, 11495–11514 (2017).
pubmed: 29059321 pmcid: 5714238 doi: 10.1093/nar/gkx937
Chen, L. & Vitkup, D. Distribution of orphan metabolic activities. Trends Biotechnol. 25, 343–348 (2007).
pubmed: 17580095 doi: 10.1016/j.tibtech.2007.06.001
WHO. Global tuberculosis report 2018. 1–231. https://reliefweb.int/report/world/global-tuberculosis-report-2018 (2018).
Griffin, J. E. et al. High-resolution phenotypic profiling defines genes essential for mycobacterial growth and cholesterol catabolism. PLoS Pathog. 7, e1002251 (2011).
pubmed: 21980284 pmcid: 3182942 doi: 10.1371/journal.ppat.1002251
Grishin, N. V., Phillips, M. A. & Goldsmith, E. J. Modeling of the spatial structure of eukaryotic ornithine decarboxylases. Protein Sci. Publ. Protein Soc. 4, 1291–1304 (1995).
doi: 10.1002/pro.5560040705
Bramucci, E., Milano, T. & Pascarella, S. Genomic distribution and heterogeneity of MocR-like transcriptional factors containing a domain belonging to the superfamily of the pyridoxal-5’-phosphate dependent enzymes of fold type I. Biochem. Biophys. Res. Commun. 415, 88–93 (2011).
pubmed: 22020104 doi: 10.1016/j.bbrc.2011.10.017
El-Gebali, S. et al. The Pfam protein families database in 2019. Nucleic Acids Res. 47, D427–D432 (2019).
pubmed: 30357350 doi: 10.1093/nar/gky995
Mitchell, A. L. et al. InterPro in 2019: improving coverage, classification and access to protein sequence annotations. Nucleic Acids Res. 47, D351–D360 (2019).
pubmed: 30398656 doi: 10.1093/nar/gky1100
Huerta-Cepas, J. et al. eggNOG 4.5: a hierarchical orthology framework with improved functional annotations for eukaryotic, prokaryotic and viral sequences. Nucleic Acids Res. 44, D286–D293 (2016).
pubmed: 26582926 doi: 10.1093/nar/gkv1248
Yang, Z., Zeng, X. & Tsui, S. K.-W. Investigating function roles of hypothetical proteins encoded by the Mycobacterium tuberculosis H37Rv genome. BMC Genomics 20, 394 (2019).
pubmed: 31113361 pmcid: 6528289 doi: 10.1186/s12864-019-5746-6
Ortega, C. et al. Systematic survey of serine hydrolase activity in Mycobacterium tuberculosis defines changes associated with persistence. Cell Chem. Biol. 23, 290–298 (2016).
pubmed: 26853625 pmcid: 4803444 doi: 10.1016/j.chembiol.2016.01.003
Penn, B. H. et al. An Mtb-human protein-protein interaction map identifies a switch between host antiviral and antibacterial responses. Mol. Cell 71, 637–648.e5 (2018).
pubmed: 30118682 pmcid: 6329589 doi: 10.1016/j.molcel.2018.07.010
Wang, M., Herrmann, C. J., Simonovic, M., Szklarczyk, D. & von Mering, C. Version 4.0 of PaxDb: protein abundance data, integrated across model organisms, tissues, and cell-lines. Proteomics 15, 3163–3168 (2015).
pubmed: 25656970 pmcid: 6680238 doi: 10.1002/pmic.201400441
Ehrt, S., Schnappinger, D. & Rhee, K. Y. Metabolic principles of persistence and pathogenicity in Mycobacterium tuberculosis. Nat. Rev. Microbiol. 16, 496–507 (2018).
pubmed: 29691481 pmcid: 6045436 doi: 10.1038/s41579-018-0013-4
Gouzy, A. et al. Mycobacterium tuberculosis nitrogen assimilation and host colonization require aspartate. Nat. Chem. Biol. 9, 674–676 (2013).
pubmed: 24077180 doi: 10.1038/nchembio.1355
Gouzy, A., Poquet, Y. & Neyrolles, O. Nitrogen metabolism in Mycobacterium tuberculosis physiology and virulence. Nat. Rev. Microbiol. 12, 729–737 (2014).
pubmed: 25244084 doi: 10.1038/nrmicro3349
Johnson, E. O. et al. Large-scale chemical-genetics yields new M. tuberculosis inhibitor classes. Nature 571, 72–78 (2019).
pubmed: 31217586 doi: 10.1038/s41586-019-1315-z
de Carvalho, L. P. S. et al. Activity-based metabolomic profiling of enzymatic function: identification of Rv1248c as a mycobacterial 2-hydroxy-3-oxoadipate synthase. Chem. Biol. 17, 323–332 (2010).
