The Mouse Heart Mitochondria N Terminome Provides Insights into ClpXP-Mediated Proteolysis.


Journal

Molecular & cellular proteomics : MCP
ISSN: 1535-9484
Titre abrégé: Mol Cell Proteomics
Pays: United States
ID NLM: 101125647

Informations de publication

Date de publication:
08 2020
Historique:
received: 08 04 2020
revised: 24 05 2020
pubmed: 30 5 2020
medline: 28 5 2021
entrez: 30 5 2020
Statut: ppublish

Résumé

The mammalian mitochondrial proteome consists of more than 1100 annotated proteins and their proteostasis is regulated by only a few ATP-dependent protease complexes. Technical advances in protein mass spectrometry allowed for detailed description of the mitoproteome from different species and tissues and their changes under specific conditions. However, protease-substrate relations within mitochondria are still poorly understood. Here, we combined Terminal Amine Isotope Labeling of Substrates (TAILS) N termini profiling of heart mitochondria proteomes isolated from wild type and

Identifiants

pubmed: 32467259
pii: S1535-9476(20)34965-3
doi: 10.1074/mcp.RA120.002082
pmc: PMC8014998
pii:
doi:

Substances chimiques

Mitochondrial Proteins 0
Proteome 0
CLPP protein, mouse EC 3.4.21.92
Endopeptidase Clp EC 3.4.21.92
CLPX protein, mouse EC 3.6.1.3

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1330-1345

Informations de copyright

© 2020 Hofsetz et al.

Déclaration de conflit d'intérêts

Conflict of interest—Authors declare no competing interests.

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Auteurs

Eduard Hofsetz (E)

Institute for Mitochondrial Diseases and Aging at CECAD Research Centre, and Center for Molecular Medicine Cologne (CMMC), Medical Faculty, University of Cologne, Cologne, Germany; Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases (CECAD), Cologne, Germany, Medical Faculty and University Hospital, University of Cologne, Cologne, Germany.

Fatih Demir (F)

Central Institute for Engineering, Electronics and Analytics, ZEA-3, Forschungszentrum Jülich, Germany.

Karolina Szczepanowska (K)

Institute for Mitochondrial Diseases and Aging at CECAD Research Centre, and Center for Molecular Medicine Cologne (CMMC), Medical Faculty, University of Cologne, Cologne, Germany; Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases (CECAD), Cologne, Germany, Medical Faculty and University Hospital, University of Cologne, Cologne, Germany.

Alexandra Kukat (A)

Institute for Mitochondrial Diseases and Aging at CECAD Research Centre, and Center for Molecular Medicine Cologne (CMMC), Medical Faculty, University of Cologne, Cologne, Germany; Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases (CECAD), Cologne, Germany, Medical Faculty and University Hospital, University of Cologne, Cologne, Germany.

Jayachandran N Kizhakkedathu (JN)

Centre for Blood Research, School of Biomedical Engineering, Department of Pathology & Laboratory Medicine, Department of Chemistry, University of British Columbia, Vancouver, British Columbia, Canada.

Aleksandra Trifunovic (A)

Institute for Mitochondrial Diseases and Aging at CECAD Research Centre, and Center for Molecular Medicine Cologne (CMMC), Medical Faculty, University of Cologne, Cologne, Germany; Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases (CECAD), Cologne, Germany, Medical Faculty and University Hospital, University of Cologne, Cologne, Germany. Electronic address: aleksandra.trifunovic@uk-koeln.de.

Pitter F Huesgen (PF)

Cologne Excellence Cluster on Cellular Stress Responses in Aging Associated Diseases (CECAD), Cologne, Germany, Medical Faculty and University Hospital, University of Cologne, Cologne, Germany; Central Institute for Engineering, Electronics and Analytics, ZEA-3, Forschungszentrum Jülich, Germany; Institute for Biochemistry, Faculty of Mathematics and Natural Sciences, University of Cologne, Cologne, Germany. Electronic address: p.huesgen@fz-juelich.de.

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Classifications MeSH