Long non-coding RNA LASSIE regulates shear stress sensing and endothelial barrier function.


Journal

Communications biology
ISSN: 2399-3642
Titre abrégé: Commun Biol
Pays: England
ID NLM: 101719179

Informations de publication

Date de publication:
26 05 2020
Historique:
received: 10 03 2020
accepted: 04 05 2020
entrez: 28 5 2020
pubmed: 28 5 2020
medline: 16 6 2021
Statut: epublish

Résumé

Blood vessels are constantly exposed to shear stress, a biomechanical force generated by blood flow. Normal shear stress sensing and barrier function are crucial for vascular homeostasis and are controlled by adherens junctions (AJs). Here we show that AJs are stabilized by the shear stress-induced long non-coding RNA LASSIE (linc00520). Silencing of LASSIE in endothelial cells impairs cell survival, cell-cell contacts and cell alignment in the direction of flow. LASSIE associates with junction proteins (e.g. PECAM-1) and the intermediate filament protein nestin, as identified by RNA affinity purification. The AJs component VE-cadherin showed decreased stabilization, due to reduced interaction with nestin and the microtubule cytoskeleton in the absence of LASSIE. This study identifies LASSIE as link between nestin and VE-cadherin, and describes nestin as crucial component in the endothelial response to shear stress. Furthermore, this study indicates that LASSIE regulates barrier function by connecting AJs to the cytoskeleton.

Identifiants

pubmed: 32457386
doi: 10.1038/s42003-020-0987-0
pii: 10.1038/s42003-020-0987-0
pmc: PMC7251106
doi:

Substances chimiques

RNA, Long Noncoding 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

265

Références

Dekker, R. J. et al. KLF2 provokes a gene expression pattern that establishes functional quiescent differentiation of the endothelium. Blood 107, 4354–4363 (2006).
pubmed: 16455954 doi: 10.1182/blood-2005-08-3465
Parmar, K. M. et al. Integration of flow-dependent endothelial phenotypes by Kruppel-like factor 2. J. Clin. Invest. 116, 49–58 (2006).
pubmed: 16341264 doi: 10.1172/JCI24787
Hamik, A. et al. Kruppel-like factor 4 regulates endothelial inflammation. J. Biol. Chem. 282, 13769–13779 (2007).
pubmed: 17339326 doi: 10.1074/jbc.M700078200
Dewey, C. F. Jr., Bussolari, S. R., Gimbrone, M. A. Jr. & Davies, P. F. The dynamic response of vascular endothelial cells to fluid shear stress. J. Biomech. Eng. 103, 177–185 (1981).
pubmed: 7278196 doi: 10.1115/1.3138276
Kris, A. S., Kamm, R. D. & Sieminski, A. L. VASP involvement in force-mediated adherens junction strengthening. Biochem. Biophys. Res. Commun. 375, 134–138 (2008).
pubmed: 18680720 pmcid: 2561196 doi: 10.1016/j.bbrc.2008.07.132
Liu, Z. et al. Mechanical tugging force regulates the size of cell-cell junctions. Proc. Natl Acad. Sci. USA 107, 9944–9949 (2010).
pubmed: 20463286 doi: 10.1073/pnas.0914547107
Weis, S. M. Vascular permeability in cardiovascular disease and cancer. Curr. Opin. Hematol. 15, 243–249 (2008).
pubmed: 18391792 doi: 10.1097/MOH.0b013e3282f97d86
Mehta, D. & Malik, A. B. Signaling mechanisms regulating endothelial permeability. Physiol. Rev. 86, 279–367 (2006).
pubmed: 16371600 doi: 10.1152/physrev.00012.2005
Gonzalez-Mariscal, L., Tapia, R. & Chamorro, D. Crosstalk of tight junction components with signaling pathways. Biochim. Biophys. Acta 1778, 729–756 (2008).
pubmed: 17950242 doi: 10.1016/j.bbamem.2007.08.018
Bazzoni, G. & Dejana, E. Endothelial cell-to-cell junctions: molecular organization and role in vascular homeostasis. Physiol. Rev. 84, 869–901 (2004).
pubmed: 15269339 doi: 10.1152/physrev.00035.2003
Wallez, Y. & Huber, P. Endothelial adherens and tight junctions in vascular homeostasis, inflammation and angiogenesis. Biochim. Biophys. Acta 1778, 794–809 (2008).
pubmed: 17961505 doi: 10.1016/j.bbamem.2007.09.003
Dejana, E., Tournier-Lasserve, E. & Weinstein, B. M. The control of vascular integrity by endothelial cell junctions: molecular basis and pathological implications. Dev. Cell 16, 209–221 (2009).
pubmed: 19217423 doi: 10.1016/j.devcel.2009.01.004
Vestweber, D. VE-cadherin: the major endothelial adhesion molecule controlling cellular junctions and blood vessel formation. Arterioscler. Thromb. Vasc. Biol. 28, 223–232 (2008).
pubmed: 18162609 doi: 10.1161/ATVBAHA.107.158014
Biswas, P. et al. Identification of the regions of PECAM-1 involved in beta- and gamma-catenin associations. Biochem. Biophys. Res. Commun. 329, 1225–1233 (2005).
pubmed: 15766557 doi: 10.1016/j.bbrc.2005.02.095
Tzima, E. et al. A mechanosensory complex that mediates the endothelial cell response to fluid shear stress. Nature 437, 426–431 (2005).
doi: 10.1038/nature03952
Conway, D. E. et al. Fluid shear stress on endothelial cells modulates mechanical tension across VE-cadherin and PECAM-1. Curr. Biol. 23, 1024–1030 (2013).
pubmed: 23684974 pmcid: 23684974 doi: 10.1016/j.cub.2013.04.049
Alieva, I. B. Role of microtubule cytoskeleton in regulation of endothelial barrier function. Biochemistry 79, 964–975 (2014).
pubmed: 25385022
Djebali, S. et al. Landscape of transcription in human cells. Nature 489, 101–108 (2012).
pubmed: 3684276 pmcid: 3684276 doi: 10.1038/nature11233
Mercer, T. R., Dinger, M. E. & Mattick, J. S. Long non-coding RNAs: insights into functions. Nat. Rev. Genet. 10, 155–159 (2009).
pubmed: 19188922 doi: 10.1038/nrg2521
Tsai, M. C. et al. Long noncoding RNA as modular scaffold of histone modification complexes. Science 329, 689–693 (2010).
pubmed: 20616235 pmcid: 2967777 doi: 10.1126/science.1192002
