Chronic administration of methylphenidate did not affect memory and GDNF levels but increase astrogliosis in adult male rat's hippocampus.


Journal

Journal of chemical neuroanatomy
ISSN: 1873-6300
Titre abrégé: J Chem Neuroanat
Pays: Netherlands
ID NLM: 8902615

Informations de publication

Date de publication:
10 2020
Historique:
received: 01 03 2020
revised: 02 05 2020
accepted: 27 05 2020
pubmed: 3 6 2020
medline: 7 4 2021
entrez: 3 6 2020
Statut: ppublish

Résumé

ADHD is the most common developmental disorder affecting approximately three to seven percent of school-aged children and 2.5 percent of adults worldwide. The drug of choice for the pharmacotherapy of ADHD is Methylphenidate (MPH). However, there is growing concerns about side effects resulting from its potential interference with brain anatomical and behavioral development. This article focuses on the adverse effects of MPH on the rat's hippocampus. The animals received an oral dose of 5 mg/kg MPH or normal saline, as the vehicle, on a daily basis for 30 days. Y-maze test, passive avoidance, Barnes maze and field potential recording were conducted. Western blot for detecting the neurotrophic factor of GDNF and immunohistochemistry of astrogliosis were performed. Our results revealed that MPH treatment suppressed the willingness of rats to explore new environments. Also, it had no effect on improving long-term potentiation, long-term memory and spatial memory in the MPH group as opposed to the control group. There was also a significant increase of astrogliosis in the treated rats' hippocampi. On the other hand, there was not a significant relationship between MPH administration and the decrement of the GDNF level. We encourage the need to conduct more research on the adverse effects of MPH on the brain.

Sections du résumé

BACKGROUND
ADHD is the most common developmental disorder affecting approximately three to seven percent of school-aged children and 2.5 percent of adults worldwide. The drug of choice for the pharmacotherapy of ADHD is Methylphenidate (MPH). However, there is growing concerns about side effects resulting from its potential interference with brain anatomical and behavioral development.
AIM
This article focuses on the adverse effects of MPH on the rat's hippocampus.
METHODS
The animals received an oral dose of 5 mg/kg MPH or normal saline, as the vehicle, on a daily basis for 30 days. Y-maze test, passive avoidance, Barnes maze and field potential recording were conducted. Western blot for detecting the neurotrophic factor of GDNF and immunohistochemistry of astrogliosis were performed.
RESULTS
Our results revealed that MPH treatment suppressed the willingness of rats to explore new environments. Also, it had no effect on improving long-term potentiation, long-term memory and spatial memory in the MPH group as opposed to the control group. There was also a significant increase of astrogliosis in the treated rats' hippocampi. On the other hand, there was not a significant relationship between MPH administration and the decrement of the GDNF level.
CONCLUSION
We encourage the need to conduct more research on the adverse effects of MPH on the brain.

Identifiants

pubmed: 32485223
pii: S0891-0618(20)30087-9
doi: 10.1016/j.jchemneu.2020.101818
pii:
doi:

Substances chimiques

Central Nervous System Stimulants 0
Glial Cell Line-Derived Neurotrophic Factor 0
Methylphenidate 207ZZ9QZ49

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

101818

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors have no conflict of interest to declare.

Auteurs

Gholam Hossein Meftahi (GH)

Neuroscience Research Center, Baqiyatallah University of Medical Sciences, Tehran, Iran.

Maral Moafi (M)

Hearing Disorders Research Center, Loghman Hakim Hospital, Shahid Beheshti University of Medical Sciences, Iran University of Medical Sciences, Tehran, Iran.

Seyed Hamidreza Mirbehbahani (SH)

Neuroscience Research Center, Shahid Beheshti University of Medical Sciences, Tehran, Iran.

Farid Fotouhi (F)

Hearing Disorders Research Center, Loghman Hakim Hospital, Shahid Beheshti University of Medical Sciences, Iran University of Medical Sciences, Tehran, Iran.

Hossein Toreyhi (H)

Hearing Disorders Research Center, Loghman Hakim Hospital, Shahid Beheshti University of Medical Sciences, Iran University of Medical Sciences, Tehran, Iran.

Samira Ezi (S)

Department of Cell Biology and Anatomical Sciences, School of Medicine, Shahid Beheshti University of Medical Sciences, Tehran, Iran.

Fakhroddin Aghajanpour (F)

Department of Cell Biology and Anatomical Sciences, School of Medicine, Shahid Beheshti University of Medical Sciences, Tehran, Iran.

Ali Forouzannia (A)

Hearing Disorders Research Center, Loghman Hakim Hospital, Shahid Beheshti University of Medical Sciences, Iran University of Medical Sciences, Tehran, Iran.

Mahdi Eskandarian Boroujeni (ME)

Department of Human Molecular Genetics, Institute of Molecular Biology and Biotechnology, Faculty of Biology, Adam Mickiewicz University, Poznan, Poland.

Tahmineh Peirouvi (T)

Department of Histology, School of Medicine, Urmia University of Medical Sciences, Iran. Electronic address: tpeirouvi@yahoo.co.uk.

Hojjat Allah Abbaszadeh (HA)

Hearing Disorders Research Center, Loghman Hakim Hospital, Shahid Beheshti University of Medical Sciences, Iran University of Medical Sciences, Tehran, Iran.

Abbas Aliaghaei (A)

Hearing Disorders Research Center, Loghman Hakim Hospital, Shahid Beheshti University of Medical Sciences, Iran University of Medical Sciences, Tehran, Iran; Department of Cell Biology and Anatomical Sciences, School of Medicine, Shahid Beheshti University of Medical Sciences, Tehran, Iran. Electronic address: aghaei60@gmail.com.

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Classifications MeSH