A rat arteriovenous graft model using decellularized vein.


Journal

Vascular
ISSN: 1708-539X
Titre abrégé: Vascular
Pays: England
ID NLM: 101196722

Informations de publication

Date de publication:
Oct 2020
Historique:
pubmed: 12 5 2020
medline: 22 9 2020
entrez: 12 5 2020
Statut: ppublish

Résumé

The high rate of clinical failure of prosthetic arteriovenous grafts continues to suggest the need for novel tissue-engineered vascular grafts. We tested the hypothesis that the decellularized rat jugular vein could be successfully used as a conduit and that it would support reendothelialization as well as adaptation to the arterial environment. Autologous (control) or heterologous decellularized jugular vein (1 cm length, 1 mm diameter) was sewn between the inferior vena cava and aorta as an arteriovenous graft in Wistar rats. Rats were sacrificed on postoperative day 21 for examination. All rats survived, and grafts had 100% patency in both the control and decellularized groups. Both control and decellularized jugular vein grafts showed similar rates of reendothelialization, smooth muscle cell deposition, macrophage infiltration, and cell turnover. The outflow veins distal to the grafts showed similar adaptation to the arteriovenous flow. Both CD34, CD90 and nestin positive cells, as well as M1-type and M2-type macrophages accumulated around the graft. This model shows that decellularized vein can be successfully used as an arteriovenous graft between the rat aorta and the inferior vena cava. Several types of cells, including progenitor cells and macrophages, are present in the host response to these grafts in this model. This model can be used to test the application of arteriovenous grafts before conducting large animal experiments.

Sections du résumé

BACKGROUND BACKGROUND
The high rate of clinical failure of prosthetic arteriovenous grafts continues to suggest the need for novel tissue-engineered vascular grafts. We tested the hypothesis that the decellularized rat jugular vein could be successfully used as a conduit and that it would support reendothelialization as well as adaptation to the arterial environment.
MATERIALS AND METHODS METHODS
Autologous (control) or heterologous decellularized jugular vein (1 cm length, 1 mm diameter) was sewn between the inferior vena cava and aorta as an arteriovenous graft in Wistar rats. Rats were sacrificed on postoperative day 21 for examination.
RESULTS RESULTS
All rats survived, and grafts had 100% patency in both the control and decellularized groups. Both control and decellularized jugular vein grafts showed similar rates of reendothelialization, smooth muscle cell deposition, macrophage infiltration, and cell turnover. The outflow veins distal to the grafts showed similar adaptation to the arteriovenous flow. Both CD34, CD90 and nestin positive cells, as well as M1-type and M2-type macrophages accumulated around the graft.
CONCLUSIONS CONCLUSIONS
This model shows that decellularized vein can be successfully used as an arteriovenous graft between the rat aorta and the inferior vena cava. Several types of cells, including progenitor cells and macrophages, are present in the host response to these grafts in this model. This model can be used to test the application of arteriovenous grafts before conducting large animal experiments.

Identifiants

pubmed: 32390561
doi: 10.1177/1708538120923191
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

664-672

Auteurs

Hualong Bai (H)

Department of Vascular and Endovascular Surgery, First Affiliated Hospital of Zhengzhou University, Henan, China.
Key Vascular Physiology and Applied Research Laboratory of Zhengzhou City, Henan, China.

Zhiwei Wang (Z)

Department of Vascular and Endovascular Surgery, First Affiliated Hospital of Zhengzhou University, Henan, China.

Mingxing Li (M)

Department of Vascular and Endovascular Surgery, First Affiliated Hospital of Zhengzhou University, Henan, China.

Peng Sun (P)

Department of Vascular and Endovascular Surgery, First Affiliated Hospital of Zhengzhou University, Henan, China.

Wang Wang (W)

Key Vascular Physiology and Applied Research Laboratory of Zhengzhou City, Henan, China.
Department of Physiology, Medical school of Zhengzhou University, Henan, China.

Weizhen Liu (W)

Key Vascular Physiology and Applied Research Laboratory of Zhengzhou City, Henan, China.
Department of Physiology, Medical school of Zhengzhou University, Henan, China.

Shunbo Wei (S)

Department of Vascular and Endovascular Surgery, First Affiliated Hospital of Zhengzhou University, Henan, China.

Zhiju Wang (Z)

Key Vascular Physiology and Applied Research Laboratory of Zhengzhou City, Henan, China.
Department of Physiology, Medical school of Zhengzhou University, Henan, China.

Ying Xing (Y)

Key Vascular Physiology and Applied Research Laboratory of Zhengzhou City, Henan, China.
Department of Physiology, Medical school of Zhengzhou University, Henan, China.

Alan Dardik (A)

The Vascular Biology and Therapeutics Program, Yale University School of Medicine, New Haven, CT, USA.
Department of Surgery and of Cellular and Molecular Physiology, Yale University School of Medicine, New Haven, CT, USA.

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Classifications MeSH