TAOK1 positively regulates TLR4-induced inflammatory responses by promoting ERK1/2 activation in macrophages.

ERK1/2 Inflammation TAOK1 TLR TPL2 TRAF6

Journal

Molecular immunology
ISSN: 1872-9142
Titre abrégé: Mol Immunol
Pays: England
ID NLM: 7905289

Informations de publication

Date de publication:
25 Apr 2020
Historique:
received: 17 12 2019
revised: 08 04 2020
accepted: 10 04 2020
pubmed: 29 4 2020
medline: 29 4 2020
entrez: 29 4 2020
Statut: aheadofprint

Résumé

Thousand and one amino acid kinase 1 (TAOK1) is a member of Ste20-like kinases, but its function in regulating inflammatory responses remains largely unknown. In this study, we identify TAOK1 as a positive regulator of TLR4-triggered inflammatory responses in macrophages. TAOK1 increases LPS-induced production of pro-inflammatory cytokine such as IL-6, TNF-α and IL12p40 in macrophages. TAOK1 deficient mice showed decreased susceptibility to endotoxin shock, with less pro-inflammatory cytokine production than control mice. TAOK1 promotes LPS-induced activation of ERK1/2 by constitutively interacting with TRAF6 and TPL2. These finding unravel the important role of TAOK1 as a positive regulator of TLR4-induced inflammatory responses.

Identifiants

pubmed: 32344244
pii: S0161-5890(19)30890-9
doi: 10.1016/j.molimm.2020.04.006
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

124-131

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Auteurs

Lingxi Zhu (L)

Clinical Cancer Institute, Translational Medicine Center, Second Military Medical University, Shanghai 200433, China.

Qingzhuo Yu (Q)

Clinical Cancer Institute, Translational Medicine Center, Second Military Medical University, Shanghai 200433, China.

Peng Gao (P)

Clinical Cancer Institute, Translational Medicine Center, Second Military Medical University, Shanghai 200433, China.

Qianru Liu (Q)

Department of Cell Biology, School of Basic Medical Sciences, Zhejiang University, Hangzhou 310058, China.

Xiaogang Luo (X)

Department of Cell Biology, School of Basic Medical Sciences, Zhejiang University, Hangzhou 310058, China.

Guixian Jiang (G)

Clinical Cancer Institute, Translational Medicine Center, Second Military Medical University, Shanghai 200433, China.

Ruihua Ji (R)

Department of Anesthesiology, Changzheng Hospital, Second Military Medical University, Shanghai 200433, China.

Rui Yang (R)

Department of Anesthesiology, Changzheng Hospital, Second Military Medical University, Shanghai 200433, China.

Xianwei Ma (X)

Scientific Research Center, Shanghai Public Health Clinical Center, Fudan University, Shanghai, 201508, China.

Jing Xu (J)

Clinical Cancer Institute, Translational Medicine Center, Second Military Medical University, Shanghai 200433, China.

Hongbin Yuan (H)

Department of Anesthesiology, Changzheng Hospital, Second Military Medical University, Shanghai 200433, China.

Jun Zhou (J)

Department of Cell Biology, School of Basic Medical Sciences, Zhejiang University, Hangzhou 310058, China. Electronic address: zhjjwm300@zju.edu.cn.

Huazhang An (H)

Clinical Cancer Institute, Translational Medicine Center, Second Military Medical University, Shanghai 200433, China. Electronic address: anhz@immunol.org.

Classifications MeSH