Acid-Base Balance Disorders During Kidney Preservation in Cold Ischemia.


Journal

Transplantation proceedings
ISSN: 1873-2623
Titre abrégé: Transplant Proc
Pays: United States
ID NLM: 0243532

Informations de publication

Date de publication:
Sep 2020
Historique:
received: 08 12 2019
revised: 06 01 2020
accepted: 26 01 2020
pubmed: 27 4 2020
medline: 16 12 2020
entrez: 27 4 2020
Statut: ppublish

Résumé

Acid-base balance disorders are a crucial element of ischemia-reperfusion injury during organ transplantation. Hypoxia during organ procurement and storage cause cellular homeostasis imbalance with impact on further graft function. Acidosis in preserved kidney caused by lactate accumulation may have an important role as a common denominator of various pathways leading to cellular damage. Our trial sought to answer questions regarding a range of pH alterations in the kidney before the transplantation, their potential cause, and how this may affect further outcome of the kidney transplantation procedure. Perfusion fluid for pH analysis was obtained from perfusion pump (PP) or through kidney flushing at the end of preservation depending on the storage method. A total of 66 sample results were collated with the data from the transplant registry, hospitalization, and outpatient department. Statistical analysis was conducted linking pH results with factors related to donor, recipient, preservation, and outcome according to designed schematics. Mean perfusate pH was significantly lower in simple hypothermia (SH) vs the PP storage group (6.77 vs 7.11; P < .001). All samples of perfusate pH in the SH group were below physiological values (<7.35), and in 10% of samples in the SH group, pH >7.00. We concluded that kidney storage in cold ischemia is associated with organ acidosis independent of preservation method and that SH is correlated with significantly bigger acidosis than storage in PP, which is an important procedure removing an excessive amount of hydrogen ions from kidney microcirculation, decreasing cell damage.

Sections du résumé

BACKGROUND BACKGROUND
Acid-base balance disorders are a crucial element of ischemia-reperfusion injury during organ transplantation. Hypoxia during organ procurement and storage cause cellular homeostasis imbalance with impact on further graft function. Acidosis in preserved kidney caused by lactate accumulation may have an important role as a common denominator of various pathways leading to cellular damage.
METHODS METHODS
Our trial sought to answer questions regarding a range of pH alterations in the kidney before the transplantation, their potential cause, and how this may affect further outcome of the kidney transplantation procedure. Perfusion fluid for pH analysis was obtained from perfusion pump (PP) or through kidney flushing at the end of preservation depending on the storage method.
RESULTS RESULTS
A total of 66 sample results were collated with the data from the transplant registry, hospitalization, and outpatient department. Statistical analysis was conducted linking pH results with factors related to donor, recipient, preservation, and outcome according to designed schematics. Mean perfusate pH was significantly lower in simple hypothermia (SH) vs the PP storage group (6.77 vs 7.11; P < .001). All samples of perfusate pH in the SH group were below physiological values (<7.35), and in 10% of samples in the SH group, pH >7.00.
CONCLUSIONS CONCLUSIONS
We concluded that kidney storage in cold ischemia is associated with organ acidosis independent of preservation method and that SH is correlated with significantly bigger acidosis than storage in PP, which is an important procedure removing an excessive amount of hydrogen ions from kidney microcirculation, decreasing cell damage.

Identifiants

pubmed: 32334797
pii: S0041-1345(19)31674-4
doi: 10.1016/j.transproceed.2020.01.099
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2036-2042

Informations de copyright

Copyright © 2020 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Karol Tejchman (K)

Department of General and Transplantation Surgery, Pomeranian Medical University, Szczecin, Poland. Electronic address: ktej78@pum.edu.pl.

Anita Sierocka (A)

Department of General Mini-invasive and Gastroenterological Surgery, Pomeranian Medical University, Szczecin, Poland.

Maciej Kotowski (M)

Department of General and Transplantation Surgery, Pomeranian Medical University, Szczecin, Poland.

Labib Zair (L)

Department of General and Transplantation Surgery, Pomeranian Medical University, Szczecin, Poland.

Ewa Pilichowska (E)

Department of General and Transplantation Surgery, Pomeranian Medical University, Szczecin, Poland.

Marek Ostrowski (M)

Department of General and Transplantation Surgery, Pomeranian Medical University, Szczecin, Poland.

Jerzy Sieńko (J)

Department of General and Transplantation Surgery, Pomeranian Medical University, Szczecin, Poland.

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