Acute symptomatic seizures following intracerebral hemorrhage in the rat collagenase model.


Journal

Epilepsy research
ISSN: 1872-6844
Titre abrégé: Epilepsy Res
Pays: Netherlands
ID NLM: 8703089

Informations de publication

Date de publication:
08 2020
Historique:
received: 30 01 2020
revised: 22 04 2020
accepted: 05 05 2020
pubmed: 5 6 2020
medline: 1 10 2021
entrez: 5 6 2020
Statut: ppublish

Résumé

Intracerebral hemorrhage (ICH) is a known risk factor for the development of seizures, but little is known about the pathophysiology of seizures in the acute phase post-ICH and their influence on functional outcome. With the use of an animal model, the underlying pathophysiology could be further unraveled. The aim of our study was to optimize the rat collagenase stroke model for the detection of acute symptomatic seizures using video-EEG monitoring. Male Sprague-Dawley rats were implanted with scalp electrodes and a craniotomy was made for later injection of collagenase. After one week of baseline video-EEG recording, rats were injected with 0.6 U collagenase in 0.7 μL saline in left striatum, in close proximity of the piriform cortex, and immediately reconnected to the video-EEG setup for 7 days. Occurrence of clinical and electrographic seizures was assessed and functional deficits were evaluated on several time points using the cylinder test, Neurological Deficit Scale (NDS) and forelimb placing test. At day 7 post-ICH, animals were euthanized. The volume and cortical involvement of the hemorrhage were assessed by histological examination of the brain tissue, using Cresyl violet stain. Collagenase injection induced ICH in all animals with a mean volume of 27 mm³ (SEM 7 mm³, range 4-92 mm³). Functional deficits were present in all animals injected with collagenase (pre-ICH vs post-ICH, p < 0.001). Epileptic seizures occurred in 5/11 animals and started between 1 and 61 h after ICH induction. Behavioral changes were observed in 13/15 seizures. Injecting rats with 0.6 U of collagenase is a useful model to study the occurrence of acute symptomatic seizures post-ICH as it results in ICH in all animals without mortality, 45% incidence of ICH-induced acute symptomatic seizures and measurable functional deficits.

Sections du résumé

BACKGROUND AND PURPOSE
Intracerebral hemorrhage (ICH) is a known risk factor for the development of seizures, but little is known about the pathophysiology of seizures in the acute phase post-ICH and their influence on functional outcome. With the use of an animal model, the underlying pathophysiology could be further unraveled. The aim of our study was to optimize the rat collagenase stroke model for the detection of acute symptomatic seizures using video-EEG monitoring.
METHODS
Male Sprague-Dawley rats were implanted with scalp electrodes and a craniotomy was made for later injection of collagenase. After one week of baseline video-EEG recording, rats were injected with 0.6 U collagenase in 0.7 μL saline in left striatum, in close proximity of the piriform cortex, and immediately reconnected to the video-EEG setup for 7 days. Occurrence of clinical and electrographic seizures was assessed and functional deficits were evaluated on several time points using the cylinder test, Neurological Deficit Scale (NDS) and forelimb placing test. At day 7 post-ICH, animals were euthanized. The volume and cortical involvement of the hemorrhage were assessed by histological examination of the brain tissue, using Cresyl violet stain.
RESULTS
Collagenase injection induced ICH in all animals with a mean volume of 27 mm³ (SEM 7 mm³, range 4-92 mm³). Functional deficits were present in all animals injected with collagenase (pre-ICH vs post-ICH, p < 0.001). Epileptic seizures occurred in 5/11 animals and started between 1 and 61 h after ICH induction. Behavioral changes were observed in 13/15 seizures.
CONCLUSIONS
Injecting rats with 0.6 U of collagenase is a useful model to study the occurrence of acute symptomatic seizures post-ICH as it results in ICH in all animals without mortality, 45% incidence of ICH-induced acute symptomatic seizures and measurable functional deficits.

Identifiants

pubmed: 32497986
pii: S0920-1211(20)30064-4
doi: 10.1016/j.eplepsyres.2020.106364
pii:
doi:

Substances chimiques

Collagenases EC 3.4.24.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

106364

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Charlotte Germonpré (C)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Silke Proesmans (S)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Charlotte Bouckaert (C)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Latoya Stevens (L)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Mathieu Sprengers (M)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Kristl Vonck (K)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Evelien Carrette (E)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Wytse Wadman (W)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Paul Boon (P)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Robrecht Raedt (R)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium.

Veerle De Herdt (V)

4BRAIN, Department of Neurology, Ghent University Hospital, Ghent, Belgium. Electronic address: veerle.deherdt@uzgent.be.

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