Histologically Diverse BAP1-Deficient Melanocytic Tumors in a Patient With BAP1 Tumor Predisposition Syndrome.


Journal

The American Journal of dermatopathology
ISSN: 1533-0311
Titre abrégé: Am J Dermatopathol
Pays: United States
ID NLM: 7911005

Informations de publication

Date de publication:
Nov 2020
Historique:
pubmed: 11 7 2020
medline: 14 8 2021
entrez: 11 7 2020
Statut: ppublish

Résumé

BRCA1-associated protein-1 (BAP1)-deficient cutaneous tumors are common in patients with BAP1 tumor predisposition syndrome, frequently presenting before other associated neoplasms, and can serve as an early marker to identify individuals with this disease. The typical lesions are dermal based and composed of a combination of larger epithelioid melanocytes with abundant glassy cytoplasm and smaller cells resembling those of a conventional nevus. There is often a component of interspersed lymphocytes. However, BAP1-deficient melanocytic tumors can show a spectrum of histologic appearances, ranging from lesions with pure epithelioid, pure conventional nevus, or rhabdoid cells and tumors with an intraepidermal component. To demonstrate such morphologic variation, we present a case of a 50-year-old woman with multiple histologically diverse BAP1-deficient melanocytic tumors and germline BAP1 mutation, identified after a diagnosis of pleural mesothelioma. We also discuss the pathogenesis and potential histopathological and clinical indications of germline versus sporadic etiology in the assessment of BAP1-deficient melanocytic tumors.

Identifiants

pubmed: 32649346
doi: 10.1097/DAD.0000000000001719
pii: 00000372-202011000-00011
doi:

Substances chimiques

BAP1 protein, human 0
Tumor Suppressor Proteins 0
Ubiquitin Thiolesterase EC 3.4.19.12

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

872-875

Références

Online Mendelian Inheritance in Man, OMIM. Number: 614327. Baltimore, MD: Johns Hopkins University; 2013. [cited March 25, 2020]. Available at: https://www.omim.org/entry/614327. Accessed October 10, 2019.
Rawson R, Watson G, Maher A, et al. Germline BAP1 mutations also predispose to cutaneous squamous cell carcinoma. Pathology 2017;49:539–542.
Walpole S, Pritchard A, Cebulla C, et al. Comprehensive study of the clinical phenotype of germline BAP1 variant-carrying families worldwide. J Natl Cancer Inst. 2018;110:1328–1341.
Chau C, van Doorn R, van Poppelen N, et al. Families with BAP1-tumor predisposition syndrome in the Netherlands: path to identification and a proposal for genetic screening guidelines. Cancers 2019;11:1114.
Abdel-Rahman C, Pilarski R, Cebulla C, et al. Germline BAP1 mutation predisposes to uveal melanoma, lung adenocarcinoma, meningioma, and other cancers. Cancer Genet. 2011;48:856–859.
Carbone M. BAP1 and cancer. Nat Rev Cancer. 2013;13:153–159.
Rai K, Pilarski R, Cebulla C, et al. Comprehensive review of BAP1 tumor predisposition syndrome with report of two new cases. Clin Genet. 2016;89:285–294.
Yu H, Pak H, Hammond-Martel I, et al. Tumor suppressor and deubiquitinase BAP1 promotes DNA double-strand break repair. Proc Natl Acad Sci USA. 2014;111:285–290.
Testa J, Cheung M, Pei J, et al. Germline BAP1 mutations predispose to malignant mesothelioma. Nat Genet. 2011;43:1022–1025.
Weisner T, Obenauf A, Murali R, et al. Germline mutations in BAP1 predispose to melanocytic tumors. Nat Genet. 2011;43:1018–1021.
Zhang A, Rush P, Tsao H, et al. BRCA1-associated protein (BAP1)-inactivated melanocytic tumors. J Cutan Pathol. 2019;46:965–972.
Harbour J, Onken M, Roberson E, et al. Frequent mutation of BAP1 in metastasizing uveal melanomas. Science 2010:330:1410–1413.
Busam K, Sung J, Weisner T, et al. Combined BRAFV600E-positive melanocytic lesions with large epithelioid cells lacking BAP1 expression and conventional nevomelanocytes. Am J Surg Pathol. 2013;37:193–199.
Elder D, Massi D, Scolyer R, et al, eds. World Health Organization Classification of Tumours. Lyon, France: International Agency for Research on Cancer (IARC); 2018.
Garfield E, Walton K, Quan V, et al. Histomorphologic spectrum of germline-related and sporadic BAP1-inactivated melanocytic tumors. Dermatopathology. 2018;79:525–534.
O'Shea S, Mitra A, Graham J, et al. Histopathology of melanocytic lesions in a family with an inherited BAP1 mutation. J Cutan Pathol. 2016;43:287–289.
Ardakani N, Palmer D, Wood B. BAP1 deficient malignant melanoma arising from the intradermal component of a congenital melanocytic naevus. Pathology. 2015;47:707–710.
Aung P, Nagarajan P, Tetzlaff M, et al. Melanoma with loss of BAP1 expression in patients with No family history of BAP1-associated cancer susceptibility syndrome: a case series. Am J Dermatopathol. 2019;41:167–179.
Palmedo G, Hantschke M, Rütten A, et al. The T1796A mutation of the BRAF gene is absent in Spitz nevi. J Cutan Pathol. 2004;31:266–270.
Gammon B, Traczyk T, Gerami P. Clumped perinuclear BAP1 expression is a frequent finding in sporadic epithelioid Spitz tumors. J Cutan Pathol. 2013;40:538–542.
Haugh A, Njauw C, Bubley J, et al. Genotypic and phenotypic features of BAP1 cancer syndrome: a report of 8 new families and review of cases in the literature. JAMA Dermatol. 2017;153:999–1006.
Marusic Z, Buljan M, Busam K. Histomorphologic spectrum of BAP1 negative melanocytic neoplasms in a family with BAP1-associated cancer susceptibility syndrome. J Cutan Pathol. 2015;42:406–412.
Wysozan T, Khelifa S, Turchan K, et al. The morphologic spectrum of germline-mutated BAP1-inactivated melanocytic tumors includes lesions with conventional nevic melanocytes: a case report and review of literature. J Cutan Pathol. 2019;46:852–857.
Murali R, Wiesner T, Scolyer R. Tumours associated with BAP1 mutations. Pathology. 2013;45:116–126.
National Center for Biotechnology Information. ClinVar. VCV000655445.1. 2018. Available at: https://www.ncbi.nlm.nih.gov/clinvar/variation/VCV000655445.1. Accessed October 2, 2019.
Pastorino S, Yoshikawa Y, Pass H, et al. A subset of mesotheliomas with improved survival occurring in carriers of BAP1 and other germline mutations. J Clin Oncol. 2018;36:3485–3494.
Carbone M, Kanodia S, Chao A, et al. Consensus report of the 2015 Weinman International Conference on mesothelioma. J Thorac Oncol. 2016;11:1246–1262.

