CD-1
Diabetic kidney disease
Fibrosis
N-acetyl-seryl-aspartyl-lysyl-proline
Journal
Journal of diabetes investigation
ISSN: 2040-1124
Titre abrégé: J Diabetes Investig
Pays: Japan
ID NLM: 101520702
Informations de publication
Date de publication:
Nov 2020
Nov 2020
Historique:
received:
24
12
2019
revised:
28
03
2020
accepted:
11
05
2020
pubmed:
31
5
2020
medline:
20
8
2021
entrez:
31
5
2020
Statut:
ppublish
Résumé
To establish novel therapies to combat diabetic kidney disease, a human disease-relevant animal model is essential. However, a type 2 diabetic mouse model presenting progressive kidney fibrosis has not yet been established. Kidneys of streptozotocin-induced diabetic CD-1 mice showed severe fibrosis compared with other backgrounds of mice associated with the suppression of antifibrotic peptide N-acetyl-seryl-aspartyl-lysyl-proline. The BKS background (BKS We generated CD-1 Male CD-1 These results suggest that CD-1
Identifiants
pubmed: 32472621
doi: 10.1111/jdi.13311
pmc: PMC7610117
doi:
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
1470-1481Subventions
Organisme : Japan Society for the Promotion of Science
ID : 19K08738
Organisme : Japan Society for the Promotion of Science
ID : 26460403
Organisme : Japan Society for the Promotion of Science
ID : 25282028
Organisme : Japan Society for the Promotion of Science
ID : 25670414
Organisme : Grant for Promoted Research
ID : S2016-3
Organisme : Grant for Promoted Research
ID : S2017-1
Organisme : Kanazawa Medical University
ID : K2017-16
Informations de copyright
© 2020 The Authors. Journal of Diabetes Investigation published by Asian Association for the Study of Diabetes (AASD) and John Wiley & Sons Australia, Ltd.
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