3-Dimensional architecture of the human multi-tRNA synthetase complex.


Journal

Nucleic acids research
ISSN: 1362-4962
Titre abrégé: Nucleic Acids Res
Pays: England
ID NLM: 0411011

Informations de publication

Date de publication:
04 09 2020
Historique:
accepted: 06 07 2020
revised: 08 06 2020
received: 13 03 2020
pubmed: 10 7 2020
medline: 21 10 2020
entrez: 10 7 2020
Statut: ppublish

Résumé

In mammalian cells, eight cytoplasmic aminoacyl-tRNA synthetases (AARS), and three non-synthetase proteins, reside in a large multi-tRNA synthetase complex (MSC). AARSs have critical roles in interpretation of the genetic code during protein synthesis, and in non-canonical functions unrelated to translation. Nonetheless, the structure and function of the MSC remain unclear. Partial or complete crystal structures of all MSC constituents have been reported; however, the structure of the holo-MSC has not been resolved. We have taken advantage of cross-linking mass spectrometry (XL-MS) and molecular docking to interrogate the three-dimensional architecture of the MSC in human HEK293T cells. The XL-MS approach uniquely provides structural information on flexibly appended domains, characteristic of nearly all MSC constituents. Using the MS-cleavable cross-linker, disuccinimidyl sulfoxide, inter-protein cross-links spanning all MSC constituents were observed, including cross-links between eight protein pairs not previously known to interact. Intra-protein cross-links defined new structural relationships between domains in several constituents. Unexpectedly, an asymmetric AARS distribution was observed featuring a clustering of tRNA anti-codon binding domains on one MSC face. Possibly, the non-uniform localization improves efficiency of delivery of charged tRNA's to an interacting ribosome during translation. In summary, we show a highly compact, 3D structural model of the human holo-MSC.

Identifiants

pubmed: 32644155
pii: 5869352
doi: 10.1093/nar/gkaa569
pmc: PMC7470956
doi:

Substances chimiques

Multiprotein Complexes 0
Amino Acyl-tRNA Synthetases EC 6.1.1.-

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

8740-8754

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL128300
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL128268
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM086430
Pays : United States
Organisme : NHLBI NIH HHS
ID : P01 HL029582
Pays : United States
Organisme : NIH HHS
ID : S10 OD023436
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK123236
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK124203
Pays : United States
Organisme : NHLBI NIH HHS
ID : P01 HL076491
Pays : United States

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press on behalf of Nucleic Acids Research.

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Auteurs

Krishnendu Khan (K)

Department of Cardiovascular and Metabolic Sciences, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, OH 44195, USA.

Camelia Baleanu-Gogonea (C)

Department of Chemistry, Cleveland State University, Cleveland, OH 44115, USA.

Belinda Willard (B)

Lerner Research Institute Proteomics and Metabolomics Core, Cleveland Clinic Foundation, Cleveland, OH 44195, USA.

Valentin Gogonea (V)

Department of Chemistry, Cleveland State University, Cleveland, OH 44115, USA.

Paul L Fox (PL)

Department of Cardiovascular and Metabolic Sciences, Lerner Research Institute, Cleveland Clinic Foundation, Cleveland, OH 44195, USA.

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Classifications MeSH