Cohesin promotes stochastic domain intermingling to ensure proper regulation of boundary-proximal genes.


Journal

Nature genetics
ISSN: 1546-1718
Titre abrégé: Nat Genet
Pays: United States
ID NLM: 9216904

Informations de publication

Date de publication:
08 2020
Historique:
received: 27 08 2019
accepted: 18 05 2020
pubmed: 24 6 2020
medline: 27 10 2020
entrez: 24 6 2020
Statut: ppublish

Résumé

The human genome can be segmented into topologically associating domains (TADs), which have been proposed to spatially sequester genes and regulatory elements through chromatin looping. Interactions between TADs have also been suggested, presumably because of variable boundary positions across individual cells. However, the nature, extent and consequence of these dynamic boundaries remain unclear. Here, we combine high-resolution imaging with Oligopaint technology to quantify the interaction frequencies across both weak and strong boundaries. We find that chromatin intermingling across population-defined boundaries is widespread but that the extent of permissibility is locus-specific. Cohesin depletion, which abolishes domain formation at the population level, does not induce ectopic interactions but instead reduces interactions across all boundaries tested. In contrast, WAPL or CTCF depletion increases inter-domain contacts in a cohesin-dependent manner. Reduced chromatin intermingling due to cohesin loss affects the topology and transcriptional bursting frequencies of genes near boundaries. We propose that cohesin occasionally bypasses boundaries to promote incorporation of boundary-proximal genes into neighboring domains.

Identifiants

pubmed: 32572210
doi: 10.1038/s41588-020-0647-9
pii: 10.1038/s41588-020-0647-9
pmc: PMC7416539
mid: NIHMS1595869
doi:

Substances chimiques

Cell Cycle Proteins 0
Chromatin 0
Chromosomal Proteins, Non-Histone 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

840-848

Subventions

Organisme : NIGMS NIH HHS
ID : R35 GM128903
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM008216
Pays : United States

Commentaires et corrections

Type : CommentIn

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Auteurs

Jennifer M Luppino (JM)

Department of Genetics, Penn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Daniel S Park (DS)

Department of Genetics, Penn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Son C Nguyen (SC)

Department of Genetics, Penn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Yemin Lan (Y)

Department of Cell and Developmental Biology, Penn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Zhuxuan Xu (Z)

Department of Genetics, Penn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Rebecca Yunker (R)

Department of Genetics, Penn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Eric F Joyce (EF)

Department of Genetics, Penn Epigenetics Institute, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA. erjoyce@upenn.edu.

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