Potential pathophysiological mechanisms leading to increased COVID-19 susceptibility and severity in obesity.

Angiotensin converting enzyme 2 Coronavirus Cytokines Obesity Vitamin D

Journal

Obesity medicine
ISSN: 2451-8476
Titre abrégé: Obes Med
Pays: England
ID NLM: 101690300

Informations de publication

Date de publication:
Sep 2020
Historique:
received: 16 05 2020
revised: 18 05 2020
accepted: 21 05 2020
entrez: 6 6 2020
pubmed: 6 6 2020
medline: 6 6 2020
Statut: ppublish

Résumé

As we are facing worldwide pandemic of COVID-19, we aimed to identify potential pathophysiological mechanisms leading to increased COVID-19 susceptibility and severity in obesity. Special emphasis will be given on increased susceptibility to infections due to obesity-related low-grade chronic inflammation, higher expression of angiotensin converting enzyme-2 and pathway-associated components, as well as decreased vitamin D bioavailability, since all of them provide easier ways for the virus to enter into host cells, replicate and stunt adequate immune responses.

Identifiants

pubmed: 32501427
doi: 10.1016/j.obmed.2020.100259
pii: S2451-8476(20)30079-8
pii: 100259
pmc: PMC7255205
doi:

Types de publication

Journal Article

Langues

eng

Pagination

100259

Informations de copyright

© 2020 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declare no conflict of interest.

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Auteurs

Andrej Belančić (A)

Department of Clinical Pharmacology, University Hospital Centre Rijeka, Krešimirova 42, 51000, Rijeka, Croatia.

Andrea Kresović (A)

Department of Gastroenterology, University Hospital Centre Rijeka, Krešimirova 42, 51000, Rijeka, Croatia.

Valentino Rački (V)

Department of Neurology, University of Rijeka, Faculty of Medicine, Braće Branchetta 20, 51000, Rijeka, Croatia.

Classifications MeSH