CX3CR1-Targeted PLGA Nanoparticles Reduce Microglia Activation and Pain Behavior in Rats with Spinal Nerve Ligation.


Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
14 May 2020
Historique:
received: 23 04 2020
revised: 08 05 2020
accepted: 12 05 2020
entrez: 20 5 2020
pubmed: 20 5 2020
medline: 12 2 2021
Statut: epublish

Résumé

Activation of CX3CR1 in microglia plays an important role in the development of neuropathic pain. Here, we investigated whether neuropathic pain could be attenuated in spinal nerve ligation (SNL)-induced rats by reducing microglial activation through the use of poly(D,L-lactic-co-glycolic acid) (PLGA)-encapsulated CX3CR1 small-interfering RNA (siRNA) nanoparticles. After confirming the efficacy and specificity of CX3CR1 siRNA, as evidenced by its anti-inflammatory effects in lipopolysaccharide-stimulated BV2 cells in vitro, PLGA-encapsulated CX3CR1 siRNA nanoparticles were synthesized by sonication using the conventional double emulsion (W/O/W) method and administered intrathecally into SNL rats. CX3CR1 siRNA-treated rats exhibited significant reductions in the activation of microglia in the spinal dorsal horn and a downregulation of proinflammatory mediators, as well as a significant attenuation of mechanical allodynia. These data indicate that the PLGA-encapsulated CX3CR1 siRNA nanoparticles effectively reduce neuropathic pain in SNL-induced rats by reducing microglial activity and the expression of proinflammatory mediators. Therefore, we believe that PLGA-encapsulated CX3CR1 siRNA nanoparticles represent a valuable new treatment option for neuropathic pain.

Identifiants

pubmed: 32423102
pii: ijms21103469
doi: 10.3390/ijms21103469
pmc: PMC7279022
pii:
doi:

Substances chimiques

CX3C Chemokine Receptor 1 0
CX3CR1 protein, rat 0
Lipopolysaccharides 0
RNA, Small Interfering 0
Polylactic Acid-Polyglycolic Acid Copolymer 1SIA8062RS

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : National Research Foundation of Korea
ID : NRF- 2019R1A2C2004884, NRF- 2020R1A2C1007575 and NRF-2020R1C1C1005423

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Auteurs

Chan Noh (C)

Department of Anesthesiology and Pain Medicine, Chungnam National University Hospital, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Korea.

Hyo Jung Shin (HJ)

Department of Medical Science, 3 Department of Anatomy and Cell Biology, 4 Brain Research Institute, School of medicine, Chungnam National University, Daejeon 35015, Korea.

Seounghun Lee (S)

Department of Anesthesiology and Pain Medicine, Chungnam National University Hospital, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Korea.

Song I Kim (SI)

Department of Medical Science, 3 Department of Anatomy and Cell Biology, 4 Brain Research Institute, School of medicine, Chungnam National University, Daejeon 35015, Korea.

Yoon-Hee Kim (YH)

Department of Anesthesiology and Pain Medicine, Chungnam National University Hospital, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Korea.

Won Hyung Lee (WH)

Department of Anesthesiology and Pain Medicine, Chungnam National University Hospital, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Korea.

Dong Woon Kim (DW)

Department of Medical Science, 3 Department of Anatomy and Cell Biology, 4 Brain Research Institute, School of medicine, Chungnam National University, Daejeon 35015, Korea.

Sun Yeul Lee (SY)

Department of Anesthesiology and Pain Medicine, Chungnam National University Hospital, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Korea.

Young Kwon Ko (YK)

Department of Anesthesiology and Pain Medicine, Chungnam National University Hospital, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Korea.

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Classifications MeSH