pubmed: 20416504 pmcid: 2878197 doi: 10.1016/j.chembiol.2010.03.009
Shen, H. et al. The human knockout gene CLYBL connects itaconate to vitamin B12. Cell 171, 771–782.e11 (2017).
pubmed: 29056341 pmcid: 5827971 doi: 10.1016/j.cell.2017.09.051
Cooper, A. J. L. & Kuhara, T. α-Ketoglutaramate: an overlooked metabolite of glutamine and a biomarker for hepatic encephalopathy and inborn errors of the urea cycle. Metab. Brain Dis. 29, 991–1006 (2014).
pubmed: 24234505 doi: 10.1007/s11011-013-9444-9
Nobe, Y. et al. The novel substrate recognition mechanism utilized by aspartate aminotransferase of the extreme thermophile Thermus thermophilus HB8. J. Biol. Chem. 273, 29554–29564 (1998).
pubmed: 9792664 doi: 10.1074/jbc.273.45.29554
Yagi, T., Kagamiyama, H., Nozaki, M. & Soda, K. [17] Glutamate-aspartate transaminase from microorganisms. in Methods in Enzymology, Vol. 113 83–89 (ed. Meister, A.) (Academic Press, 1985).
Han, Q., Fang, J. & Li, J. Kynurenine aminotransferase and glutamine transaminase K of Escherichia coli: identity with aspartate aminotransferase. Biochem. J. 360, 617–623 (2001).
pubmed: 11736651 pmcid: 1222264 doi: 10.1042/bj3600617
Sigrist, C. J. A. et al. New and continuing developments at PROSITE. Nucleic Acids Res. 41, D344–D347 (2013).
pubmed: 23161676 doi: 10.1093/nar/gks1067
Son, H. F. & Kim, K.-J. Structural insights into a novel class of aspartate aminotransferase from Corynebacterium glutamicum. PloS ONE 11, e0158402 (2016).
pubmed: 27355211 pmcid: 4927141 doi: 10.1371/journal.pone.0158402
Dolzan, M. et al. Crystal structure and reactivity of YbdL from Escherichia coli identify a methionine aminotransferase function. FEBS Lett. 571, 141–146 (2004).
pubmed: 15280032 doi: 10.1016/j.febslet.2004.06.075
Mehta, P. K., Hale, T. I. & Christen, P. Aminotransferases: demonstration of homology and division into evolutionary subgroups. Eur. J. Biochem. 214, 549–561 (1993).
pubmed: 8513804 doi: 10.1111/j.1432-1033.1993.tb17953.x
Malashkevich, V. N., Onuffer, J. J., Kirsch, J. F. & Jansonius, J. N. Alternating arginine-modulated substrate specificity in an engineered tyrosine aminotransferase. Nat. Struct. Biol. 2, 548–553 (1995).
pubmed: 7664122 doi: 10.1038/nsb0795-548
Wrenger, C. et al. Specific inhibition of the aspartate aminotransferase of Plasmodium falciparum. J. Mol. Biol. 405, 956–971 (2011).
pubmed: 21087616 doi: 10.1016/j.jmb.2010.11.018
Guidetti, P., Amori, L., Sapko, M. T., Okuno, E. & Schwarcz, R. Mitochondrial aspartate aminotransferase: a third kynurenate-producing enzyme in the mammalian brain. J. Neurochem. 102, 103–111 (2007).
pubmed: 17442055 doi: 10.1111/j.1471-4159.2007.04556.x
Duff, S. M. G. et al. The enzymology of alanine aminotransferase (AlaAT) isoforms from Hordeum vulgare and other organisms, and the HvAlaAT crystal structure. Arch. Biochem. Biophys. 528, 90–101 (2012).
pubmed: 22750542 doi: 10.1016/j.abb.2012.06.006
Carroll, P., Pashley, C. A. & Parish, T. Functional analysis of GlnE, an essential adenylyl transferase in Mycobacterium tuberculosis. J. Bacteriol. 190, 4894–4902 (2008).
pubmed: 18469098 pmcid: 2446997 doi: 10.1128/JB.00166-08
Yuan, J. et al. Metabolomics-driven quantitative analysis of ammonia assimilation in E. coli. Mol. Syst. Biol. 5, 302 (2009).
pubmed: 19690571 pmcid: 2736657 doi: 10.1038/msb.2009.60
Tullius, M. V., Harth, G. & Horwitz, M. A. Glutamine synthetase GlnA1 is essential for growth of Mycobacterium tuberculosis in human THP-1 macrophages and guinea pigs. Infect. Immun. 71, 3927–3936 (2003).
pubmed: 12819079 pmcid: 162033 doi: 10.1128/IAI.71.7.3927-3936.2003
Catazaro, J., Caprez, A., Guru, A., Swanson, D. & Powers, R. Functional evolution of PLP-dependent enzymes based on active-site structural similarities. Proteins Struct. Funct. Bioinforma. 82, 2597–2608 (2014).
