In vitro interaction of isavuconazole and anidulafungin against azole-susceptible and azole-resistant Aspergillus fumigatus isolates.


Journal

The Journal of antimicrobial chemotherapy
ISSN: 1460-2091
Titre abrégé: J Antimicrob Chemother
Pays: England
ID NLM: 7513617

Informations de publication

Date de publication:
01 09 2020
Historique:
received: 25 11 2019
revised: 24 02 2020
accepted: 15 04 2020
pubmed: 10 6 2020
medline: 25 6 2021
entrez: 10 6 2020
Statut: ppublish

Résumé

The voriconazole and echinocandin combination has been found to be synergistic in vitro and in vivo against most Aspergillus fumigatus isolates, both with a WT azole phenotype and an azole-resistant phenotype. The interaction between isavuconazole and echinocandins is less well studied. This is especially true for azole-resistant isolates. We investigated the in vitro interaction between isavuconazole and anidulafungin for 30 A. fumigatus isolates including 18 azole-resistant isolates with various isavuconazole resistance phenotypes. The isavuconazole/anidulafungin interaction was studied by using an adapted EUCAST-based 2D (12 × 8) chequerboard broth microdilution colorimetric assay using XTT. The interaction was analysed by FIC index (FICi) analysis and Bliss independence (BI) interaction analysis. Both the FICi analysis and the BI analysis showed synergistic interaction between isavuconazole and anidulafungin for the majority of WT and azole-resistant isolates. As we did not see significant beneficial effects of combination therapy in TR46/Y121F/T289A isolates at clinically achievable drug concentrations, it is unlikely that TR46/Y121F/T289A infections would benefit from isavuconazole and anidulafungin combination therapy. In regions with high azole resistance rates this combination may benefit patients with WT disease, azole-resistant invasive aspergillosis and those with mixed azole-susceptible and azole-resistant infection, but may not be beneficial for aspergillosis due to isolates with high isavuconazole resistance, such as TR46/Y121F/T289A isolates.

Sections du résumé

BACKGROUND
The voriconazole and echinocandin combination has been found to be synergistic in vitro and in vivo against most Aspergillus fumigatus isolates, both with a WT azole phenotype and an azole-resistant phenotype. The interaction between isavuconazole and echinocandins is less well studied. This is especially true for azole-resistant isolates.
OBJECTIVES
We investigated the in vitro interaction between isavuconazole and anidulafungin for 30 A. fumigatus isolates including 18 azole-resistant isolates with various isavuconazole resistance phenotypes.
METHODS
The isavuconazole/anidulafungin interaction was studied by using an adapted EUCAST-based 2D (12 × 8) chequerboard broth microdilution colorimetric assay using XTT. The interaction was analysed by FIC index (FICi) analysis and Bliss independence (BI) interaction analysis.
RESULTS
Both the FICi analysis and the BI analysis showed synergistic interaction between isavuconazole and anidulafungin for the majority of WT and azole-resistant isolates. As we did not see significant beneficial effects of combination therapy in TR46/Y121F/T289A isolates at clinically achievable drug concentrations, it is unlikely that TR46/Y121F/T289A infections would benefit from isavuconazole and anidulafungin combination therapy.
CONCLUSIONS
In regions with high azole resistance rates this combination may benefit patients with WT disease, azole-resistant invasive aspergillosis and those with mixed azole-susceptible and azole-resistant infection, but may not be beneficial for aspergillosis due to isolates with high isavuconazole resistance, such as TR46/Y121F/T289A isolates.

Identifiants

pubmed: 32516368
pii: 5855124
doi: 10.1093/jac/dkaa185
pmc: PMC7443724
doi:

Substances chimiques

Antifungal Agents 0
Azoles 0
Fungal Proteins 0
Nitriles 0
Pyridines 0
Triazoles 0
isavuconazole 60UTO373KE
Anidulafungin 9HLM53094I

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2582-2586

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press on behalf of the British Society for Antimicrobial Chemotherapy.

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Auteurs

J B Buil (JB)

Department of Medical Microbiology, Radboud University Medical Center, Nijmegen, The Netherlands.
Center of Expertise in Mycology Radboudumc/CWZ, Nijmegen, The Netherlands.

R J M Brüggemann (RJM)

Center of Expertise in Mycology Radboudumc/CWZ, Nijmegen, The Netherlands.
Department of Pharmacy, Radboud University Medical Center, Nijmegen, The Netherlands.

L Bedin Denardi (L)

Programa de Pós-Graduação em Ciências Farmacêuticas, Centro de Ciências da Saúde, Universidade Federal de Santa Maria, Santa Maria, RS, Brasil.

W J G Melchers (WJG)

Department of Medical Microbiology, Radboud University Medical Center, Nijmegen, The Netherlands.
Center of Expertise in Mycology Radboudumc/CWZ, Nijmegen, The Netherlands.

P E Verweij (PE)

Department of Medical Microbiology, Radboud University Medical Center, Nijmegen, The Netherlands.
Center of Expertise in Mycology Radboudumc/CWZ, Nijmegen, The Netherlands.

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Classifications MeSH