Vitamin D-Cathelicidin Axis: at the Crossroads between Protective Immunity and Pathological Inflammation during Infection.

Autophagy Cathelicidin Immunity Infections Inflammation Vitamin D

Journal

Immune network
ISSN: 1598-2629
Titre abrégé: Immune Netw
Pays: Korea (South)
ID NLM: 101137270

Informations de publication

Date de publication:
Apr 2020
Historique:
received: 27 10 2019
revised: 28 01 2020
accepted: 30 01 2020
entrez: 13 5 2020
pubmed: 13 5 2020
medline: 13 5 2020
Statut: epublish

Résumé

Vitamin D signaling plays an essential role in innate defense against intracellular microorganisms via the generation of the antimicrobial protein cathelicidin. In addition to directly binding to and killing a range of pathogens, cathelicidin acts as a secondary messenger driving vitamin D-mediated inflammation during infection. Recent studies have elucidated the biological and clinical functions of cathelicidin in the context of vitamin D signaling. The vitamin D-cathelicidin axis is involved in the activation of autophagy, which enhances antimicrobial effects against diverse pathogens. Vitamin D studies have also revealed positive and negative regulatory effects of cathelicidin on inflammatory responses to pathogenic stimuli. Diverse innate and adaptive immune signals crosstalk with functional vitamin D receptor signals to enhance the role of cathelicidin action in cell-autonomous effector systems. In this review, we discuss recent findings that demonstrate how the vitamin D-cathelicidin pathway regulates autophagy machinery, protective immune defenses, and inflammation, and contributes to immune cooperation between innate and adaptive immunity. Understanding how the vitamin D-cathelicidin axis operates in the host response to infection will create opportunities for the development of new therapeutic approaches against a variety of infectious diseases.

Identifiants

pubmed: 32395364
doi: 10.4110/in.2020.20.e12
pmc: PMC7192829
doi:

Types de publication

Journal Article Review

Langues

eng

Pagination

e12

Informations de copyright

Copyright © 2020. The Korean Association of Immunologists.

Déclaration de conflit d'intérêts

Conflict of Interest: The authors declare no potential conflicts of interest.

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Auteurs

Chaeuk Chung (C)

Division of Pulmonary and Critical Care, Department of Internal Medicine, Chungnam National University School of Medicine, Daejeon 35015, Korea.

Prashanta Silwal (P)

Infection Control Convergence Research Center, Chungnam National University School of Medicine, Daejeon 35015, Korea.
Department of Microbiology, Chungnam National University School of Medicine, Daejeon 35015, Korea.

Insoo Kim (I)

Infection Control Convergence Research Center, Chungnam National University School of Medicine, Daejeon 35015, Korea.
Department of Microbiology, Chungnam National University School of Medicine, Daejeon 35015, Korea.

Robert L Modlin (RL)

Division of Dermatology, Department of Medicine, David Geffen School of Medicine at the University of California, Los Angeles, Los Angeles, CA 90095, USA.
Department of Microbiology, Immunology and Molecular Genetics, University of California, Los Angeles, Los Angeles, CA 90095, USA.

Eun-Kyeong Jo (EK)

Infection Control Convergence Research Center, Chungnam National University School of Medicine, Daejeon 35015, Korea.
Department of Microbiology, Chungnam National University School of Medicine, Daejeon 35015, Korea.
Department of Medical Science, Chungnam National University School of Medicine, Daejeon 35015, Korea.

Classifications MeSH