A global treatments for coronaviruses including COVID-19.


Journal

Journal of cellular physiology
ISSN: 1097-4652
Titre abrégé: J Cell Physiol
Pays: United States
ID NLM: 0050222

Informations de publication

Date de publication:
12 2020
Historique:
received: 15 04 2020
accepted: 03 05 2020
pubmed: 13 5 2020
medline: 8 10 2020
entrez: 13 5 2020
Statut: ppublish

Résumé

In late December 2019 in Wuhan, China, several patients with viral pneumonia were identified as 2019 novel coronavirus (2019-nCoV). So far, there are no specific treatments for patients with coronavirus disease-19 (COVID-19), and the treatments available today are based on previous experience with similar viruses such as severe acute respiratory syndrome-related coronavirus (SARS-CoV), Middle East respiratory syndrome coronavirus (MERS-CoV), and Influenza virus. In this article, we have tried to reach a therapeutic window of drugs available to patients with COVID-19. Cathepsin L is required for entry of the 2019-nCoV virus into the cell as target teicoplanin inhibits virus replication. Angiotensin-converting-enzyme 2 (ACE2) in soluble form as a recombinant protein can prevent the spread of coronavirus by restricting binding and entry. In patients with COVID-19, hydroxychloroquine decreases the inflammatory response and cytokine storm, but overdose causes toxicity and mortality. Neuraminidase inhibitors such as oseltamivir, peramivir, and zanamivir are invalid for 2019-nCoV and are not recommended for treatment but protease inhibitors such as lopinavir/ritonavir (LPV/r) inhibit the progression of MERS-CoV disease and can be useful for patients of COVID-19 and, in combination with Arbidol, has a direct antiviral effect on early replication of SARS-CoV. Ribavirin reduces hemoglobin concentrations in respiratory patients, and remdesivir improves respiratory symptoms. Use of ribavirin in combination with LPV/r in patients with SARS-CoV reduces acute respiratory distress syndrome and mortality, which has a significant protective effect with the addition of corticosteroids. Favipiravir increases clinical recovery and reduces respiratory problems and has a stronger antiviral effect than LPV/r. currently, appropriate treatment for patients with COVID-19 is an ACE2 inhibitor and a clinical problem reducing agent such as favipiravir in addition to hydroxychloroquine and corticosteroids.

Identifiants

pubmed: 32394467
doi: 10.1002/jcp.29785
pmc: PMC7273044
doi:

Substances chimiques

Antiviral Agents 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

9133-9142

Informations de copyright

© 2020 Wiley Periodicals LLC.

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Auteurs

Bahman Yousefi (B)

Department of Immunology, Semnan University of Medical Sciences, Semnan, Iran.

Saeid Valizadeh (S)

Department of Bacteriology and Virology, Semnan University of Medical Sciences, Semnan, Iran.

Hadi Ghaffari (H)

Department of Bacteriology and Virology, Semnan University of Medical Sciences, Semnan, Iran.

Azadeh Vahedi (A)

Department of Bacteriology and Virology, Semnan University of Medical Sciences, Semnan, Iran.

Mohsen Karbalaei (M)

Department of Microbiology and Virology, School of Medicine, Jiroft University of Medical Sciences, Jiroft, Iran.

Majid Eslami (M)

Department of Bacteriology and Virology, Semnan University of Medical Sciences, Semnan, Iran.
Cancer Research Center, Semnan University of Medical Sciences, Semnan, Iran.

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