Genotype analysis of ofloxacin-resistant multidrug-resistant


Journal

The Indian journal of medical research
ISSN: 0971-5916
Titre abrégé: Indian J Med Res
Pays: India
ID NLM: 0374701

Informations de publication

Date de publication:
04 2020
Historique:
entrez: 29 5 2020
pubmed: 29 5 2020
medline: 4 3 2021
Statut: ppublish

Résumé

Drug resistance surveillance offers useful information on trends of drug resistance and the efficacy of control measures. Studies and reports of drug-resistant mutations and phenotypic assays thus become important. This study was conducted to investigate the molecular characteristics of ofloxacin (OFX)-resistant, multidrug-resistant tuberculosis (MDR-TB) isolates from different geographical regions of India and their association with strains of different genotypes. Further, the nitrate reductase assay (NRA) was tested against Mycobacteria Growth Indicator Tube(MGIT) for the determination of OFX resistance as an alternative and cost-effective method. A total of 116 Mycobacterium tuberculosis isolates were used to assess the mutations in the gyrA, gyrB genes and resistance levels to OFX. Mutational analysis in gyrA and gyrB genes and genotype analysis of M. tuberculosis isolates was done by gene-specific polymerase chain reaction (PCR) followed by DNA sequencing and spoligotyping, respectively. Three (6.25%), 12 (44.44%) and 12 (29.27%) MDR-TB isolates from western, northern and southern India, respectively, were found to be OFX-resistant MDR-TB isolates. OFX resistance was observed to be significantly higher in MDR-TB cases for all study regions. Beijing genotypes from northern India were observed to be associated with OFX-resistant MDR-TB cases (P <0.05). Among 35 (30.15%) phenotypically OFX-resistant isolates, 22 (62.86%) had mutations in the gyrA gene and two (5.71%) isolates had mutations in the gyrB gene. These results caution against the PCR-based prediction of OFX resistance patterns and highlight the need for searching other genetic loci for the detection of mutations conferring resistance to OFX in M. tuberculosis. Our study also showed the usefulness of NRA as an alternative method to detect OFX resistance.

Sections du résumé

Background & objectives
Drug resistance surveillance offers useful information on trends of drug resistance and the efficacy of control measures. Studies and reports of drug-resistant mutations and phenotypic assays thus become important. This study was conducted to investigate the molecular characteristics of ofloxacin (OFX)-resistant, multidrug-resistant tuberculosis (MDR-TB) isolates from different geographical regions of India and their association with strains of different genotypes. Further, the nitrate reductase assay (NRA) was tested against Mycobacteria Growth Indicator Tube(MGIT) for the determination of OFX resistance as an alternative and cost-effective method.
Methods
A total of 116 Mycobacterium tuberculosis isolates were used to assess the mutations in the gyrA, gyrB genes and resistance levels to OFX. Mutational analysis in gyrA and gyrB genes and genotype analysis of M. tuberculosis isolates was done by gene-specific polymerase chain reaction (PCR) followed by DNA sequencing and spoligotyping, respectively.
Results
Three (6.25%), 12 (44.44%) and 12 (29.27%) MDR-TB isolates from western, northern and southern India, respectively, were found to be OFX-resistant MDR-TB isolates. OFX resistance was observed to be significantly higher in MDR-TB cases for all study regions. Beijing genotypes from northern India were observed to be associated with OFX-resistant MDR-TB cases (P <0.05). Among 35 (30.15%) phenotypically OFX-resistant isolates, 22 (62.86%) had mutations in the gyrA gene and two (5.71%) isolates had mutations in the gyrB gene.
Interpretation & conclusions
These results caution against the PCR-based prediction of OFX resistance patterns and highlight the need for searching other genetic loci for the detection of mutations conferring resistance to OFX in M. tuberculosis. Our study also showed the usefulness of NRA as an alternative method to detect OFX resistance.