doi: 10.1002/prot.24624
Onuffer, J. J. & Kirsch, J. F. Redesign of the substrate specificity of Escherichia coli aspartate aminotransferase to that of escherichia coli tyrosine aminotransferase by homology modeling and site-directed mutagenesis. Protein Sci. 4, 1750–1757 (1995).
pubmed: 8528073 pmcid: 2143225 doi: 10.1002/pro.5560040910
Cole, S. T. et al. Deciphering the biology of Mycobacterium tuberculosis from the complete genome sequence. Nature 393, 537–544 (1998).
pubmed: 9634230 doi: 10.1038/31159
Amorim Franco, T. M., Hegde, S. & Blanchard, J. S. Chemical mechanism of the branched-chain aminotransferase IlvE from Mycobacterium tuberculosis. Biochemistry 55, 6295–6303 (2016).
pubmed: 27780341 pmcid: 5443349 doi: 10.1021/acs.biochem.6b00928
Bhor, V. M., Dev, S., Vasanthakumar, G. R. & Surolia, A. Spectral and kinetic characterization of 7,8-diaminopelargonic acid synthase from Mycobacterium tuberculosis. IUBMB Life 58, 225–233 (2006).
pubmed: 16754301 doi: 10.1080/15216540600746997
Nasir, N., Anant, A., Vyas, R. & Biswal, B. K. Crystal structures of Mycobacterium tuberculosis HspAT and ArAT reveal structural basis of their distinct substrate specificities. Sci. Rep. 6, 18880 (2016).
pubmed: 26738801 pmcid: 4703992 doi: 10.1038/srep18880
Holt, M. C. et al. Biochemical characterization and structure-based mutational analysis provide insight into the binding and mechanism of action of novel aspartate aminotransferase inhibitors. Biochemistry 57, 6604–6614 (2018).
pubmed: 30365304 pmcid: 6487875 doi: 10.1021/acs.biochem.8b00914
Gouzy, A. et al. Mycobacterium tuberculosis exploits asparagine to assimilate nitrogen and resist acid stress during infection. PLoS Pathog. 10, e1003928 (2014).
pubmed: 24586151 pmcid: 3930563 doi: 10.1371/journal.ppat.1003928
Agapova, A. et al. Flexible nitrogen utilisation by the metabolic generalist pathogen Mycobacterium tuberculosis. eLife 8, e41129 (2019).
Lal, P. B., Schneider, B. L., Vu, K. & Reitzer, L. The redundant aminotransferases in lysine and arginine synthesis and the extent of aminotransferase redundancy in Escherichia coli. Mol. Microbiol. 94, 843–856 (2014).
pubmed: 25243376 doi: 10.1111/mmi.12801
Gelfand, D. H. & Steinberg, R. A. Escherichia coli mutants deficient in the aspartate and aromatic amino acid aminotransferases. J. Bacteriol. 130, 429–440 (1977).
pubmed: 15983 pmcid: 235221 doi: 10.1128/JB.130.1.429-440.1977
Bennett, B. D. et al. Absolute metabolite concentrations and implied enzyme active site occupancy in Escherichia coli. Nat. Chem. Biol. 5, 593–599 (2009).
pubmed: 19561621 pmcid: 2754216 doi: 10.1038/nchembio.186
Soga, T. et al. Quantitative metabolome analysis using capillary electrophoresis mass spectrometry. J. Proteome Res. 2, 488–494 (2003).
pubmed: 14582645 doi: 10.1021/pr034020m
Sugimoto, M. et al. MMMDB: mouse multiple tissue metabolome database. Nucleic Acids Res. 40, D809–D814 (2012).
pubmed: 22139941 doi: 10.1093/nar/gkr1170
Reitzer, L. Nitrogen assimilation and global regulation in Escherichia coli. Annu. Rev. Microbiol. 57, 155–176 (2003).
pubmed: 12730324 doi: 10.1146/annurev.micro.57.030502.090820
Reitzer, L. Biosynthesis of glutamate, aspartate, asparagine, L-alanine, and D-alanine. EcoSal Plus 1, 1–18 (2004).
doi: 10.1128/ecosalplus.3.6.1.3
Somashekar, B. S. et al. Metabolic profiling of lung granuloma in Mycobacterium tuberculosis infected guinea pigs: ex vivo 1H magic angle spinning NMR studies. J. Proteome Res. 10, 4186–4195 (2011).
pubmed: 21732701 doi: 10.1021/pr2003352
Borah, K. et al. Intracellular Mycobacterium tuberculosis exploits multiple host nitrogen sources during growth in human macrophages. Cell Rep. 29, 3580–3591.e4 (2019).
