Impaired cerebrovascular reactivity is associated with recurrent stroke in patients with severe intracranial arterial stenosis: A C02 BOLD fMRI study.

BOLD fMRI Cerebral vascular reactivity Cerebral vascular reserve Hypercapnia Intracranial arterial stenosis Stroke

Journal

Journal of neuroradiology = Journal de neuroradiologie
ISSN: 0150-9861
Titre abrégé: J Neuroradiol
Pays: France
ID NLM: 7705086

Informations de publication

Date de publication:
Sep 2021
Historique:
received: 06 03 2020
revised: 08 04 2020
accepted: 28 04 2020
pubmed: 30 5 2020
medline: 26 11 2021
entrez: 30 5 2020
Statut: ppublish

Résumé

Severe intracranial atherosclerotic stenosis (SIAS) remains at risk of recurrent ischemic events despite intensive medical management. Exhausted cerebrovascular reserve seems to be associated with higher risk of recurrent stroke. We used whole brain MRI to estimate basal perfusion using dynamic susceptibility contrast and cerebrovascular reactivity (CVR) to hypercapnic challenge (CO2 inhalation) using BOLD contrast, in 20 patients with symptomatic SIAS (>70%) of the middle cerebral artery (MCA) or the distal internal carotid artery. We studied relationships between individual clinical, biological, radiological baseline characteristics, recurrent ischemic events, basal perfusion parameters (mean transit time, delay, time to peak, cerebral blood flow and volume), and CVR measured in MCA territories (CVRMCA), and reported using laterality indices (LI). Ten patients had an impaired CVR with (|LI| CVRMCA≥0.08). During a mean follow-up of 3.3 years, all recurrent ipsilateral ischemic events occurred within the first year. They were more frequent in impaired CVRMCA group (n=7/10 patients) than in normal CVRMCA group (n=1/10), with different survival curves (log rank, P=0.007). Impaired CVR is associated with an increased rate of recurrent stroke in patients with symptomatic SIAS. CVR mapping should be used as a well tolerated method to select higher-risk patients in further therapeutic trials such as endovascular procedures.

Sections du résumé

BACKGROUND AND PURPOSE OBJECTIVE
Severe intracranial atherosclerotic stenosis (SIAS) remains at risk of recurrent ischemic events despite intensive medical management. Exhausted cerebrovascular reserve seems to be associated with higher risk of recurrent stroke.
MATERIALS AND METHODS METHODS
We used whole brain MRI to estimate basal perfusion using dynamic susceptibility contrast and cerebrovascular reactivity (CVR) to hypercapnic challenge (CO2 inhalation) using BOLD contrast, in 20 patients with symptomatic SIAS (>70%) of the middle cerebral artery (MCA) or the distal internal carotid artery. We studied relationships between individual clinical, biological, radiological baseline characteristics, recurrent ischemic events, basal perfusion parameters (mean transit time, delay, time to peak, cerebral blood flow and volume), and CVR measured in MCA territories (CVRMCA), and reported using laterality indices (LI).
RESULTS RESULTS
Ten patients had an impaired CVR with (|LI| CVRMCA≥0.08). During a mean follow-up of 3.3 years, all recurrent ipsilateral ischemic events occurred within the first year. They were more frequent in impaired CVRMCA group (n=7/10 patients) than in normal CVRMCA group (n=1/10), with different survival curves (log rank, P=0.007).
CONCLUSION CONCLUSIONS
Impaired CVR is associated with an increased rate of recurrent stroke in patients with symptomatic SIAS. CVR mapping should be used as a well tolerated method to select higher-risk patients in further therapeutic trials such as endovascular procedures.

Identifiants

pubmed: 32466863
pii: S0150-9861(20)30165-6
doi: 10.1016/j.neurad.2020.04.005
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

339-345

Informations de copyright

Copyright © 2020. Published by Elsevier Masson SAS.

Auteurs

Jérémie Papassin (J)

Stroke Unit, Grenoble Alps University Hospital, 38043 Grenoble, France; Stroke Unit, Metropole Savoie Hospital, 73000 Chambéry, France; University Grenoble Alps, Grenoble Institute of Neurosciences, 38042 Grenoble, France.

Olivier Heck (O)

Department of Neuroradiology, Grenoble Alps University Hospital, 38043 Grenoble, France.

Eric Condamine (E)

Inserm, CNRS, University Grenoble Alps, Grenoble Alps University Hospital, IRMaGe, CS 10217, 38043 Grenoble cedex 9, France.

Johan Pietras (J)

Inserm, CNRS, University Grenoble Alps, Grenoble Alps University Hospital, IRMaGe, CS 10217, 38043 Grenoble cedex 9, France.

Olivier Detante (O)

Stroke Unit, Grenoble Alps University Hospital, 38043 Grenoble, France; University Grenoble Alps, Grenoble Institute of Neurosciences, 38042 Grenoble, France.

Alexandre Krainik (A)

University Grenoble Alps, Grenoble Institute of Neurosciences, 38042 Grenoble, France; Department of Neuroradiology, Grenoble Alps University Hospital, 38043 Grenoble, France; Inserm, CNRS, University Grenoble Alps, Grenoble Alps University Hospital, IRMaGe, CS 10217, 38043 Grenoble cedex 9, France. Electronic address: akrainik@chu-grenoble.fr.

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