Cooperation Between Hypoxia-Inducible Factor 1α and Activating Transcription Factor 4 in Sleep Apnea-Mediated Myocardial Injury.


Journal

The Canadian journal of cardiology
ISSN: 1916-7075
Titre abrégé: Can J Cardiol
Pays: England
ID NLM: 8510280

Informations de publication

Date de publication:
06 2020
Historique:
received: 17 12 2019
revised: 01 04 2020
accepted: 01 04 2020
pubmed: 11 5 2020
medline: 17 2 2021
entrez: 11 5 2020
Statut: ppublish

Résumé

Chronic intermittent hypoxia (CIH) occurring during sleep apnea amplifies infarct size owing to ischemia-reperfusion. CIH activates hypoxia-inducible factor 1 (HIF-1) and activating transcription factor 4 (ATF4). However, whether HIF-1 and ATF4 interact to promote cardiomyocyte death remains unexplored. For the first time, we observed that in myocardium from apneic patients, CCAAT enhancer-binding protein homologous protein (CHOP) expression is increased and HIF-1α expression is correlated with sleep apnea severity. In mice, single-allele deletion of HIF-1α prevents CIH increase in CHOP expression and infarct size. We uncovered a physical interaction between HIF-1α and ATF4 in CIH that may represent a novel cardiomyocyte death complex.

Identifiants

pubmed: 32387037
pii: S0828-282X(20)30324-X
doi: 10.1016/j.cjca.2020.04.002
pii:
doi:

Substances chimiques

ATF4 protein, human 0
Atf4 protein, mouse 0
CCAAT-Enhancer-Binding Proteins 0
HIF1A protein, human 0
Hif1a protein, mouse 0
Hypoxia-Inducible Factor 1, alpha Subunit 0
Activating Transcription Factor 4 145891-90-3

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

936-940

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2020 Canadian Cardiovascular Society. Published by Elsevier Inc. All rights reserved.

Auteurs

Sophie Moulin (S)

Laboratoire HP2, Institut National de la Santé et de la Recherche Médicale, Université Grenoble Alpes, Grenoble, France; Institut National de la Santé et de la Recherche Médicale U1042, Grenoble, France.

Amandine Thomas (A)

Laboratoire HP2, Institut National de la Santé et de la Recherche Médicale, Université Grenoble Alpes, Grenoble, France; Institut National de la Santé et de la Recherche Médicale U1042, Grenoble, France.

Claire Arnaud (C)

Laboratoire HP2, Institut National de la Santé et de la Recherche Médicale, Université Grenoble Alpes, Grenoble, France; Institut National de la Santé et de la Recherche Médicale U1042, Grenoble, France.

Michael Arzt (M)

Department of Internal Medicine II, University Hospital Regensburg, Regensburg, Germany.

Stefan Wagner (S)

Department of Internal Medicine II, University Hospital Regensburg, Regensburg, Germany.

Lars S Maier (LS)

Department of Internal Medicine II, University Hospital Regensburg, Regensburg, Germany.

Jean-Louis Pépin (JL)

Laboratoire HP2, Institut National de la Santé et de la Recherche Médicale, Université Grenoble Alpes, Grenoble, France; Institut National de la Santé et de la Recherche Médicale U1042, Grenoble, France; Centre Hospitalier Universitaire des Alpes, Grenoble, France.

Diane Godin-Ribuot (D)

Laboratoire HP2, Institut National de la Santé et de la Recherche Médicale, Université Grenoble Alpes, Grenoble, France; Institut National de la Santé et de la Recherche Médicale U1042, Grenoble, France.

Jonathan Gaucher (J)

Laboratoire HP2, Institut National de la Santé et de la Recherche Médicale, Université Grenoble Alpes, Grenoble, France; Institut National de la Santé et de la Recherche Médicale U1042, Grenoble, France.

Elise Belaidi (E)

Laboratoire HP2, Institut National de la Santé et de la Recherche Médicale, Université Grenoble Alpes, Grenoble, France; Institut National de la Santé et de la Recherche Médicale U1042, Grenoble, France. Electronic address: elise.belaidi-corsat@univ-grenoble-alpes.fr.

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Classifications MeSH