The Novel
Animals
Antioxidants
/ pharmacology
Calcification, Physiologic
/ drug effects
Cell Line
Cells, Cultured
Mice
Muscle, Smooth, Vascular
/ cytology
Myocytes, Smooth Muscle
/ drug effects
Oxidation-Reduction
/ drug effects
Oxidative Stress
/ drug effects
Peroxides
/ pharmacology
Reactive Oxygen Species
/ metabolism
Vascular Calcification
/ drug therapy
calcification
dihydroperoxides
phosphate
reactive oxygen species
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
29 Jun 2020
29 Jun 2020
Historique:
received:
26
05
2020
revised:
25
06
2020
accepted:
28
06
2020
entrez:
3
7
2020
pubmed:
3
7
2020
medline:
20
2
2021
Statut:
epublish
Résumé
The excessive intake of phosphate (Pi), or chronic kidney disease (CKD), can cause hyperphosphatemia and eventually lead to ectopic calcification, resulting in cerebrovascular diseases. It has been reported that reactive oxygen species (ROS), induced by high concentrations of Pi loading, play a key role in vascular calcification. Therefore, ROS suppression may be a useful treatment strategy for vascular calcification. 12AC3O is a newly synthesized
Identifiants
pubmed: 32610684
pii: ijms21134628
doi: 10.3390/ijms21134628
pmc: PMC7369805
pii:
doi:
Substances chimiques
Antioxidants
0
Peroxides
0
Reactive Oxygen Species
0
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Japan Society for the Promotion of Science
ID : 17K19666
Organisme : Japan Society for the Promotion of Science
ID : JP17K08311
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