Atezolizumab, vemurafenib, and cobimetinib as first-line treatment for unresectable advanced BRAF


Journal

Lancet (London, England)
ISSN: 1474-547X
Titre abrégé: Lancet
Pays: England
ID NLM: 2985213R

Informations de publication

Date de publication:
13 06 2020
Historique:
received: 02 03 2020
revised: 02 04 2020
accepted: 14 04 2020
entrez: 15 6 2020
pubmed: 15 6 2020
medline: 24 6 2020
Statut: ppublish

Résumé

IMspire150 aimed to evaluate first-line combination treatment with BRAF plus MEK inhibitors and immune checkpoint therapy in BRAF IMspire150 was a randomised, double-blind, placebo-controlled phase 3 study done at 112 institutes in 20 countries. Patients with unresectable stage IIIc-IV, BRAF Between Jan 13, 2017, and April 26, 2018, 777 patients were screened and 514 were enrolled and randomly assigned to the atezolizumab group (n=256) or control group (n=258). At a median follow-up of 18·9 months (IQR 10·4-23·8), progression-free survival as assessed by the study investigator was significantly prolonged with atezolizumab versus control (15·1 vs 10·6 months; hazard ratio [HR] 0·78; 95% CI 0·63-0·97; p=0·025). Common treatment-related adverse events (>30%) in the atezolizumab and control groups were blood creatinine phosphokinase increased (51·3% vs 44·8%), diarrhoea (42·2% vs 46·6%), rash (40·9%, both groups), arthralgia (39·1% vs 28·1%), pyrexia (38·7% vs 26·0%), alanine aminotransferase increased (33·9% vs 22·8%), and lipase increased (32·2% vs 27·4%); 13% of patients in the atezolizumab group and 16% in the control group stopped all treatment because of adverse events. The addition of atezolizumab to targeted therapy with vemurafenib and cobimetinib was safe and tolerable and significantly increased progression-free survival in patients with BRAF F Hoffmann-La Roche and Genentech.

Sections du résumé

BACKGROUND
IMspire150 aimed to evaluate first-line combination treatment with BRAF plus MEK inhibitors and immune checkpoint therapy in BRAF
METHODS
IMspire150 was a randomised, double-blind, placebo-controlled phase 3 study done at 112 institutes in 20 countries. Patients with unresectable stage IIIc-IV, BRAF
FINDINGS
Between Jan 13, 2017, and April 26, 2018, 777 patients were screened and 514 were enrolled and randomly assigned to the atezolizumab group (n=256) or control group (n=258). At a median follow-up of 18·9 months (IQR 10·4-23·8), progression-free survival as assessed by the study investigator was significantly prolonged with atezolizumab versus control (15·1 vs 10·6 months; hazard ratio [HR] 0·78; 95% CI 0·63-0·97; p=0·025). Common treatment-related adverse events (>30%) in the atezolizumab and control groups were blood creatinine phosphokinase increased (51·3% vs 44·8%), diarrhoea (42·2% vs 46·6%), rash (40·9%, both groups), arthralgia (39·1% vs 28·1%), pyrexia (38·7% vs 26·0%), alanine aminotransferase increased (33·9% vs 22·8%), and lipase increased (32·2% vs 27·4%); 13% of patients in the atezolizumab group and 16% in the control group stopped all treatment because of adverse events.
INTERPRETATION
The addition of atezolizumab to targeted therapy with vemurafenib and cobimetinib was safe and tolerable and significantly increased progression-free survival in patients with BRAF
FUNDING
F Hoffmann-La Roche and Genentech.

Identifiants

pubmed: 32534646
pii: S0140-6736(20)30934-X
doi: 10.1016/S0140-6736(20)30934-X
pii:
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
Azetidines 0
Piperidines 0
Vemurafenib 207SMY3FQT
atezolizumab 52CMI0WC3Y
BRAF protein, human EC 2.7.11.1
Proto-Oncogene Proteins B-raf EC 2.7.11.1
cobimetinib ER29L26N1X

Banques de données

ClinicalTrials.gov
['NCT02908672']

Types de publication

Clinical Trial, Phase III Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1835-1844

Commentaires et corrections

Type : CommentIn
Type : ErratumIn

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Auteurs

Ralf Gutzmer (R)

Haut-Tumor-Zentrum Hannover, Klinik für Dermatologie, Allergologie und Venerologie, Medizinische Hochschule Hannover, Hannover, Germany. Electronic address: gutzmer.ralf@mh-hannover.de.

Daniil Stroyakovskiy (D)

Moscow City Oncology Hospital Number 62 of Moscow Healthcare Department, Moscow, Russia.

Helen Gogas (H)

First Department of Medicine, Laiko General Hospital, National and Kapodistrian University of Athens, Greece.

Caroline Robert (C)

Gustave Roussy and Université Paris-Saclay, Villejuif-Paris, France.

Karl Lewis (K)

University of Colorado Comprehensive Cancer Center, Aurora, CO, USA.

Svetlana Protsenko (S)

Department of Chemotherapy and Innovative Technologies, NN Petrov National Medical Research Center of Oncology, St Petersburg, Russia.

Rodrigo P Pereira (RP)

Hospital de Clínicas de Porto Alegre, Porto Alegre, Brazil.

Thomas Eigentler (T)

University Hospital Tübingen, Tübingen, Germany.

Piotr Rutkowski (P)

Department of Soft Tissue-Bone Sarcoma and Melanoma, Maria Sklodowska-Curie National Research Institute of Oncology, Warsaw, Poland.

Lev Demidov (L)

NN Blokhin Russian Cancer Research Center, Ministry of Health, Moscow, Russia.

Georgy Moiseevich Manikhas (GM)

St Petersburg Oncology Hospital, St Petersburg, Russia.

Yibing Yan (Y)

Genentech, South San Francisco, CA, USA.

Kuan-Chieh Huang (KC)

Genentech, South San Francisco, CA, USA.

Anne Uyei (A)

Genentech, South San Francisco, CA, USA.

Virginia McNally (V)

Roche Products, Welwyn Garden City, UK.

Grant A McArthur (GA)

Melanoma and Skin Service and Cancer Therapeutics Program, Peter MacCallum Cancer Centre, Melbourne, VIC, Australia.

Paolo A Ascierto (PA)

Istituto Nazionale Tumori IRCCS Fondazione G Pascale, Naples, Italy.

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Classifications MeSH