An elevated CA 19-9 is associated with invasive cancer and worse survival in IPMN.


Journal

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
ISSN: 1424-3911
Titre abrégé: Pancreatology
Pays: Switzerland
ID NLM: 100966936

Informations de publication

Date de publication:
Jun 2020
Historique:
received: 16 01 2020
revised: 27 03 2020
accepted: 06 04 2020
pubmed: 26 4 2020
medline: 10 4 2021
entrez: 26 4 2020
Statut: ppublish

Résumé

Current guidelines for IPMN include an elevated serum carbohydrate antigen (CA) 19-9 among the worrisome features. However, the correlation of CA 19-9 with histological malignant features and survival is unclear. Serum CEA is also currently used for preoperative management of IPMN, although its measurement is not evidence-based. Accordingly, we aimed to assess the role of these tumor markers as predictors of malignancy in IPMN. IPMN resected between 1998 and 2018 at Massachusetts General Hospital were analyzed. Clinical, pathological and survival data were collected and compared to preoperative levels of CA 19-9 and CEA. Receiver operating characteristic (ROC) and Cox regression analyses were performed considering cut-offs of 37 U/ml (CA 19-9) and 5 μg/l (CEA). Analysis of 594 patients showed that preoperative CA 19-9 levels > 37 U/ml (n = 128) were associated with an increased likelihood of invasive carcinoma when compared to normal levels (45.3% vs. 18.0%, P < 0.001), while there was no difference with respect to high-grade dysplasia (32.9% vs 31.9%, P = 0.88). The proportion of concurrent pancreatic cancer was higher in patients with CA 19-9 > 37 U/ml (17.2% vs 4.9%, P < 0.001). An elevated CA 19-9 was also associated with worse overall and disease-free survival (HR = 1.943, P = 0.007 and HR = 2.484, P < 0.001 respectively). CEA levels did not correlate with malignancy. In patients with IPMN, serum CA19-9 > 37 U/ml is associated with invasive IPMN and concurrent pancreatic cancer as well as worse survival, but not with high-grade dysplasia. Serum CEA appears to have minimal utility in the management of these patients.

Sections du résumé

BACKGROUND BACKGROUND
Current guidelines for IPMN include an elevated serum carbohydrate antigen (CA) 19-9 among the worrisome features. However, the correlation of CA 19-9 with histological malignant features and survival is unclear. Serum CEA is also currently used for preoperative management of IPMN, although its measurement is not evidence-based. Accordingly, we aimed to assess the role of these tumor markers as predictors of malignancy in IPMN.
METHODS METHODS
IPMN resected between 1998 and 2018 at Massachusetts General Hospital were analyzed. Clinical, pathological and survival data were collected and compared to preoperative levels of CA 19-9 and CEA. Receiver operating characteristic (ROC) and Cox regression analyses were performed considering cut-offs of 37 U/ml (CA 19-9) and 5 μg/l (CEA).
RESULTS RESULTS
Analysis of 594 patients showed that preoperative CA 19-9 levels > 37 U/ml (n = 128) were associated with an increased likelihood of invasive carcinoma when compared to normal levels (45.3% vs. 18.0%, P < 0.001), while there was no difference with respect to high-grade dysplasia (32.9% vs 31.9%, P = 0.88). The proportion of concurrent pancreatic cancer was higher in patients with CA 19-9 > 37 U/ml (17.2% vs 4.9%, P < 0.001). An elevated CA 19-9 was also associated with worse overall and disease-free survival (HR = 1.943, P = 0.007 and HR = 2.484, P < 0.001 respectively). CEA levels did not correlate with malignancy.
CONCLUSION CONCLUSIONS
In patients with IPMN, serum CA19-9 > 37 U/ml is associated with invasive IPMN and concurrent pancreatic cancer as well as worse survival, but not with high-grade dysplasia. Serum CEA appears to have minimal utility in the management of these patients.

Identifiants

pubmed: 32332003
pii: S1424-3903(20)30131-9
doi: 10.1016/j.pan.2020.04.002
pii:
doi:

Substances chimiques

Biomarkers, Tumor 0
CA-19-9 Antigen 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

729-735

Informations de copyright

Copyright © 2020 IAP and EPC. Published by Elsevier B.V. All rights reserved.

Auteurs

D Ciprani (D)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

V Morales-Oyarvide (V)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

M Qadan (M)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

T Hank (T)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

M Weniger (M)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

J M Harrison (JM)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

C Rodrigues (C)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

N K Horick (NK)

Biostatistics Center, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

M Mino-Kenudson (M)

Department of Pathology, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

C R Ferrone (CR)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

A L Warshaw (AL)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

K D Lillemoe (KD)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

C Fernández-Del Castillo (C)

Department of Surgery, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA. Electronic address: CFernandez@mgh.harvard.edu.

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Classifications MeSH