Kurian, L. et al. Identification of novel long noncoding RNAs underlying vertebrate cardiovascular development. Circulation 131, 1278–1290 (2015).
pubmed: 25739401 pmcid: 4443804 doi: 10.1161/CIRCULATIONAHA.114.013303
Tichon, A. et al. A conserved abundant cytoplasmic long noncoding RNA modulates repression by Pumilio proteins in human cells. Nat. Commun. 7, 12209 (2016).
pubmed: 27406171 pmcid: 4947167 doi: 10.1038/ncomms12209
Hu, G., Lou, Z. & Gupta, M. The long non-coding RNA GAS5 cooperates with the eukaryotic translation initiation factor 4E to regulate c-Myc translation. PLoS One 9, e107016 (2014).
pubmed: 25197831 pmcid: 4157848 doi: 10.1371/journal.pone.0107016
Kaneko, S. et al. Interactions between JARID2 and noncoding RNAs regulate PRC2 recruitment to chromatin. Mol. Cell 53, 290–300 (2014).
pubmed: 24374312 doi: 10.1016/j.molcel.2013.11.012
Boon, R. A. et al. Long noncoding RNA Meg3 controls endothelial cell aging and function: implications for regenerative angiogenesis. J. Am. Coll. Cardiol. 68, 2589–2591 (2016).
pubmed: 27931619 doi: 10.1016/j.jacc.2016.09.949
Leisegang, M. S. et al. Long noncoding RNA MANTIS facilitates endothelial angiogenic function. Circulation 136, 65–79 (2017).
pubmed: 28351900 pmcid: 5491227 doi: 10.1161/CIRCULATIONAHA.116.026991
Neumann, P. et al. The lncRNA GATA6-AS epigenetically regulates endothelial gene expression via interaction with LOXL2. Nat. Commun. 9, 237 (2018).
pubmed: 29339785 pmcid: 5770451 doi: 10.1038/s41467-017-02431-1
Doddaballapur, A. et al. Laminar shear stress inhibits endothelial cell metabolism via KLF2-mediated repression of PFKFB3. Arterioscler. Thromb. Vasc. Biol. 35, 137–145 (2015).
pubmed: 25359860 doi: 10.1161/ATVBAHA.114.304277
Wang, L. et al. CPAT: Coding-Potential Assessment Tool using an alignment-free logistic regression model. Nucleic Acids Res. 41, e74 (2013).
pubmed: 23335781 pmcid: 3616698 doi: 10.1093/nar/gkt006
Sehnert, A. J. et al. Cardiac troponin T is essential in sarcomere assembly and cardiac contractility. Nat. Genet. 31, 106–110 (2002).
pubmed: 11967535 doi: 10.1038/ng875
Michalik, K. M. et al. Long noncoding RNA MALAT1 regulates endothelial cell function and vessel growth. Circ. Res. 114, 1389–1397 (2014).
pubmed: 24602777 doi: 10.1161/CIRCRESAHA.114.303265
Battich, N., Stoeger, T. & Pelkmans, L. Image-based transcriptomics in thousands of single human cells at single-molecule resolution. Nat. Methods 10, 1127–1133 (2013).
pubmed: 24097269 doi: 10.1038/nmeth.2657
Szulcek, R., Bogaard, H. J. & van Nieuw Amerongen, G. P. Electric cell-substrate impedance sensing for the quantification of endothelial proliferation, barrier function, and motility. J .Vis. Exp. 28, 51300 (2014).
Shay-Salit, A. et al. VEGF receptor 2 and the adherens junction as a mechanical transducer in vascular endothelial cells. Proc. Natl Acad. Sci. USA 99, 9462–9467 (2002).
pubmed: 12080144 doi: 10.1073/pnas.142224299
Chang, L. & Goldman, R. D. Intermediate filaments mediate cytoskeletal crosstalk. Nat. Rev. Mol. Cell Biol. 5, 601–613 (2004).
pubmed: 15366704 doi: 10.1038/nrm1438
Birukova, A. A. et al. Microtubule disassembly induces cytoskeletal remodeling and lung vascular barrier dysfunction: role of Rho-dependent mechanisms. J. Cell Physiol. 201, 55–70 (2004).
pubmed: 15281089 doi: 10.1002/jcp.20055
Man, H. S. J. et al. Angiogenic patterning by STEEL, an endothelial-enriched long noncoding RNA. Proc. Natl Acad. Sci. USA 115, 2401–2406 (2018).
pubmed: 29467285 doi: 10.1073/pnas.1715182115
Lyu, Q. et al. SENCR stabilizes vascular endothelial cell adherens junctions through interaction with CKAP4. Proc. Natl Acad. Sci. USA 116, 546–555 (2019).
pubmed: 30584103 doi: 10.1073/pnas.1810729116
Johnsson, P., Lipovich, L., Grander, D. & Morris, K. V. Evolutionary conservation of long non-coding RNAs; sequence, structure, function. Biochim. Biophys. Acta 1840, 1063–1071 (2014).
