The cerebral blood flow deficits in Parkinson's disease with mild cognitive impairment using arterial spin labeling MRI.

Arterial spin labeling MRI Cerebral blood flow Microtubule-associated protein tau (MAPT) Mild cognitive impairment Parkinson’s disease

Journal

Journal of neural transmission (Vienna, Austria : 1996)
ISSN: 1435-1463
Titre abrégé: J Neural Transm (Vienna)
Pays: Austria
ID NLM: 9702341

Informations de publication

Date de publication:
09 2020
Historique:
received: 28 01 2020
accepted: 30 06 2020
pubmed: 8 7 2020
medline: 16 10 2021
entrez: 8 7 2020
Statut: ppublish

Résumé

Parkinson's disease (PD) with mild cognitive impairment (PD-MCI) is currently diagnosed based on an arbitrarily predefined standard deviation of neuropsychological test scores, and more objective biomarkers for PD-MCI diagnosis are needed. The purpose of this study was to define possible brain perfusion-based biomarkers of not only mild cognitive impairment, but also risky gene carriers in PD using arterial spin labeling magnetic resonance imaging (ASL-MRI). Fifteen healthy controls (HC), 26 cognitively normal PD (PD-CN), and 27 PD-MCI subjects participated in this study. ASL-MRI data were acquired by signal targeting with alternating radio-frequency labeling with Look-Locker sequence at 3 T. Single nucleotide polymorphism genotyping for rs9468 [microtubule-associated protein tau (MAPT) H1/H1 versus H1/H2 haplotype] was performed using a Stratagene Mx3005p real-time polymerase chain-reaction system (Agilent Technologies, USA). There were 15 subjects with MAPT H1/H1 and 11 subjects with MAPT H1/H2 within PD-MCI, and 33 subjects with MAPT H1/H1 and 19 subjects with MAPT H1/H2 within all PD. Voxel-wise differences of cerebral blood flow (CBF) values between HC, PD-CN and PD-MCI were assessed by one-way analysis of variance followed by pairwise post hoc comparisons. Further, the subgroup of PD patients carrying the risky MAPT H1/H1 haplotype was compared with noncarriers (MAPT H1/H2 haplotype) in terms of CBF by a two-sample t test. A pattern that could be summarized as "posterior hypoperfusion" (PH) differentiated the PD-MCI group from the HC group with an accuracy of 92.6% (sensitivity = 93%, specificity = 93%). Additionally, the PD patients with MAPT H1/H1 haplotype had decreased perfusion than the ones with H1/H2 haplotype at the posterior areas of the visual network (VN), default mode network (DMN), and dorsal attention network (DAN). The PH-type pattern in ASL-MRI could be employed as a biomarker of both current cognitive impairment and future cognitive decline in PD.

Identifiants

pubmed: 32632889
doi: 10.1007/s00702-020-02227-6
pii: 10.1007/s00702-020-02227-6
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1285-1294

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Auteurs

Dilek Betul Arslan (DB)

Institute of Biomedical Engineering, Bogazici University, Istanbul, Turkey.

Hakan Gurvit (H)

Behavioral Neurology and Movement Disorders Unit, Department of Neurology, Istanbul Faculty of Medicine, Istanbul University, Istanbul, Turkey.

Ozan Genc (O)

Institute of Biomedical Engineering, Bogazici University, Istanbul, Turkey.

Ani Kicik (A)

Neuroimaging Unit, Hulusi Behcet Life Sciences Research Center, Istanbul University, Istanbul, Turkey.
Department of Physiology, Faculty of Medicine, Demiroglu Bilim University, Istanbul, Turkey.

Kardelen Eryurek (K)

Neuroimaging Unit, Hulusi Behcet Life Sciences Research Center, Istanbul University, Istanbul, Turkey.
Department of Neuroscience, Aziz Sancar Institute of Experimental Medicine, Istanbul University, Istanbul, Turkey.

Sevim Cengiz (S)

Institute of Biomedical Engineering, Bogazici University, Istanbul, Turkey.

Emel Erdogdu (E)

Neuroimaging Unit, Hulusi Behcet Life Sciences Research Center, Istanbul University, Istanbul, Turkey.
Department of Psychology, Faculty of Arts and Sciences, Isik University, Istanbul, Turkey.

Zerrin Yildirim (Z)

Behavioral Neurology and Movement Disorders Unit, Department of Neurology, Istanbul Faculty of Medicine, Istanbul University, Istanbul, Turkey.

Zeynep Tufekcioglu (Z)

Behavioral Neurology and Movement Disorders Unit, Department of Neurology, Istanbul Faculty of Medicine, Istanbul University, Istanbul, Turkey.

Aziz Müfit Uluğ (AM)

Institute of Biomedical Engineering, Bogazici University, Istanbul, Turkey.
CorTechs Labs, San Diego, CA, USA.

Basar Bilgic (B)

Behavioral Neurology and Movement Disorders Unit, Department of Neurology, Istanbul Faculty of Medicine, Istanbul University, Istanbul, Turkey.

Hasmet Hanagasi (H)

Behavioral Neurology and Movement Disorders Unit, Department of Neurology, Istanbul Faculty of Medicine, Istanbul University, Istanbul, Turkey.

Erdem Tuzun (E)

Department of Neuroscience, Aziz Sancar Institute of Experimental Medicine, Istanbul University, Istanbul, Turkey.

Tamer Demiralp (T)

Neuroimaging Unit, Hulusi Behcet Life Sciences Research Center, Istanbul University, Istanbul, Turkey.
Department of Physiology, Istanbul Faculty of Medicine, Istanbul University, Istanbul, Turkey.

Esin Ozturk-Isik (E)

Institute of Biomedical Engineering, Bogazici University, Istanbul, Turkey. esin.ozturk@boun.edu.tr.

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