Pro-Inflammatory Signaling Upregulates a Neurotoxic Conotoxin-Like Protein Encrypted Within Human Endogenous Retrovirus-K.
HIV Tat
NF-κB
amyotrophic lateral sclerosis
conotoxin
endogenous retrovirus
myelination
necroptosis
Journal
Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052
Informations de publication
Date de publication:
30 06 2020
30 06 2020
Historique:
received:
30
05
2020
revised:
20
06
2020
accepted:
25
06
2020
entrez:
8
7
2020
pubmed:
8
7
2020
medline:
13
4
2021
Statut:
epublish
Résumé
Motor neuron degeneration and spinal cord demyelination are hallmark pathological events in Amyotrophic Lateral Sclerosis (ALS). Endogenous retrovirus-K (ERVK) expression has an established association with ALS neuropathology, with murine modeling pointing to a role for the ERVK envelope (
Identifiants
pubmed: 32629888
pii: cells9071584
doi: 10.3390/cells9071584
pmc: PMC7407490
pii:
doi:
Substances chimiques
Conotoxins
0
NF-kappa B
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : CIHR
Pays : Canada
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