Physiopathology of the Permeability Transition Pore: Molecular Mechanisms in Human Pathology.
Cyclosporin A
apoptosis
cancer
ischemia/reperfusion
mitochondrial permeability transition
necrosis
neurodegeneration
Journal
Biomolecules
ISSN: 2218-273X
Titre abrégé: Biomolecules
Pays: Switzerland
ID NLM: 101596414
Informations de publication
Date de publication:
04 07 2020
04 07 2020
Historique:
received:
09
06
2020
revised:
29
06
2020
accepted:
02
07
2020
entrez:
9
7
2020
pubmed:
9
7
2020
medline:
20
4
2021
Statut:
epublish
Résumé
Mitochondrial permeability transition (MPT) is the sudden loss in the permeability of the inner mitochondrial membrane (IMM) to low-molecular-weight solutes. Due to osmotic forces, MPT is paralleled by a massive influx of water into the mitochondrial matrix, eventually leading to the structural collapse of the organelle. Thus, MPT can initiate outer-mitochondrial-membrane permeabilization (MOMP), promoting the activation of the apoptotic caspase cascade and caspase-independent cell-death mechanisms. The induction of MPT is mostly dependent on mitochondrial reactive oxygen species (ROS) and Ca
Identifiants
pubmed: 32635556
pii: biom10070998
doi: 10.3390/biom10070998
pmc: PMC7408088
pii:
doi:
Substances chimiques
Mitochondrial Permeability Transition Pore
0
Reactive Oxygen Species
0
Calcium
SY7Q814VUP
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
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