Myocyte-specific enhancer factor 2D promotes tumorigenesis and progression in tongue squamous cell carcinoma.

Tongue squamous cell carcinoma (TSCC) myocyte-specific enhancer factor 2 (MEF2D) proliferation tumorigenesis

Journal

International journal of clinical and experimental pathology
ISSN: 1936-2625
Titre abrégé: Int J Clin Exp Pathol
Pays: United States
ID NLM: 101480565

Informations de publication

Date de publication:
2020
Historique:
received: 21 10 2019
accepted: 25 03 2020
entrez: 9 6 2020
pubmed: 9 6 2020
medline: 9 6 2020
Statut: epublish

Résumé

Tongue squamous cell carcinoma (TSCC) ranks as one of the most common cancers worldwide and has a poor prognosis. Myocyte-specific enhancer factor 2 (MEF2D) has recently been considered as a novel factor involved in cancer development. In the present study, the function and underlying mechanism of MEF2D in TSCC were investigated. The levels of MEF2D mRNA and protein were determined in human TSCC samples by RT-qPCR and western blot, respectively. The interaction between MEF2D expression and clinicopathologic features was evaluated by IHC and analysis of clinical information. MEF2D-mediated effects on proliferation, migration, and invasion were explored in TSCC cells after transfection with MEF2D-siRNA. The results showed higher expression of MEF2D at both the mRNA and protein levels in TSCC carcinoma tissues than in paracarcinoma tissues. Furthermore, high expression of MEF2D was positively correlated with tumor differentiation and lymphatic metastasis. MEF2D knocked down TSCCA cells also had reduced proliferative, migratory, and invasive abilities compared to those of control cells. Together, these data confirmed that knockdown of MEF2D might suppress the growth and metastasis of TSCC, which further indicated that MEF2D might serve as a therapeutic target for TSCC treatment.

Identifiants

pubmed: 32509064
pmc: PMC7270656

Types de publication

Journal Article

Langues

eng

Pagination

934-943

Informations de copyright

IJCEP Copyright © 2020.

Déclaration de conflit d'intérêts

None.

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Auteurs

Yang Liu (Y)

Department of Oncology, Qingdao Municipal Hospital Qingdao, China.

Wenjun Yu (W)

Department of Oncology, Qingdao Municipal Hospital Qingdao, China.

Yan Zhu (Y)

Department of Oncology, Qingdao Municipal Hospital Qingdao, China.

Zhenni Sun (Z)

Department of Oncology, Qingdao Municipal Hospital Qingdao, China.

Xingang Huang (X)

Department of Pathology, Qingdao Municipal Hospital Qingdao, China.

Jianhua Zhou (J)

Department of Stomatology, Qingdao Municipal Hospital Qingdao, China.

Ruyong Yao (R)

Central Laboratory, The Affiliated Hospital of Qingdao University Qingdao 266001, China.

Qian Zhang (Q)

Central Laboratory, The Affiliated Hospital of Qingdao University Qingdao 266001, China.

Jianzhong Qiu (J)

Department of Stomatology, Qingdao Municipal Hospital Qingdao, China.

Lu Yue (L)

Department of Oncology, Qingdao Municipal Hospital Qingdao, China.

Classifications MeSH