pubmed: 24184936 doi: 10.1016/j.bbagen.2013.10.035
Tsuruta, D. & Jones, J. C. The vimentin cytoskeleton regulates focal contact size and adhesion of endothelial cells subjected to shear stress. J. Cell Sci. 116, 4977–4984 (2003).
pubmed: 14625391 doi: 10.1242/jcs.00823
Nieuwenhuizen, R. P. et al. Co-orientation: quantifying simultaneous co-localization and orientational alignment of filaments in light microscopy. PLoS One 10, e0131756 (2015).
pubmed: 26161965 pmcid: 4498647 doi: 10.1371/journal.pone.0131756
Nieminen, M. et al. Vimentin function in lymphocyte adhesion and transcellular migration. Nat. Cell Biol. 8, 156–162 (2006).
pubmed: 16429129 doi: 10.1038/ncb1355
Liu, T. et al. Modulating endothelial barrier function by targeting vimentin phosphorylation. J. Cell Physiol. 229, 1484–1493 (2014).
pubmed: 24648251 doi: 10.1002/jcp.24590
Gan, Z. et al. Vimentin intermediate filaments template microtubule networks to enhance persistence in cell polarity and directed migration. Cell Syst. 3, 252–263.e258 (2016).
pubmed: 27667364 pmcid: 5055390 doi: 10.1016/j.cels.2016.08.007
Spagnuolo, R. et al. Gas1 is induced by VE-cadherin and vascular endothelial growth factor and inhibits endothelial cell apoptosis. Blood 103, 3005–3012 (2004).
pubmed: 15070677 doi: 10.1182/blood-2003-07-2459
Yue, B. et al. A positive feed-forward loop between LncRNA-CYTOR and Wnt/beta-catenin signaling promotes metastasis of colon cancer. Mol. Ther. 26, 1287–1298 (2018).
pubmed: 29606502 pmcid: 5993983 doi: 10.1016/j.ymthe.2018.02.024
Kloc, M. et al. Potential structural role of non-coding and coding RNAs in the organization of the cytoskeleton at the vegetal cortex of Xenopus oocytes. Development 132, 3445–3457 (2005).
pubmed: 16000384 doi: 10.1242/dev.01919
Szulcek, R. et al. Delayed Microvascular Shear Adaptation in Pulmonary Arterial Hypertension. Role of Platelet Endothelial Cell Adhesion Molecule-1 Cleavage. Am. J. Respir. Crit. Care Med. 193, 1410–1420 (2016).
pubmed: 26760925 pmcid: 6915853 doi: 10.1164/rccm.201506-1231OC
Daniel, A. E. et al. Plasminogen activator inhibitor-1 controls vascular integrity by regulating VE-cadherin trafficking. PLoS One 10, e0145684 (2015).
pubmed: 26714278 pmcid: 4694698 doi: 10.1371/journal.pone.0145684
Gagnon, K. T., Li, L., Chu, Y., Janowski, B. A. & Corey, D. R. RNAi factors are present and active in human cell nuclei. Cell Rep. 6, 211–221 (2014).
pubmed: 24388755 pmcid: 3916906 doi: 10.1016/j.celrep.2013.12.013
Wang, S. et al. Endothelial cation channel PIEZO1 controls blood pressure by mediating flow-induced ATP release. J. Clin. Invest. 126, 4527–4536 (2016).
pubmed: 27797339 pmcid: 5127677 doi: 10.1172/JCI87343
Korff, T. & Augustin, H. G. Integration of endothelial cells in multicellular spheroids prevents apoptosis and induces differentiation. J. Cell Biol. 143, 1341–1352 (1998).
pubmed: 9832561 pmcid: 2133072 doi: 10.1083/jcb.143.5.1341
Giaever, I. & Keese, C. R. Micromotion of mammalian cells measured electrically. Proc. Natl Acad. Sci. USA 88, 7896–7900 (1991).
pubmed: 1881923 doi: 10.1073/pnas.88.17.7896
Cox, J. & Mann, M. MaxQuant enables high peptide identification rates, individualized p.p.b.-range mass accuracies and proteome-wide protein quantification. Nat. Biotechnol. 26, 1367–1372 (2008).
pubmed: 19029910 pmcid: 19029910 doi: 10.1038/nbt.1511
Tyanova, S. et al. The Perseus computational platform for comprehensive analysis of (prote)omics data. Nat. Methods 13, 731–740 (2016).
pubmed: 27348712 pmcid: 27348712 doi: 10.1038/nmeth.3901
Perez-Riverol, Y. et al. The PRIDE database and related tools and resources in 2019: improving support for quantification data. Nucleic Acids Res. 47, D442–D450 (2019).
pubmed: 30395289 pmcid: 30395289 doi: 10.1093/nar/gky1106
Kwon, H. B. et al. In vivo modulation of endothelial polarization by apelin receptor signalling. Nat. Commun. 7, 11805 (2016).
pubmed: 27248505 pmcid: 4895482 doi: 10.1038/ncomms11805