Identifiants

pubmed: 32461400
pii: IndianJMedRes_2020_151_4_361_284218
doi: 10.4103/ijmr.IJMR_493_18
pmc: PMC7371067
doi:

Substances chimiques

Antitubercular Agents 0
Ofloxacin A4P49JAZ9H
Peptide Hydrolases EC 3.4.-
nitrate reductase inactivating enzyme EC 3.4.-

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

361-370

Déclaration de conflit d'intérêts

None

Références

Antimicrob Agents Chemother. 2014;58(4):1997-2005
pubmed: 24419342
J Antimicrob Chemother. 2009 Jun;63(6):1173-8
pubmed: 19329799
Indian J Med Res. 2016 Mar;143(3):341-7
pubmed: 27241648
Indian J Med Microbiol. 2017 Jan-Mar;35(1):69-73
pubmed: 28303821
J Clin Microbiol. 2002 Feb;40(2):553-5
pubmed: 11825971
Biomedica. 2015 Apr-Jun;35(2):285-91
pubmed: 26535551
J Clin Microbiol. 1999 Aug;37(8):2607-18
pubmed: 10405410
Emerg Infect Dis. 2007 Sep;13(9):1429-31
pubmed: 18252133
J Clin Microbiol. 2014 Aug;52(8):2876-80
pubmed: 24871222
Antimicrob Agents Chemother. 2004 Aug;48(8):3175-8
pubmed: 15273144
Indian J Med Res. 2012 Oct;136(4):599-604
pubmed: 23168700
J Antimicrob Chemother. 2012 Apr;67(4):819-31
pubmed: 22279180
Antimicrob Agents Chemother. 2009 Oct;53(10):4498-500
pubmed: 19687244
Antimicrob Agents Chemother. 2009 Nov;53(11):4835-9
pubmed: 19721073
J Infect Dev Ctries. 2010 Nov 24;4(11):695-703
pubmed: 21252446
J Clin Microbiol. 1993 Feb;31(2):406-9
pubmed: 8381814
J Health Popul Nutr. 2011 Feb;29(1):20-5
pubmed: 21528787
J Antibiot (Tokyo). 2015 Jan;68(1):63-6
pubmed: 25052485
Sci Rep. 2018 Jun 18;8(1):9295
pubmed: 29915257
J Med Microbiol. 2014 Apr;63(Pt 4):522-527
pubmed: 24445510
Mol Diagn Ther. 2009;13(3):137-51
pubmed: 19650669
Antimicrob Agents Chemother. 2014;58(1):364-9
pubmed: 24165186
PLoS One. 2013;8(2):e55299
pubmed: 23390524
J Antimicrob Chemother. 2014 Feb;69(2):441-4
pubmed: 24008825
Diagn Microbiol Infect Dis. 2010 Nov;68(3):236-40
pubmed: 20851548
PLoS One. 2015 Mar 04;10(3):e0117421
pubmed: 25738956

Auteurs

Anamika Gupta (A)

Division of Molecular Biology, ICMR-National AIDS Research Institute, Pune, Maharashtra, India.

Pallavi Sinha (P)

Department of Microbiology, Institute of Medical Sciences, Banaras Hindu University, Varanasi, Uttar Pradesh, India.

Sunita Rathod (S)

Division of Molecular Biology, ICMR-National AIDS Research Institute, Pune, Maharashtra, India.

Siva Kumar Shanmugam (SK)

Department of Bacteriology, ICMR-National Institute for Research in Tuberculosis, Chennai, Tamil Nadu, India.

K R Uma Devi (KR)

Department of Immunology, ICMR-National Institute for Research in Tuberculosis, Chennai, Tamil Nadu, India.

Shampa Anupurba (S)

Department of Microbiology, Institute of Medical Sciences, Banaras Hindu University, Varanasi, Uttar Pradesh, India.

Vijay Nema (V)

Division of Molecular Biology, ICMR-National AIDS Research Institute, Pune, Maharashtra, India.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH