Clinical Activity and Safety of Atezolizumab in a Phase 1 Study of Patients With Relapsed/Refractory Small-Cell Lung Cancer.


Journal

Clinical lung cancer
ISSN: 1938-0690
Titre abrégé: Clin Lung Cancer
Pays: United States
ID NLM: 100893225

Informations de publication

Date de publication:
09 2020
Historique:
received: 23 12 2019
revised: 20 03 2020
accepted: 06 05 2020
pubmed: 27 6 2020
medline: 12 10 2021
entrez: 27 6 2020
Statut: ppublish

Résumé

Programmed death-ligand 1 (PD-L1) protein is expressed in various cancers, including small-cell lung cancer (SCLC). Atezolizumab inhibits PD-L1 signaling, thus restoring tumor-specific T-cell immunity. Here, we report results from the first-in-human phase 1 PCD4989g study (NCT01375842) of atezolizumab, in a cohort of patients with relapsed/refractory SCLC. Eligible patients with incurable or metastatic SCLC, which was advanced or recurrent since the last antitumor therapy, received atezolizumab 15 mg/kg or 1200 mg intravenously every 3 weeks for 16 cycles or until loss of clinical benefit. The primary endpoint was safety. Efficacy and biomarkers of antitumor activity were also assessed. Seventeen patients were enrolled. Any-grade and grade ≥3 treatment-related adverse events (TRAEs) occurred in 11 (64.7%) and 5 (29.4%) patients, respectively. The most common any-grade TRAE was fatigue (4 patients [23.5%]). Partial response to atezolizumab was achieved in 1 patient (5.9%) per Response Evaluation Criteria in Solid Tumors version 1.1 (RECIST v1.1), and 3 (17.6%) per immune-related response criteria (irRC). Durations of response were 2.8 to > 45.7 months. Median investigator-assessed progression-free survival (PFS) per RECIST v1.1 and irRC was 1.5 (95% confidence interval [CI], 1.2-2.7) and 2.9 (95% CI, 1.2-6.1) months, respectively. Median overall survival (OS) was 5.9 months (95% CI, 4.3-12.6). Patients with high (≥ median expression) T-effector gene signature and PD-L1 mRNA expression appeared to show a trend toward improved PFS (per irRC) and OS. Atezolizumab was generally well tolerated and exhibited antitumor activity in a small cohort of patients with relapsed/refractory SCLC.

Sections du résumé

BACKGROUND
Programmed death-ligand 1 (PD-L1) protein is expressed in various cancers, including small-cell lung cancer (SCLC). Atezolizumab inhibits PD-L1 signaling, thus restoring tumor-specific T-cell immunity. Here, we report results from the first-in-human phase 1 PCD4989g study (NCT01375842) of atezolizumab, in a cohort of patients with relapsed/refractory SCLC.
PATIENTS AND METHODS
Eligible patients with incurable or metastatic SCLC, which was advanced or recurrent since the last antitumor therapy, received atezolizumab 15 mg/kg or 1200 mg intravenously every 3 weeks for 16 cycles or until loss of clinical benefit. The primary endpoint was safety. Efficacy and biomarkers of antitumor activity were also assessed.
RESULTS
Seventeen patients were enrolled. Any-grade and grade ≥3 treatment-related adverse events (TRAEs) occurred in 11 (64.7%) and 5 (29.4%) patients, respectively. The most common any-grade TRAE was fatigue (4 patients [23.5%]). Partial response to atezolizumab was achieved in 1 patient (5.9%) per Response Evaluation Criteria in Solid Tumors version 1.1 (RECIST v1.1), and 3 (17.6%) per immune-related response criteria (irRC). Durations of response were 2.8 to > 45.7 months. Median investigator-assessed progression-free survival (PFS) per RECIST v1.1 and irRC was 1.5 (95% confidence interval [CI], 1.2-2.7) and 2.9 (95% CI, 1.2-6.1) months, respectively. Median overall survival (OS) was 5.9 months (95% CI, 4.3-12.6). Patients with high (≥ median expression) T-effector gene signature and PD-L1 mRNA expression appeared to show a trend toward improved PFS (per irRC) and OS.
CONCLUSION
Atezolizumab was generally well tolerated and exhibited antitumor activity in a small cohort of patients with relapsed/refractory SCLC.

Identifiants

pubmed: 32586767
pii: S1525-7304(20)30148-0
doi: 10.1016/j.cllc.2020.05.008
pii:
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
Antineoplastic Agents 0
atezolizumab 52CMI0WC3Y

Banques de données

ClinicalTrials.gov
['NCT01375842']

Types de publication

Clinical Trial, Phase I Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

455-463.e4

Informations de copyright

Copyright © 2020 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Anne C Chiang (AC)

Department of Medicine, Yale School of Medicine, Yale University, New Haven, CT. Electronic address: anne.chiang@yale.edu.

Lecia Van Dam Sequist (LVD)

Department of Medicine, Massachusetts General Hospital, Boston, MA.

Jill Gilbert (J)

Division of Hematolgy-Oncology, Department of Medicine, Vanderbilt-Ingram Cancer Center, Nashville, TN.

Paul Conkling (P)

US Oncology Research, Virginia Oncology Associates, Norfolk, VA.

Dana Thompson (D)

Department of Medical Oncology, Sarah Cannon Research Institute, Nashville, TN.

J Paul Marcoux (JP)

Dana-Farber/Brigham and Women's Cancer Center, Boston, MA.

Scott Gettinger (S)

Department of Medicine, Yale School of Medicine, Yale University, New Haven, CT.

Marcin Kowanetz (M)

Genentech, Inc., South San Francisco, CA.

Luciana Molinero (L)

Genentech, Inc., South San Francisco, CA.

Carol O'Hear (C)

Genentech, Inc., South San Francisco, CA.

Marcella Fassò (M)

Genentech, Inc., South San Francisco, CA.

Sivuonthanh Lam (S)

Genentech, Inc., South San Francisco, CA.

Michael S Gordon (MS)

Department of Hematology and Oncology, HonorHealth Research Institute, Scottsdale, AZ.

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Classifications MeSH