Auteurs

Laura Stanicek (L)

Dept. of Physiology, Amsterdam Cardiovascular Sciences (ACS), Amsterdam UMC, VU University Medical Center, Amsterdam, The Netherlands.
Institute of Cardiovascular Regeneration, Center of Molecular Medicine, Goethe-University, Frankfurt, Germany.

Noelia Lozano-Vidal (N)

Dept. of Physiology, Amsterdam Cardiovascular Sciences (ACS), Amsterdam UMC, VU University Medical Center, Amsterdam, The Netherlands.

Diewertje Ilse Bink (DI)

Dept. of Physiology, Amsterdam Cardiovascular Sciences (ACS), Amsterdam UMC, VU University Medical Center, Amsterdam, The Netherlands.

Aukie Hooglugt (A)

Dept. of Physiology, Amsterdam Cardiovascular Sciences (ACS), Amsterdam UMC, VU University Medical Center, Amsterdam, The Netherlands.
Department of Medical Biochemistry, Vascular Microenvironment and Integrity, Amsterdam Cardiovascular Sciences (ACS), Amsterdam University Medical Center, 1105 AZ, Amsterdam, The Netherlands.

Wenjie Yao (W)

Institute for Neurophysiology, Center for Molecular Medicine (CMMC), University of Cologne, Cologne, Germany.

Ilka Wittig (I)

Functional Proteomics, SFB 815 Core Unit, Faculty of Medicine, Goethe-University, Frankfurt, Germany.

Jos van Rijssel (J)

Molecular Cell Biology Laboratory, Department of Plasma Proteins, Sanquin Research and Landsteiner Laboratory, Academic Medical Center Amsterdam, University of Amsterdam, 1066 CX, Amsterdam, The Netherlands.

Jaap Diederik van Buul (JD)

Molecular Cell Biology Laboratory, Department of Plasma Proteins, Sanquin Research and Landsteiner Laboratory, Academic Medical Center Amsterdam, University of Amsterdam, 1066 CX, Amsterdam, The Netherlands.

Anke van Bergen (A)

Dept. of Physiology, Amsterdam Cardiovascular Sciences (ACS), Amsterdam UMC, VU University Medical Center, Amsterdam, The Netherlands.

Alina Klems (A)

Department of Cell and Developmental Biology, Institute of Zoology (ZOO), Karlsruhe Institute of Technology (KIT), Karlsruhe, Germany.

Anne Sophie Ramms (AS)

Department of Cell and Developmental Biology, Institute of Zoology (ZOO), Karlsruhe Institute of Technology (KIT), Karlsruhe, Germany.

Ferdinand Le Noble (F)

Department of Cell and Developmental Biology, Institute of Zoology (ZOO), Karlsruhe Institute of Technology (KIT), Karlsruhe, Germany.

Patrick Hofmann (P)

Institute of Cardiovascular Regeneration, Center of Molecular Medicine, Goethe-University, Frankfurt, Germany.
German Center for Cardiovascular Research DZHK, Partner Site Frankfurt Rhine-Main, Berlin, Germany.

Robert Szulcek (R)

Dept. of Pulmonary Diseases, Amsterdam Cardiovascular Sciences (ACS), Amsterdam UMC, VU University Medical Center, Amsterdam, The Netherlands.

ShengPeng Wang (S)

Department of Pharmacology, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Stefan Offermanns (S)

Department of Pharmacology, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Meryem Seda Ercanoglu (MS)

Institute of Virology, University Hospital Cologne, 50935, Cologne, Germany.
Center for Molecular Medicine Cologne (CMMC), University of Cologne, 50931, Cologne, Germany.

Hyouk-Bum Kwon (HB)

Department of Developmental Genetics, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Didier Stainier (D)

Department of Developmental Genetics, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Stephan Huveneers (S)

Department of Medical Biochemistry, Vascular Microenvironment and Integrity, Amsterdam Cardiovascular Sciences (ACS), Amsterdam University Medical Center, 1105 AZ, Amsterdam, The Netherlands.

Leo Kurian (L)

Institute for Neurophysiology, Center for Molecular Medicine (CMMC), University of Cologne, Cologne, Germany.

Stefanie Dimmeler (S)

Institute of Cardiovascular Regeneration, Center of Molecular Medicine, Goethe-University, Frankfurt, Germany.
German Center for Cardiovascular Research DZHK, Partner Site Frankfurt Rhine-Main, Berlin, Germany.

Reinier Abraham Boon (RA)

Dept. of Physiology, Amsterdam Cardiovascular Sciences (ACS), Amsterdam UMC, VU University Medical Center, Amsterdam, The Netherlands. r.a.boon@amsterdamumc.nl.
Institute of Cardiovascular Regeneration, Center of Molecular Medicine, Goethe-University, Frankfurt, Germany. r.a.boon@amsterdamumc.nl.
German Center for Cardiovascular Research DZHK, Partner Site Frankfurt Rhine-Main, Berlin, Germany. r.a.boon@amsterdamumc.nl.

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