pubmed: 31825837 pmcid: 6915324 doi: 10.1016/j.celrep.2019.11.037
Bhattacharyya, N. et al. An aspartate-specific solute-binding protein regulates protein kinase G activity to control glutamate metabolism in Mycobacteria. mBio 9, e00931–18 (2018).
pubmed: 30065086 pmcid: 6069109
Rieck, B. et al. PknG senses amino acid availability to control metabolism and virulence of Mycobacterium tuberculosis. PLoS Pathog. 13, e1006399 (2017).
pubmed: 28545104 pmcid: 5448819 doi: 10.1371/journal.ppat.1006399
Murphy, K. C., Papavinasasundaram, K. & Sassetti, C. M. Mycobacterial recombineering. Methods Mol. Biol. 1285, 177–199 (2015).
pubmed: 25779316 doi: 10.1007/978-1-4939-2450-9_10
Tautenhahn, R., Patti, G. J., Rinehart, D. & Siuzdak, G. XCMS Online: a web-based platform to process untargeted metabolomic data. Anal. Chem. 84, 5035–5039 (2012).
pubmed: 22533540 pmcid: 3703953 doi: 10.1021/ac300698c
Kessner, D., Chambers, M., Burke, R., Agus, D. & Mallick, P. ProteoWizard: open source software for rapid proteomics tools development. Bioinforma. Oxf. Engl. 24, 2534–2536 (2008).
doi: 10.1093/bioinformatics/btn323
Pashley, C. A., Brown, A. C., Robertson, D. & Parish, T. Identification of the Mycobacterium tuberculosis GlnE promoter and its response to nitrogen availability. Microbiology 152, 2727–2734 (2006).
pubmed: 16946267 doi: 10.1099/mic.0.28942-0
Weischenfeldt, J. & Porse, B. Bone marrow-derived macrophages (BMM): isolation and applications. Cold Spring Harb. Protoc. 2008, pdb.prot5080 (2008).
Rial, D. V. & Ceccarelli, E. A. Removal of DnaK contamination during fusion protein purifications. Protein Expr. Purif. 25, 503–507 (2002).
pubmed: 12182832 doi: 10.1016/S1046-5928(02)00024-4
Jaisson, S., Veiga-da-Cunha, M. & Van Schaftingen, E. Molecular identification of omega-amidase, the enzyme that is functionally coupled with glutamine transaminases, as the putative tumor suppressor Nit2. Biochimie 91, 1066–1071 (2009).
pubmed: 19596042 doi: 10.1016/j.biochi.2009.07.002
Winn, M. D. et al. Overview of the CCP4 suite and current developments. Acta Crystallogr. D. Biol. Crystallogr. 67, 235–242 (2011).
pubmed: 21460441 pmcid: 21460441 doi: 10.1107/S0907444910045749
Vagin, A. & Teplyakov, A. Molecular replacement with MOLREP. Acta Crystallogr. D. Biol. Crystallogr. 66, 22–25 (2010).
pubmed: 20057045 doi: 10.1107/S0907444909042589
Kabsch, W. XDS. Acta Crystallogr. D. Biol. Crystallogr. 66, 125–132 (2010).
pubmed: 20124692 pmcid: 20124692 doi: 10.1107/S0907444909047337
Adams, P. D. et al. PHENIX: building new software for automated crystallographic structure determination. Acta Crystallogr. D. Biol. Crystallogr. 58, 1948–1954 (2002).
pubmed: 12393927 doi: 10.1107/S0907444902016657
Emsley, P., Lohkamp, B., Scott, W. G. & Cowtan, K. Features and development of Coot. Acta Crystallogr. D. Biol. Crystallogr. 66, 486–501 (2010).
pubmed: 20383002 pmcid: 20383002 doi: 10.1107/S0907444910007493
Liebschner, D. et al. Polder maps: improving OMIT maps by excluding bulk solvent. Acta Crystallogr. Sect. Struct. Biol. 73, 148–157 (2017).
doi: 10.1107/S2059798316018210
Pettersen, E. F. et al. UCSF Chimera-a visualization system for exploratory research and analysis. J. Comput. Chem. 25, 1605–1612 (2004).
pubmed: 15264254 doi: 10.1002/jcc.20084
Mendler, K. et al. AnnoTree: visualization and exploration of a functionally annotated microbial tree of life. Nucleic Acids Res. 47, 4442–4448 (2019).
pubmed: 31081040 pmcid: 6511854 doi: 10.1093/nar/gkz246
Letunic, I. & Bork, P. Interactive Tree Of Life (iTOL) v4: recent updates and new developments. Nucleic Acids Res. 47, W256–W259 (2019).
pubmed: 6602468 pmcid: 6602468 doi: 10.1093/nar/gkz239

