Ectopic expression of SOX18 in Basal cell carcinoma negatively regulates tumour progression.


Journal

Journal of dermatological science
ISSN: 1873-569X
Titre abrégé: J Dermatol Sci
Pays: Netherlands
ID NLM: 9011485

Informations de publication

Date de publication:
Jun 2020
Historique:
received: 27 11 2019
revised: 14 04 2020
accepted: 16 04 2020
pubmed: 24 5 2020
medline: 15 4 2021
entrez: 24 5 2020
Statut: ppublish

Résumé

Basal Cell Carcinoma is the most common tumour and yet much remains to be determined regarding the molecular mechanisms that leads to its development. Hedgehog signal activation is sufficient for BCC induction, but the molecular mediators of BCC growth are not well understood. SoxF transcription factor Sox18 has been identified in human BCC, but its role in growth of the tumour is as yet unknown. To determine if Sox18 is involved in the regulation of Basal Cell Carcinoma growth. We analysed the function of Sox18 by combining a dominant negative Sox18 mouse model, Sox18 These data suggest that Sox18 is a hedgehog regulated mediator of tumour suppression within Basal Cell Carcinoma epidermis.

Sections du résumé

BACKGROUND BACKGROUND
Basal Cell Carcinoma is the most common tumour and yet much remains to be determined regarding the molecular mechanisms that leads to its development. Hedgehog signal activation is sufficient for BCC induction, but the molecular mediators of BCC growth are not well understood. SoxF transcription factor Sox18 has been identified in human BCC, but its role in growth of the tumour is as yet unknown.
OBJECTIVE OBJECTIVE
To determine if Sox18 is involved in the regulation of Basal Cell Carcinoma growth.
METHODS METHODS
We analysed the function of Sox18 by combining a dominant negative Sox18 mouse model, Sox18
CONCLUSIONS CONCLUSIONS
These data suggest that Sox18 is a hedgehog regulated mediator of tumour suppression within Basal Cell Carcinoma epidermis.

Identifiants

pubmed: 32444239
pii: S0923-1811(20)30142-0
doi: 10.1016/j.jdermsci.2020.04.006
pii:
doi:

Substances chimiques

Hedgehog Proteins 0
Patched-1 Receptor 0
Ptch1 protein, mouse 0
Receptors, Notch 0
SOXF Transcription Factors 0
Sox18 protein, mouse 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

179-185

Informations de copyright

Copyright © 2020 Japanese Society for Investigative Dermatology. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The Authors have no Conflict of Interest to declare.

Auteurs

Rehan Villani (R)

The University of Queensland, UQ Diamantina Institute, Translational Research Institute, Brisbane, QLD, Australia. Electronic address: r.villani@uq.edu.au.

Seen Ling Sim (SL)

The University of Queensland, UQ Diamantina Institute, Translational Research Institute, Brisbane, QLD, Australia.

Edwige Roy (E)

The University of Queensland, UQ Diamantina Institute, Translational Research Institute, Brisbane, QLD, Australia.

Brandon Wainwright (B)

The University of Queensland, Institute for Molecular Bioscience, Brisbane, QLD, Australia.

Mathias Francois (M)

The University of Queensland, Institute for Molecular Bioscience, Brisbane, QLD, Australia; The University of Sydney, The Centenary Institute, David Richmond Program for Cardio-Vascular Research: GeneRegulation and Editing, Sydney Medical School, Royal Prince Alfred Hospital, Missenden Rd, NSW, Australia.

Kiarash Khosrotehrani (K)

The University of Queensland, UQ Diamantina Institute, Translational Research Institute, Brisbane, QLD, Australia. Electronic address: k.khosrotehrani@uq.edu.au.

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Classifications MeSH