Effects of sleep apnea and kidney dysfunction on objective sleep quality in nondialyzed patients with chronic kidney disease: an ESADA study.


Journal

Journal of clinical sleep medicine : JCSM : official publication of the American Academy of Sleep Medicine
ISSN: 1550-9397
Titre abrégé: J Clin Sleep Med
Pays: United States
ID NLM: 101231977

Informations de publication

Date de publication:
15 09 2020
Historique:
pubmed: 5 5 2020
medline: 24 6 2021
entrez: 5 5 2020
Statut: ppublish

Résumé

Patients with chronic kidney disease (CKD) often report poor sleep quality, but they commonly exhibit OSA. The aim of this study was to evaluate the influence of OSA severity and of estimated glomerular filtration rate impairment on objective sleep quality in nondialyzed patients with CKD, defined as an estimated glomerular filtration rate <60 mL/min/1.73m². Polysomnographic sleep characteristics were compared between patients with (n = 430) and without CKD (n = 6,639) in the European Sleep Apnea Database cohort. Comparisons were repeated in 375 patients with CKD and 375 control patients without CKD matched for sleep center, age, sex, and AHI, and in 310 matched CKD and non-CKD patients without psychiatric disturbances. Among all patients with and without CKD, total sleep time was similar but sleep stage N1 (median 8.7% [IQR 4.8-18.0] vs 6.7% [3.6-12.7], respectively) and sleep stage R (12.6% [6.8-17.7] vs 14.2% [8.8-19.8], respectively) significantly differed (P < .0001). No difference in sleep characteristics was observed between matched patients either with or without psychiatric disturbances. After subdividing the matched patients according to AHI tertile (<25, ≥25 to <49, and ≥49 events/h) and estimated glomerular filtration rate (≥60, 45 to <60, <45 mL/min/1.73m²), we found a significant effect of AHI on sleep stages N2, N3, and R (P < .001), but there was no effect of CKD. In nondialyzed patients with CKD, objective sleep quality is influenced similarly by AHI as in patients without CKD but is not affected by CKD severity. Previously reported poor sleep quality in CKD may partly result from the high prevalence of OSA in CKD.

Identifiants

pubmed: 32364929
doi: 10.5664/jcsm.8542
pmc: PMC7970586
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1475-1481

Informations de copyright

© 2020 American Academy of Sleep Medicine.

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Auteurs

Oreste Marrone (O)

Istituto per la Ricerca e l'Innovazione Biomedica, National Research Council, Palermo, Italy.

Fabio Cibella (F)

Istituto per la Ricerca e l'Innovazione Biomedica, National Research Council, Palermo, Italy.

Gabriel Roisman (G)

Sleep Disorders Centre, Antoine-Béclère Hospital, Clamart, France.

Pawel Sliwinski (P)

Second Department of Respiratory Medicine, Institute of Tuberculosis and Lung Diseases, Warsaw, Poland.

Pavol Joppa (P)

Department of Respiratory Medicine and Tuberculosis, P.J. Safarik University, Kosice, Slovakia.

Ozen K Basoglu (OK)

Department of Chest Diseases, Ege University, Izmir, Turkey.

Izolde Bouloukaki (I)

Sleep Disorders Center, Department of Respiratory Medicine, University of Crete, Heraklion, Greece.

Sophia Schiza (S)

Sleep Disorders Center, Department of Respiratory Medicine, University of Crete, Heraklion, Greece.

Athanasia Pataka (A)

Respiratory Failure Unit, G. Papanikolaou Hospital, Aristotle University of Thessaloniki, Greece.

Richard Staats (R)

Department of Respiratory Medicine, Hospital de Santa Maria, CHULN, Lisbon, Portugal.

Johan Verbraecken (J)

Multidisciplinary Sleep Disorders Centre, Antwerp University Hospital, Antwerp, Belgium.

Jan Hedner (J)

Department of Sleep Medicine, Sahlgrenska University Hospital, Gothenburg, Sweden.

Ludger Grote (L)

Department of Sleep Medicine, Sahlgrenska University Hospital, Gothenburg, Sweden.

Maria R Bonsignore (MR)

Istituto per la Ricerca e l'Innovazione Biomedica, National Research Council, Palermo, Italy.
Biomedical Department of Internal and Specialistic Medicine, University of Palermo, Italy.

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