Clinical implications of the serum CA19-9 level in "biological borderline resectability" and "biological downstaging" in the setting of preoperative chemoradiation therapy for pancreatic cancer.


Journal

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
ISSN: 1424-3911
Titre abrégé: Pancreatology
Pays: Switzerland
ID NLM: 100966936

Informations de publication

Date de publication:
Jul 2020
Historique:
received: 10 02 2020
revised: 18 05 2020
accepted: 27 05 2020
pubmed: 22 6 2020
medline: 10 7 2021
entrez: 22 6 2020
Statut: ppublish

Résumé

Biological factors are emphasized in borderline resectable pancreatic cancer (BRPC), and CA19-9 is an important factor for biological borderline resectability (b-BR). The aim of this study was to investigate the cut-off value of CA19-9 for biological borderline resectability and "biological downstaging" in chemoradiation therapy (CRT) for pancreatic cancer (PC). A total of 407 patients with anatomically resectable PC (a-R) and BRPC (a-BR) received preoperative gemcitabine-based CRT. The b-BR was determined, according to the CA19-9 value prior to preoperative CRT (pre-CA19-9), as the subgroup of a-R cases in which the survival was comparable with that in a-BR cases. "Biological downstaging" was determined based on prognostic analyses regarding the CA19-9 value after preoperative CRT (post-CA19-9) in association with the survival of R cases (a-R cases without the b-BR factor). The 5-year survival of a-R patients with pre-CA19-9 > 120 U/mL was comparable with that of a-BR patients (44% vs 34%, p = 0.082). The survival of b-BR patients with post-CRT CA19-9 ≤ 37 U/mL (normalized) was comparably favorable with that of R patients (56% vs 65%, p = 0.369). The incidence of distant recurrence was higher in b-BR patients without post-CA19-9 normalization than in those with post-CA19-9 normalization (70% vs 50%, p = 0.003), while the incidence of local recurrence was comparable between these two groups (12% vs 13%, p = 0.986). Biological BRPC was determined to be an anatomically resectable disease with pre-CA19-9 > 120 U/mL, and post-CA19-9 normalization indicated "biological downstaging" in b-BR in the preoperative CRT strategy.

Sections du résumé

BACKGROUND BACKGROUND
Biological factors are emphasized in borderline resectable pancreatic cancer (BRPC), and CA19-9 is an important factor for biological borderline resectability (b-BR). The aim of this study was to investigate the cut-off value of CA19-9 for biological borderline resectability and "biological downstaging" in chemoradiation therapy (CRT) for pancreatic cancer (PC).
METHODS METHODS
A total of 407 patients with anatomically resectable PC (a-R) and BRPC (a-BR) received preoperative gemcitabine-based CRT. The b-BR was determined, according to the CA19-9 value prior to preoperative CRT (pre-CA19-9), as the subgroup of a-R cases in which the survival was comparable with that in a-BR cases. "Biological downstaging" was determined based on prognostic analyses regarding the CA19-9 value after preoperative CRT (post-CA19-9) in association with the survival of R cases (a-R cases without the b-BR factor).
RESULTS RESULTS
The 5-year survival of a-R patients with pre-CA19-9 > 120 U/mL was comparable with that of a-BR patients (44% vs 34%, p = 0.082). The survival of b-BR patients with post-CRT CA19-9 ≤ 37 U/mL (normalized) was comparably favorable with that of R patients (56% vs 65%, p = 0.369). The incidence of distant recurrence was higher in b-BR patients without post-CA19-9 normalization than in those with post-CA19-9 normalization (70% vs 50%, p = 0.003), while the incidence of local recurrence was comparable between these two groups (12% vs 13%, p = 0.986).
CONCLUSIONS CONCLUSIONS
Biological BRPC was determined to be an anatomically resectable disease with pre-CA19-9 > 120 U/mL, and post-CA19-9 normalization indicated "biological downstaging" in b-BR in the preoperative CRT strategy.

Identifiants

pubmed: 32563596
pii: S1424-3903(20)30189-7
doi: 10.1016/j.pan.2020.05.020
pii:
doi:

Substances chimiques

Antimetabolites, Antineoplastic 0
Biomarkers 0
CA-19-9 Antigen 0
Radiation-Sensitizing Agents 0
Deoxycytidine 0W860991D6
Gemcitabine 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

919-928

Informations de copyright

Copyright © 2020 IAP and EPC. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest There is no personal conflict of interest of any of the authors.

Auteurs

Hidenori Takahashi (H)

Department of Surgery, Osaka International Cancer Institute, Japan. Electronic address: takahasi-hi@mc.pref.osaka.jp.

Daisaku Yamada (D)

Department of Surgery, Osaka International Cancer Institute, Japan.

Kei Asukai (K)

Department of Surgery, Osaka International Cancer Institute, Japan.

Hiroshi Wada (H)

Department of Surgery, Osaka International Cancer Institute, Japan.

Shinichiro Hasegawa (S)

Department of Surgery, Osaka International Cancer Institute, Japan.

Hisashi Hara (H)

Department of Surgery, Osaka International Cancer Institute, Japan.

Naoki Shinno (N)

Department of Surgery, Osaka International Cancer Institute, Japan.

Hajime Ushigome (H)

Department of Surgery, Osaka International Cancer Institute, Japan.

Naotsugu Haraguchi (N)

Department of Surgery, Osaka International Cancer Institute, Japan.

Keijiro Sugimura (K)

Department of Surgery, Osaka International Cancer Institute, Japan.

Kazuyoshi Yamamoto (K)

Department of Surgery, Osaka International Cancer Institute, Japan.

Junichi Nishimura (J)

Department of Surgery, Osaka International Cancer Institute, Japan.

Masayoshi Yasui (M)

Department of Surgery, Osaka International Cancer Institute, Japan.

Takeshi Omori (T)

Department of Surgery, Osaka International Cancer Institute, Japan.

Hiroshi Miyata (H)

Department of Surgery, Osaka International Cancer Institute, Japan.

Masayuki Ohue (M)

Department of Surgery, Osaka International Cancer Institute, Japan.

Masahiko Yano (M)

Department of Surgery, Osaka International Cancer Institute, Japan.

Masato Sakon (M)

Department of Surgery, Osaka International Cancer Institute, Japan.

Osamu Ishikawa (O)

Department of Surgery, Osaka International Cancer Institute, Japan.

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Classifications MeSH