Innate immune recognition and modulation in hepatitis D virus infection.
Carcinoma, Hepatocellular
/ immunology
Coinfection
/ complications
Hepatitis B virus
/ genetics
Hepatitis B, Chronic
/ complications
Hepatitis D, Chronic
/ complications
Hepatitis Delta Virus
/ genetics
Hepatitis delta Antigens
/ immunology
Humans
Immune Evasion
Immunity, Innate
Interferon-Induced Helicase, IFIH1
/ metabolism
Liver
/ immunology
Liver Cirrhosis
/ immunology
Liver Neoplasms
/ immunology
Organic Anion Transporters, Sodium-Dependent
/ metabolism
RNA, Viral
/ immunology
Receptors, Pattern Recognition
/ immunology
Satellite Viruses
/ genetics
Symporters
/ metabolism
Virus Replication
/ immunology
Hepatitis B virus
Hepatitis D virus
Immune evasion
Immunosuppression
Innate immunity
Pathogen-associated molecular pattern molecules
Journal
World journal of gastroenterology
ISSN: 2219-2840
Titre abrégé: World J Gastroenterol
Pays: United States
ID NLM: 100883448
Informations de publication
Date de publication:
07 Jun 2020
07 Jun 2020
Historique:
received:
29
12
2019
revised:
30
03
2020
accepted:
23
05
2020
entrez:
20
6
2020
pubmed:
20
6
2020
medline:
1
4
2021
Statut:
ppublish
Résumé
Hepatitis D virus (HDV) is a global health threat with more than 15 million humans affected. Current treatment options are largely unsatisfactory leaving chronically infected humans at high risk to develop liver cirrhosis and hepatocellular carcinoma. HDV is the only human satellite virus known. It encodes only two proteins, and requires Hepatitis B virus (HBV) envelope protein expression for productive virion release and spread of the infection. How HDV could evolve and why HBV was selected as a helper virus remains unknown. Since the discovery of Na
Identifiants
pubmed: 32550754
doi: 10.3748/wjg.v26.i21.2781
pmc: PMC7284172
doi:
Substances chimiques
Hepatitis delta Antigens
0
Organic Anion Transporters, Sodium-Dependent
0
RNA, Viral
0
Receptors, Pattern Recognition
0
Symporters
0
sodium-bile acid cotransporter
145420-23-1
IFIH1 protein, human
EC 3.6.1.-
Interferon-Induced Helicase, IFIH1
EC 3.6.4.13
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
2781-2791Informations de copyright
©The Author(s) 2020. Published by Baishideng Publishing Group Inc. All rights reserved.
Déclaration de conflit d'intérêts
Conflict-of-interest statement: The authors declare no conflict of interests concerning the content of this article.
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