Amyloid Proteins and Peripheral Neuropathy.
amyloid neuropathies
amyloid proteins
amyloidosis
chronic pain
peripheral neuropathy
type 2 diabetes mellitus
Journal
Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052
Informations de publication
Date de publication:
26 06 2020
26 06 2020
Historique:
received:
02
06
2020
revised:
20
06
2020
accepted:
22
06
2020
entrez:
2
7
2020
pubmed:
2
7
2020
medline:
1
4
2021
Statut:
epublish
Résumé
Painful peripheral neuropathy affects millions of people worldwide. Peripheral neuropathy develops in patients with various diseases, including rare familial or acquired amyloid polyneuropathies, as well as some common diseases, including type 2 diabetes mellitus and several chronic inflammatory diseases. Intriguingly, these diseases share a histopathological feature-deposits of amyloid-forming proteins in tissues. Amyloid-forming proteins may cause tissue dysregulation and damage, including damage to nerves, and may be a common cause of neuropathy in these, and potentially other, diseases. Here, we will discuss how amyloid proteins contribute to peripheral neuropathy by reviewing the current understanding of pathogenic mechanisms in known inherited and acquired (usually rare) amyloid neuropathies. In addition, we will discuss the potential role of amyloid proteins in peripheral neuropathy in some common diseases, which are not (yet) considered as amyloid neuropathies. We conclude that there are many similarities in the molecular and cell biological defects caused by aggregation of the various amyloid proteins in these different diseases and propose a common pathogenic pathway for "peripheral amyloid neuropathies".
Identifiants
pubmed: 32604774
pii: cells9061553
doi: 10.3390/cells9061553
pmc: PMC7349787
pii:
doi:
Substances chimiques
Amyloidogenic Proteins
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
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