Auteurs

Robert S Jansen (RS)

Division of Infectious Diseases, Department of Medicine, Weill Cornell Medical College, New York, NY, 10065, USA.

Lungelo Mandyoli (L)

Department of Biochemistry and Biophysics, Texas A&M University, College Station, TX, 77843-2128, USA.

Ryan Hughes (R)

Department of Biochemistry and Biophysics, Texas A&M University, College Station, TX, 77843-2128, USA.

Shoko Wakabayashi (S)

Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA, 02115, USA.

Jessica T Pinkham (JT)

Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA, 02115, USA.

Bruna Selbach (B)

Division of Infectious Diseases, Department of Medicine, Weill Cornell Medical College, New York, NY, 10065, USA.

Kristine M Guinn (KM)

Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA, 02115, USA.

Eric J Rubin (EJ)

Department of Immunology and Infectious Diseases, Harvard T. H. Chan School of Public Health, Boston, MA, 02115, USA.

James C Sacchettini (JC)

Department of Biochemistry and Biophysics, Texas A&M University, College Station, TX, 77843-2128, USA. sacchett@tamu.edu.

Kyu Y Rhee (KY)

Division of Infectious Diseases, Department of Medicine, Weill Cornell Medical College, New York, NY, 10065, USA. kyr9001@med.cornell.edu.
Department of Microbiology & Immunology, Weill Cornell Medical College, New York, NY, 10065, USA. kyr9001@med.cornell.edu.

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