Analytical and clinical validation of rapid chemiluminescence enzyme immunoassay for urinary thioredoxin, an oxidative stress-dependent early biomarker of acute kidney injury.


Journal

Clinica chimica acta; international journal of clinical chemistry
ISSN: 1873-3492
Titre abrégé: Clin Chim Acta
Pays: Netherlands
ID NLM: 1302422

Informations de publication

Date de publication:
Aug 2020
Historique:
received: 11 03 2020
revised: 22 04 2020
accepted: 22 04 2020
pubmed: 30 4 2020
medline: 14 1 2021
entrez: 30 4 2020
Statut: ppublish

Résumé

Oxidative stress is now recognized to be an important therapeutic target in kidney diseases. However, there are currently no biomarkers that can be used clinically to diagnose renal oxidative stress. A rapid assay system for urinary thioredoxin 1, an oxidative stress-dependent biomarker of acute kidney injury (AKI), was developed as a chemiluminescence enzyme immunoassay and validated analytically and clinically. Analytic evaluation revealed that hemolytic hemoglobin caused measurements to be abnormally high, above the detectable range. However, urine sediment containing red blood cells did not affect the measurements. Assays using our proposed chemiluminescence enzyme immunoassay were completed within as little as 6 min, whereas a conventional ELISA > 4 h. Aciduria <pH 6.0 led to a significant underestimation of thioredoxin 1 concentrations. However, the effects of aciduria were completely reversible with use of a buffer developed for pH adjustment. Urinary thioredoxin 1 was increased in patients with AKI, but was unaffected by extrarenal oxidative stress diseases, including hypoxemia and myocardial infarction, or by chronic kidney disease in which serum creatinine concentrations were comparable. These results suggest that the chemiluminescent enzyme immunoassay system for urinary thioredoxin 1 enables rapid and specific diagnosis of AKI associated with oxidative stress.

Sections du résumé

BACKGROUND BACKGROUND
Oxidative stress is now recognized to be an important therapeutic target in kidney diseases. However, there are currently no biomarkers that can be used clinically to diagnose renal oxidative stress.
METHODS METHODS
A rapid assay system for urinary thioredoxin 1, an oxidative stress-dependent biomarker of acute kidney injury (AKI), was developed as a chemiluminescence enzyme immunoassay and validated analytically and clinically.
RESULTS RESULTS
Analytic evaluation revealed that hemolytic hemoglobin caused measurements to be abnormally high, above the detectable range. However, urine sediment containing red blood cells did not affect the measurements. Assays using our proposed chemiluminescence enzyme immunoassay were completed within as little as 6 min, whereas a conventional ELISA > 4 h. Aciduria <pH 6.0 led to a significant underestimation of thioredoxin 1 concentrations. However, the effects of aciduria were completely reversible with use of a buffer developed for pH adjustment. Urinary thioredoxin 1 was increased in patients with AKI, but was unaffected by extrarenal oxidative stress diseases, including hypoxemia and myocardial infarction, or by chronic kidney disease in which serum creatinine concentrations were comparable.
CONCLUSIONS CONCLUSIONS
These results suggest that the chemiluminescent enzyme immunoassay system for urinary thioredoxin 1 enables rapid and specific diagnosis of AKI associated with oxidative stress.

Identifiants

pubmed: 32348784
pii: S0009-8981(20)30180-7
doi: 10.1016/j.cca.2020.04.025
pii:
doi:

Substances chimiques

Biomarkers 0
TXN protein, human 0
Thioredoxins 52500-60-4

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

271-279

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Seiji Yokoi (S)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Kenji Kasuno (K)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan; Life Science Innovation Center, University of Fukui, Fukui, Japan. Electronic address: kasuno@u-fukui.ac.jp.

Kazuhisa Nishimori (K)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Sho Nishikawa (S)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Yudai Nishikawa (Y)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Sayu Morita (S)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Mamiko Kobayashi (M)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Sachiko Fukushima (S)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Daisuke Mikami (D)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Naoki Takahashi (N)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Yumiko Oota (Y)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Hideki Kimura (H)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

Yoshihiro Soya (Y)

Diagnostic System Department, Toyobo Co., Ltd., Osaka, Japan.

Shinsuke Kimata (S)

Tsuruga Institute of Biotechnology, Toyobo Co., Ltd., Osaka, Japan.

Kengo Nishimura (K)

Tsuruga Institute of Biotechnology, Toyobo Co., Ltd., Osaka, Japan.

Takahiko Ono (T)

Department of Nephrology, Amagasaki Eijinkai Clinic, Hyogo, Japan.

Eri Muso (E)

Department of Nephrology and Dialysis, Kitano Hospital, Tazuke Kofukai Medical Research Institute, Osaka, Japan.

Haruyoshi Yoshida (H)

Department of Internal Medicine, Sugita Genpaku Memorial Obama Municipal Hospital, Fukui, Japan.

Junji Yodoi (J)

Department of Biological Responses, Institute for Virus Research, Graduate School of Medicine, Kyoto University, Kyoto, Japan.

Masayuki Iwano (M)

Department of Nephrology, Faculty of Medical Sciences, University of Fukui, Fukui, Japan.

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Classifications MeSH