Auteurs

Amber Louw (A)

Department of Anatomical Pathology, PathWest Laboratory Medicine, QEII Medical Centre, Perth, WA, Australia.
School of Medical and Health Sciences, Edith Cowan University, Perth, WA.

Jenette Creaney (J)

National Centre for Asbestos Related Disease, Faculty of Health and Medical Science, University of Western Australia, Nedlands, Western Australia, Australia.
Institute of Respiratory Health, Nedlands, Western Australia, Australia.
Department of Respiratory Medicine, Sir Charles Gairdner Hospital, Nedlands, Western Australia, Australia.

Anitha Thomas (A)

Department of Anatomical Pathology, PathWest Laboratory Medicine, QEII Medical Centre, Perth, WA, Australia.
School of Pathology and Laboratory Medicine, University of Western Australia, Perth, WA, Australia; and.

Chris Van Vliet (C)

Department of Anatomical Pathology, PathWest Laboratory Medicine, QEII Medical Centre, Perth, WA, Australia.
School of Pathology and Laboratory Medicine, University of Western Australia, Perth, WA, Australia; and.

Nathan Tobias Harvey (NT)

Department of Anatomical Pathology, PathWest Laboratory Medicine, QEII Medical Centre, Perth, WA, Australia.
School of Pathology and Laboratory Medicine, University of Western Australia, Perth, WA, Australia; and.

Benjamin Andrew Wood (BA)

Department of Anatomical Pathology, PathWest Laboratory Medicine, QEII Medical Centre, Perth, WA, Australia.
School of Pathology and Laboratory Medicine, University of Western Australia, Perth, WA, Australia; and.

Nima Mesbah Ardakani (N)

Department of Anatomical Pathology, PathWest Laboratory Medicine, QEII Medical Centre, Perth, WA, Australia.
School of Pathology and Laboratory Medicine, University of Western Australia, Perth, WA, Australia; and.
College of Science, Health, Engineering and Education, Murdoch University, Perth, WA, Australia.

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