Firm parenting and youth adjustment: Stress reactivity and dyadic synchrony of respiratory sinus arrhythmia.
acute stress response
externalizing problems
parent-child dyadic synchrony
parenting
Journal
Developmental psychobiology
ISSN: 1098-2302
Titre abrégé: Dev Psychobiol
Pays: United States
ID NLM: 0164074
Informations de publication
Date de publication:
04 2021
04 2021
Historique:
revised:
11
06
2020
received:
30
09
2019
accepted:
21
06
2020
pubmed:
18
7
2020
medline:
15
12
2021
entrez:
18
7
2020
Statut:
ppublish
Résumé
Parental behaviors are potent risk and protective factors for youth development of externalizing problems. Firm control is a parenting strategy that is inconsistently linked to youth adjustment, possibly due to variations in individual biological contexts. Growing research shows that dyadic coregulation of the autonomic nervous system (e.g., parent-child physiological synchrony) is a neurobiological mechanism that links parenting to youth adjustment. However, physiological synchrony may be context-dependent (e.g., adaptive in positive interactions, maladaptive in negative interactions). We aimed to test the role of dyadic synchrony in respiratory sinus arrhythmia (RSA) during parent-child conflict as a mediator between parent firm control and youth's externalizing problems. To capture youth's stress reactivity, we also tested how galvanic skin response reactivity (GSR-R) moderated this indirect path. The sample included 101 dyads of low socioeconomic-status at-risk preadolescents and parents. Results indicated that youth higher levels of GSR-R significantly intensified the link between parent firm control and dyadic RSA synchrony during conflict. Dyadic RSA synchrony further predicted youth increased in externalizing problems. Overall, results suggest that when parents employ firm control parenting with highly reactive teens, dyadic RSA synchrony elevates, potentially modeling less optimal coping with conflict for the youth, which is associated with increased externalizing problems.
Identifiants
pubmed: 32677062
doi: 10.1002/dev.22019
pmc: PMC7854988
mid: NIHMS1620831
doi:
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
470-480Subventions
Organisme : NIDA NIH HHS
ID : K01 DA045219
Pays : United States
Informations de copyright
© 2020 Wiley Periodicals LLC.
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