Intra-abdominal hypertension in early post-kidney transplantation period is associated with impaired graft function.


Journal

Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
ISSN: 1460-2385
Titre abrégé: Nephrol Dial Transplant
Pays: England
ID NLM: 8706402

Informations de publication

Date de publication:
01 09 2020
Historique:
received: 10 12 2019
accepted: 14 04 2020
pubmed: 18 7 2020
medline: 4 2 2021
entrez: 18 7 2020
Statut: ppublish

Résumé

Moderate hyperhydration is often achieved in the early post-kidney transplantation period. Whether this strategy could lead to the development of intra-abdominal hypertension (IAH) has never been assessed so far. We aimed to study the incidence of IAH after kidney transplantation and its association with graft function recovery. We conducted a prospective monocentric study among patients undergoing kidney transplantation at the University Hospital of Reims between May 2017 and April 2019. Intravesical pressure (IVP) was monitored every 8 h from Day 0 to 3. A total of 107 patients were enrolled. Among 55 patients included in the analysis, 74.5% developed IAH. Body mass index >25 kg/m2 was associated with IAH development {odds ratio [OR] 10.4 [95% confidence interval (CI) 2.0-52.9]; P = 0.005}. A previous history of peritoneal dialysis was protective [OR 0.06 (95% CI 0.01-0.3); P = 0.001]. IAH Grades III and IV occurred in 30.9% of patients and correlated with higher Day 3 creatininaemia (419.6 ± 258.5 versus 232.5 ± 189.4 μmol/L; P = 0.02), higher delayed graft function incidence (41.2 versus 7.9%; P = 0.04), lower Kirchner index measured using scintigraphy (0.47 ± 0.09 versus 0.64 ± 0.09; P = 0.0005) and decreased Day 30 estimated glomerular filtration rate (35.8 ± 18.8 versus 52.5 ± 21.3, P = 0.05). IAH patients had higher fluid balance (P = 0.02). Evolution of IVP correlated with weight gain (P < 0.01) and central venous pressure (P < 0.001). IAH is frequent after kidney transplantation and IAH Grades III and IV are independently associated with impaired graft function. These results question current haemodynamic objectives and raise for the first time interest in intra-abdominal pressure monitoring in these patients. ClinicalTrials.gov identifier: NCT03478176.

Sections du résumé

BACKGROUND
Moderate hyperhydration is often achieved in the early post-kidney transplantation period. Whether this strategy could lead to the development of intra-abdominal hypertension (IAH) has never been assessed so far. We aimed to study the incidence of IAH after kidney transplantation and its association with graft function recovery.
METHODS
We conducted a prospective monocentric study among patients undergoing kidney transplantation at the University Hospital of Reims between May 2017 and April 2019. Intravesical pressure (IVP) was monitored every 8 h from Day 0 to 3.
RESULTS
A total of 107 patients were enrolled. Among 55 patients included in the analysis, 74.5% developed IAH. Body mass index >25 kg/m2 was associated with IAH development {odds ratio [OR] 10.4 [95% confidence interval (CI) 2.0-52.9]; P = 0.005}. A previous history of peritoneal dialysis was protective [OR 0.06 (95% CI 0.01-0.3); P = 0.001]. IAH Grades III and IV occurred in 30.9% of patients and correlated with higher Day 3 creatininaemia (419.6 ± 258.5 versus 232.5 ± 189.4 μmol/L; P = 0.02), higher delayed graft function incidence (41.2 versus 7.9%; P = 0.04), lower Kirchner index measured using scintigraphy (0.47 ± 0.09 versus 0.64 ± 0.09; P = 0.0005) and decreased Day 30 estimated glomerular filtration rate (35.8 ± 18.8 versus 52.5 ± 21.3, P = 0.05). IAH patients had higher fluid balance (P = 0.02). Evolution of IVP correlated with weight gain (P < 0.01) and central venous pressure (P < 0.001).
CONCLUSIONS
IAH is frequent after kidney transplantation and IAH Grades III and IV are independently associated with impaired graft function. These results question current haemodynamic objectives and raise for the first time interest in intra-abdominal pressure monitoring in these patients.
CLINICAL TRIAL NOTATION
ClinicalTrials.gov identifier: NCT03478176.

Identifiants

pubmed: 32678426
pii: 5873140
doi: 10.1093/ndt/gfaa104
doi:

Banques de données

ClinicalTrials.gov
['NCT03478176']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1619-1628

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved.

Auteurs

Vincent Dupont (V)

Department of Nephrology, University Hospital of Reims, Reims, France.

Alexandre Debrumetz (A)

Department of Nephrology, University Hospital of Reims, Reims, France.

Adrien Leguillou (A)

Clinical Research Unit, University Hospital of Reims, Reims, France.

David Morland (D)

Department of Nuclear Medicine, Jean Godinot Institut, Reims, France.

Alain Wynckel (A)

Department of Nephrology, University Hospital of Reims, Reims, France.

Charlotte Colosio (C)

Department of Nephrology, University Hospital of Reims, Reims, France.

Laetitia Mokri (L)

Department of Nephrology, University Hospital of Reims, Reims, France.

Betoul Schvartz (B)

Department of Nephrology, University Hospital of Reims, Reims, France.

Vincent Vuiblet (V)

Department of Nephrology, University Hospital of Reims, Reims, France.

Stephane Larre (S)

Department of Urology, University Hospital of Reims, Reims, France.

Coralie Barbe (C)

Clinical Research Unit, University Hospital of Reims, Reims, France.

Philippe Rieu (P)

Department of Nephrology, University Hospital of Reims, Reims, France.

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