Partial impairment of insulin receptor expression mimics fasting to prevent diet-induced fatty liver disease.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
29 04 2020
Historique:
received: 06 06 2018
accepted: 19 03 2020
entrez: 1 5 2020
pubmed: 1 5 2020
medline: 30 7 2020
Statut: epublish

Résumé

Excessive insulin signaling through the insulin receptor (IR) may play a role in the pathogenesis of diet-induced metabolic disease, including obesity and type 2 diabetes. Here we investigate whether heterozygous impairment of insulin receptor (IR) expression limited to peripheral, i.e. non-CNS, tissues of adult mice impacts the development of high-fat diet-induced metabolic deterioration. While exhibiting some features of insulin resistance, PerIRKO

Identifiants

pubmed: 32350271
doi: 10.1038/s41467-020-15623-z
pii: 10.1038/s41467-020-15623-z
pmc: PMC7190665
doi:

Substances chimiques

Receptor, Insulin EC 2.7.10.1
Glucose IY9XDZ35W2

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2080

Commentaires et corrections

Type : ErratumIn

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Auteurs

Troy L Merry (TL)

Energy Metabolism Laboratory, Institute of Translational Medicine, Department of Health Sciences and Technology, Swiss Federal Institute of Technology (ETH), Zürich, Switzerland. t.merry@auckland.ac.nz.
Discipline of Nutrition, School of Medical Sciences, The University of Auckland, Auckland, New Zealand. t.merry@auckland.ac.nz.
Maurice Wilkins Centre for Molecular Biodiscovery, The University of Auckland, Auckland, New Zealand. t.merry@auckland.ac.nz.

Chris P Hedges (CP)

Discipline of Nutrition, School of Medical Sciences, The University of Auckland, Auckland, New Zealand.
Maurice Wilkins Centre for Molecular Biodiscovery, The University of Auckland, Auckland, New Zealand.

Stewart W Masson (SW)

Discipline of Nutrition, School of Medical Sciences, The University of Auckland, Auckland, New Zealand.
Maurice Wilkins Centre for Molecular Biodiscovery, The University of Auckland, Auckland, New Zealand.

Beate Laube (B)

Energy Metabolism Laboratory, Institute of Translational Medicine, Department of Health Sciences and Technology, Swiss Federal Institute of Technology (ETH), Zürich, Switzerland.

Doris Pöhlmann (D)

Energy Metabolism Laboratory, Institute of Translational Medicine, Department of Health Sciences and Technology, Swiss Federal Institute of Technology (ETH), Zürich, Switzerland.

Stephan Wueest (S)

Division of Pediatric Endocrinology and Diabetology and Children's Research Centre, University Children's Hospital, Zurich, Switzerland.

Michael E Walsh (ME)

Energy Metabolism Laboratory, Institute of Translational Medicine, Department of Health Sciences and Technology, Swiss Federal Institute of Technology (ETH), Zürich, Switzerland.

Myrtha Arnold (M)

Physiology and Behavior Laboratory, Institute of Food and Nutrition, Department of Health Sciences and Technology, Swiss Federal Institute of Technology (ETH), Zürich, Switzerland.

Wolfgang Langhans (W)

Physiology and Behavior Laboratory, Institute of Food and Nutrition, Department of Health Sciences and Technology, Swiss Federal Institute of Technology (ETH), Zürich, Switzerland.

Daniel Konrad (D)

Division of Pediatric Endocrinology and Diabetology and Children's Research Centre, University Children's Hospital, Zurich, Switzerland.

Kim Zarse (K)

Energy Metabolism Laboratory, Institute of Translational Medicine, Department of Health Sciences and Technology, Swiss Federal Institute of Technology (ETH), Zürich, Switzerland.

Michael Ristow (M)

Energy Metabolism Laboratory, Institute of Translational Medicine, Department of Health Sciences and Technology, Swiss Federal Institute of Technology (ETH), Zürich, Switzerland. michael-ristow@ethz.ch.

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