The risk of hip and non-vertebral fractures in type 1 and type 2 diabetes: A systematic review and meta-analysis update.


Journal

Bone
ISSN: 1873-2763
Titre abrégé: Bone
Pays: United States
ID NLM: 8504048

Informations de publication

Date de publication:
08 2020
Historique:
received: 24 04 2020
revised: 22 05 2020
accepted: 25 05 2020
pubmed: 2 6 2020
medline: 22 6 2021
entrez: 2 6 2020
Statut: ppublish

Résumé

Diabetes is associated with increased fracture risk but we do not know what affects this risk. We investigated the risk of hip and non-vertebral fractures in diabetes and whether this risk was affected by age, gender, body mass index, diabetes type and duration, insulin use and diabetic complications. We selected a previously published review to be updated. MEDLINE, Embase and Cochrane databases were searched up to March 2020. We included observational studies with age and gender-adjusted risk of fractures in adults with diabetes compared to adults without diabetes. We extracted data from published reports that we summarised using random effects model. From the 3140 records identified, 49 were included, 42 in the hip fracture analysis, reporting data from 17,571,738 participants with 319,652 fractures and 17 in the non-vertebral fracture review, reporting data from 2,978,487 participants with 181,228 fractures. We found an increase in the risk of fracture in diabetes both for hip (RR 4.93, 3.06-7.95, in type 1 diabetes and RR1.33, 1.19-1.49, in type 2 diabetes) and for non-vertebral fractures (RR 1.92, 0.92-3.99, in type 1 and RR 1.19, 1,11-1.28 in type 2). At the hip, the risk was higher in the younger population in both type 1 and type 2 diabetes. In those with type 2 diabetes, longer diabetes duration and insulin use was associated with an increased risk. We did not investigate the effect of bone density, falls, anti-diabetic drugs and hypoglycemia. Diabetes is associated with an increase in both hip and non-vertebral fracture risk.

Sections du résumé

BACKGROUND
Diabetes is associated with increased fracture risk but we do not know what affects this risk. We investigated the risk of hip and non-vertebral fractures in diabetes and whether this risk was affected by age, gender, body mass index, diabetes type and duration, insulin use and diabetic complications.
METHODS
We selected a previously published review to be updated. MEDLINE, Embase and Cochrane databases were searched up to March 2020. We included observational studies with age and gender-adjusted risk of fractures in adults with diabetes compared to adults without diabetes. We extracted data from published reports that we summarised using random effects model.
FINDINGS
From the 3140 records identified, 49 were included, 42 in the hip fracture analysis, reporting data from 17,571,738 participants with 319,652 fractures and 17 in the non-vertebral fracture review, reporting data from 2,978,487 participants with 181,228 fractures. We found an increase in the risk of fracture in diabetes both for hip (RR 4.93, 3.06-7.95, in type 1 diabetes and RR1.33, 1.19-1.49, in type 2 diabetes) and for non-vertebral fractures (RR 1.92, 0.92-3.99, in type 1 and RR 1.19, 1,11-1.28 in type 2). At the hip, the risk was higher in the younger population in both type 1 and type 2 diabetes. In those with type 2 diabetes, longer diabetes duration and insulin use was associated with an increased risk. We did not investigate the effect of bone density, falls, anti-diabetic drugs and hypoglycemia.
CONCLUSION
Diabetes is associated with an increase in both hip and non-vertebral fracture risk.

Identifiants

pubmed: 32480023
pii: S8756-3282(20)30237-4
doi: 10.1016/j.bone.2020.115457
pii:
doi:

Types de publication

Journal Article Meta-Analysis Research Support, Non-U.S. Gov't Systematic Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

115457

Subventions

Organisme : Medical Research Council
ID : MR/P020941/1
Pays : United Kingdom

Informations de copyright

Copyright © 2020. Published by Elsevier Inc.

Déclaration de conflit d'intérêts

Declaration of competing interest TV received grant funding from Amgen Inc. and personal support from Conselho Nacional de Desenvolvimento Científico e Tecnológico - CNPq- Brazil. MS received grant funding from Amgen Inc. and personal fees from the Centre For Integrated Research Into Musculoskeletal Ageing (CIMA) and Osteoporosis 2000. SH received grant funding from Amgen Inc. EP received grant funding from Amgen Inc. AS received grant funding from Amgen Inc. SC received consultancy and grant funding from Amgen Inc. IA received grant funding from Amgen Inc. RE received consultancy funding from Immunodiagnostic Systems, Roche Diagnostics, GSK Nutrition, FNIH, Mereo, Lilly, Sandoz, Nittobo, Abbvie, Samsung, Haoma Medica and grant funding from Nittobo, Immunodiagnostic Systems, Roche Diagnostics, Amgen Inc. and Alexion.

Auteurs

Tatiane Vilaca (T)

Academic Unit of Bone Metabolism, The Mellanby Centre for Bone Research, University of Sheffield, UK. Electronic address: tvilaca1@sheffield.ac.uk.

Marian Schini (M)

Academic Unit of Bone Metabolism, The Mellanby Centre for Bone Research, University of Sheffield, UK. Electronic address: m.schini@sheffield.ac.uk.

Susan Harnan (S)

School of Health and Related Research (ScHARR), University of Sheffield, UK. Electronic address: s.harnan@sheffield.ac.uk.

Anthea Sutton (A)

School of Health and Related Research (ScHARR), University of Sheffield, UK. Electronic address: a.sutton@sheffield.ac.uk.

Edith Poku (E)

School of Health and Related Research (ScHARR), University of Sheffield, UK. Electronic address: e.poku@sheffield.ac.uk.

Isabel E Allen (IE)

University of California, San Francisco, USA. Electronic address: isabel.allen@ucsf.edu.

Steven R Cummings (SR)

San Francisco Coordinating Center, Sutter Health, University of California, San Francisco, USA. Electronic address: SCummings@sfcc-cpmc.net.

Richard Eastell (R)

Academic Unit of Bone Metabolism, The Mellanby Centre for Bone Research, University of Sheffield, UK. Electronic address: r.eastell@sheffield.ac.uk.

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