Coagulation factor XIII activity predicts left ventricular remodelling after acute myocardial infarction.

Blood coagulation factor XIII Cardiac magnetic resonance imaging Healing and remodelling processes ST-elevation myocardial infarction

Journal

ESC heart failure
ISSN: 2055-5822
Titre abrégé: ESC Heart Fail
Pays: England
ID NLM: 101669191

Informations de publication

Date de publication:
10 2020
Historique:
received: 09 12 2019
revised: 21 04 2020
accepted: 07 05 2020
pubmed: 18 6 2020
medline: 22 6 2021
entrez: 18 6 2020
Statut: ppublish

Résumé

Acute myocardial infarction (MI) is the major cause of chronic heart failure. The activity of blood coagulation factor XIII (FXIIIa) plays an important role in rodents as a healing factor after MI, whereas its role in healing and remodelling processes in humans remains unclear. We prospectively evaluated the relevance of FXIIIa after acute MI as a potential early prognostic marker for adequate healing. This monocentric prospective cohort study investigated cardiac remodelling in patients with ST-elevation MI and followed them up for 1 year. Serum FXIIIa was serially assessed during the first 9 days after MI and after 2, 6, and 12 months. Cardiac magnetic resonance imaging was performed within 4 days after MI (Scan 1), after 7 to 9 days (Scan 2), and after 12 months (Scan 3). The FXIII valine-to-leucine (V34L) single-nucleotide polymorphism rs5985 was genotyped. One hundred forty-six patients were investigated (mean age 58 ± 11 years, 13% women). Median FXIIIa was 118% (quartiles, 102-132%) and dropped to a trough on the second day after MI: 109% (98-109%; P < 0.001). FXIIIa recovered slowly over time, reaching the baseline level after 2 to 6 months and surpassed baseline levels only after 12 months: 124% (110-142%). The development of FXIIIa after MI was independent of the genotype. FXIIIa on Day 2 was strongly and inversely associated with the relative size of MI in Scan 1 (Spearman's ρ = -0.31; P = 0.01) and Scan 3 (ρ = -0.39; P < 0.01) and positively associated with left ventricular ejection fraction: ρ = 0.32 (P < 0.01) and ρ = 0.24 (P = 0.04), respectively. FXIII activity after MI is highly dynamic, exhibiting a significant decline in the early healing period, with reconstitution 6 months later. Depressed FXIIIa early after MI predicted a greater size of MI and lower left ventricular ejection fraction after 1 year. The clinical relevance of these findings awaits to be tested in a randomized trial.

Identifiants

pubmed: 32548915
doi: 10.1002/ehf2.12774
pmc: PMC7524135
doi:

Substances chimiques

Factor XIII 9013-56-3

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2354-2364

Subventions

Organisme : Bundesministerium für Bildung und Forschung
ID : 01ES0816
Pays : International
Organisme : Bundesministerium für Bildung und Forschung
ID : 01ES01901
Pays : International
Organisme : Bundesministerium für Bildung und Forschung
ID : 01ES01902
Pays : International
Organisme : Bundesministerium für Bildung und Forschung
ID : 01EO1004
Pays : International

Informations de copyright

© 2020 The Authors. ESC Heart Failure published by John Wiley & Sons Ltd on behalf of the European Society of Cardiology.

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Auteurs

Anna Frey (A)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department of Internal Medicine I, University Hospital Würzburg, Würzburg, Germany.

Tobias Gassenmaier (T)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Institute of Radiology, University Hospital Würzburg, Würzburg, Germany.

Ulrich Hofmann (U)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department of Internal Medicine I, University Hospital Würzburg, Würzburg, Germany.

Dominik Schmitt (D)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department of Internal Medicine I, University Hospital Würzburg, Würzburg, Germany.

Georg Fette (G)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department for Artificial Intelligence and Applied Computer Science, University of Würzburg, Würzburg, Germany.

Almuth Marx (A)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.

Sabine Herterich (S)

Division of Laboratory Medicine, University Hospital Würzburg, Würzburg, Germany.

Valérie Boivin-Jahns (V)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department of Pharmacology and Toxicology, University of Würzburg, Würzburg, Germany.

Georg Ertl (G)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department of Internal Medicine I, University Hospital Würzburg, Würzburg, Germany.

Thorsten Bley (T)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Institute of Radiology, University Hospital Würzburg, Würzburg, Germany.

Stefan Frantz (S)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department of Internal Medicine I, University Hospital Würzburg, Würzburg, Germany.

Roland Jahns (R)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department of Internal Medicine I, University Hospital Würzburg, Würzburg, Germany.
Interdisciplinary Bank of Biomaterials and Data Würzburg, University Hospital Würzburg, Würzburg, Germany.

Stefan Störk (S)

Comprehensive Heart Failure Center Würzburg, University Hospital Würzburg, Würzburg, Germany.
Department of Internal Medicine I, University Hospital Würzburg, Würzburg, Germany.

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