EHD2 is a Predictive Biomarker of Chemotherapy Efficacy in Triple Negative Breast Carcinoma.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
14 05 2020
Historique:
received: 03 10 2019
accepted: 27 04 2020
entrez: 16 5 2020
pubmed: 16 5 2020
medline: 15 12 2020
Statut: epublish

Résumé

EHD2 is a mechanotransducing ATPase localized in caveolae invaginations at the plasma membrane. EHD2 has recently been associated with several human cancers, however the significance of EHD2 transcript levels in cancer prognosis remains debated. Breast cancer is the most commonly occurring cancer in women and prognosis is variable depending on the subtypes. Triple negative breast cancer (TNBC) often has a poor therapeutic response. The aim of this study was to assess the prognostic significance of EHD2 transcripts and protein expression levels in breast carcinomas. We found that low EHD2 levels were associated with enhanced proliferation, migration and invasion of TNBC cells. EHD2 expression was significantly reduced in TNBC tissues and the loss of EHD2 led to higher expression of the pro-tumoral cytokine IL-8. In apparent contradiction with in vitro data, multivariate analysis of two independent cohorts of breast cancer patients revealed that low EHD2 was in fact associated with good prognosis in the highly proliferative TNBC subtype. Accordingly, TNBC low EHD2 expressers were found to benefit the most from chemotherapy when compared to all subtypes of breast cancers. Our study validates EHD2 expression level as an independent prognostic factor of metastasis-free survival and as a new predictive marker of chemotherapy efficacy in TNBC patients.

Identifiants

pubmed: 32409676
doi: 10.1038/s41598-020-65054-5
pii: 10.1038/s41598-020-65054-5
pmc: PMC7224205
doi:

Substances chimiques

Biomarkers, Tumor 0
Carrier Proteins 0
EHD2 protein, human 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

7998

Références

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Auteurs

Wei-Wei Shen (WW)

Institut Curie - Centre de Recherche, PSL Research University, Membrane Dynamics and Mechanics of Intracellular Signaling team, 75248, Paris, cedex 05, France.
Institut National de la Santé et de la Recherche Médicale (INSERM), U1143, Paris, France.
Centre National de la Recherche Scientifique (CNRS), UMR, 3666, Paris, France.

Ivan Bièche (I)

Pharmacogenomics Unit, Department of Genetics, Institut Curie, 26 rue d'Ulm, 75248, Paris, cedex 05, France.

Laetitia Fuhrmann (L)

Department of Pathology, Institut Curie, 26 rue d'Ulm, 75248, Paris, cedex 05, France.

Sophie Vacher (S)

Pharmacogenomics Unit, Department of Genetics, Institut Curie, 26 rue d'Ulm, 75248, Paris, cedex 05, France.

Anne Vincent-Salomon (A)

Pharmacogenomics Unit, Department of Genetics, Institut Curie, 26 rue d'Ulm, 75248, Paris, cedex 05, France.
Institut Curie, PSL Research University, INSERM U830, 26 rue d'Ulm, 75248, Paris, cedex 05, France.

Stéphanie Torrino (S)

CNRS UMR7275, Institut de Pharmacologie Cellulaire et Moléculaire, Université Côte d'Azur, 06560, Valbonne, France. stephanie.torrino@unice.fr.

Christophe Lamaze (C)

Institut Curie - Centre de Recherche, PSL Research University, Membrane Dynamics and Mechanics of Intracellular Signaling team, 75248, Paris, cedex 05, France. christophe.lamaze@curie.fr.
Institut National de la Santé et de la Recherche Médicale (INSERM), U1143, Paris, France. christophe.lamaze@curie.fr.
Centre National de la Recherche Scientifique (CNRS), UMR, 3666, Paris, France. christophe.lamaze@curie.fr.

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