Association between bacterial homoplastic variants and radiological pathology in tuberculosis.


Journal

Thorax
ISSN: 1468-3296
Titre abrégé: Thorax
Pays: England
ID NLM: 0417353

Informations de publication

Date de publication:
07 2020
Historique:
received: 05 03 2019
revised: 23 03 2020
accepted: 23 03 2020
pubmed: 18 6 2020
medline: 20 8 2020
entrez: 18 6 2020
Statut: ppublish

Résumé

Understanding how pathogen genetic factors contribute to pathology in TB could enable tailored treatments to the most pathogenic and infectious strains. New strategies are needed to control drug-resistant TB, which requires longer and costlier treatment. We hypothesised that the severity of radiological pathology on the chest radiograph in TB disease was associated with variants arising independently, multiple times (homoplasies) in the We performed whole genome sequencing (Illumina HiSeq2000 platform) on Two mutations were significantly associated with more widespread radiological pathology in a multivariable regression model controlling for confounding variables (Rv2828c.141, RR 1.3, 95% CI 1.21 to 1.39, p<0.01; rpoC.1040 95% CI 1.77 to 2.16, RR 1.9, p<0.01). The rpoB.450 mutation was associated with less extensive radiological pathology (RR 0.81, 95% CI 0.69 to 0.94, p=0.03), suggestive of a bacterial fitness cost for this mutation in vivo. Patients with a previous episode of TB disease and those between 10 and 30 years of age also had significantly increased radiological pathology. This study is the first to compare the

Sections du résumé

BACKGROUND
Understanding how pathogen genetic factors contribute to pathology in TB could enable tailored treatments to the most pathogenic and infectious strains. New strategies are needed to control drug-resistant TB, which requires longer and costlier treatment. We hypothesised that the severity of radiological pathology on the chest radiograph in TB disease was associated with variants arising independently, multiple times (homoplasies) in the
METHODS
We performed whole genome sequencing (Illumina HiSeq2000 platform) on
RESULTS
Two mutations were significantly associated with more widespread radiological pathology in a multivariable regression model controlling for confounding variables (Rv2828c.141, RR 1.3, 95% CI 1.21 to 1.39, p<0.01; rpoC.1040 95% CI 1.77 to 2.16, RR 1.9, p<0.01). The rpoB.450 mutation was associated with less extensive radiological pathology (RR 0.81, 95% CI 0.69 to 0.94, p=0.03), suggestive of a bacterial fitness cost for this mutation in vivo. Patients with a previous episode of TB disease and those between 10 and 30 years of age also had significantly increased radiological pathology.
CONCLUSIONS
This study is the first to compare the

Identifiants

pubmed: 32546574
pii: thoraxjnl-2019-213281
doi: 10.1136/thoraxjnl-2019-213281
pmc: PMC7361023
doi:

Substances chimiques

Bacterial Proteins 0
DNA, Bacterial 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

584-591

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom

Informations de copyright

© Author(s) (or their employer(s)) 2020. Re-use permitted under CC BY. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: JP and SP are consultants to Next Gen Diagnostics Llc.

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Auteurs

Louis Grandjean (L)

Department of Medicine, Imperial College London, London, UK l.grandjean@imperial.ac.uk.
Laboratorio de Investigacion y Enfermedades Infecciosas, Cayetano Heredia Pervuvian University, Lima, Peru.
Institute of Child Health, UCL Division of Infection and Immunity, London, UK.

Joha Monteserin (J)

Instituto Nacional de Enfermedades Infecciosas INEI-ANLIS, Administración Nacional de Laboratorios e Institutos de Salud Dr Carlos G Malbrán, Buenos Aires, Argentina.

Robert Gilman (R)

Laboratorio de Investigacion y Enfermedades Infecciosas, Cayetano Heredia Pervuvian University, Lima, Peru.
Johns Hopkins Bloomberg School of Public Health, Johns Hopkins University, Baltimore, Maryland, USA.

Julia Pauschardt (J)

Laboratorio de Investigacion y Enfermedades Infecciosas, Cayetano Heredia Pervuvian University, Lima, Peru.

Sakib Rokadiya (S)

Faculty of Medicine, Imperial College London, London, UK.

Cesar Bonilla (C)

Unidad Tecnica de Tuberculosis MDR, Ministerio de Salud, Lima, Peru.

Viviana Ritacco (V)

Instituto Nacional de Enfermedades Infecciosas INEI-ANLIS, Administración Nacional de Laboratorios e Institutos de Salud Dr Carlos G Malbrán, Buenos Aires, Argentina.

Julia Rios Vidal (JR)

Unidad Tecnica de Tuberculosis MDR, Ministerio de Salud, Lima, Peru.

Julian Parkhill (J)

Pathogen Genomics Group, Wellcome Trust Sanger Institute, Cambridge, UK.

Sharon Peacock (S)

Faculty of Medicine, University of Cambridge, Cambridge, UK.

David Aj Moore (DA)

TB Centre, London School of Hygiene and Tropical Medicine, London, UK.

Francois Balloux (F)

UCL Genetics Institute, University College London, London, UK.

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Classifications MeSH