Curcumin protects thymus against D-galactose-induced senescence in mice.


Journal

Naunyn-Schmiedeberg's archives of pharmacology
ISSN: 1432-1912
Titre abrégé: Naunyn Schmiedebergs Arch Pharmacol
Pays: Germany
ID NLM: 0326264

Informations de publication

Date de publication:
02 2021
Historique:
received: 04 01 2020
accepted: 08 07 2020
pubmed: 21 7 2020
medline: 26 10 2021
entrez: 21 7 2020
Statut: ppublish

Résumé

Senescence-related decline of thymus affects immune function in the elderly population and contributes to the prevalence of many relevant diseases like cancer, autoimmune diseases, and other chronic diseases. In this study, we investigated the therapeutic effects of curcumin, an agent that could counter aging, and explored its optimal intake and the alteration of autoimmune regulator (Aire) after curcumin treatment in the D-galactose (D-gal)-induced accelerated aging mice. ICR mice were intraperitoneally injected with D-gal for 8 weeks to establish the accelerated aging model and given curcumin with 50, 100, and 200 mg/kg body weight per day by gavage, respectively, for 6 weeks. It indicated that the D-gal-treated mice developed structural changes in the thymi compared with the control group without D-gal and curcumin treatment. As the supplements of curcumin, it resulted in a restoration of the normal thymic anatomy with an increase of proliferating cells and a reduction of apoptotic cells in the thymi of the D-gal-induced aging model mice. Curcumin administration could also expand the expression level of Aire from mRNA level and protein level. The current study demonstrated that curcumin could ameliorate senescence-related thymus involution via upregulating Aire expression, suggesting that curcumin can rejuvenate senescence-associated alterations of thymus induced by D-gal accumulation.

Identifiants

pubmed: 32686020
doi: 10.1007/s00210-020-01945-8
pii: 10.1007/s00210-020-01945-8
doi:

Substances chimiques

Protective Agents 0
Transcription Factors 0
Curcumin IT942ZTH98
Galactose X2RN3Q8DNE

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

411-420

Subventions

Organisme : Youth Development Fund from School of Basic Medical Sciences, Zhengzhou University
ID : No. JCYXY2016-YQ-05

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Auteurs

Jie-Han Li (JH)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China.

Ting-Ting Wei (TT)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China.

Li Guo (L)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China.

Jia-Hui Cao (JH)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China.

Yuan-Kang Feng (YK)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China.

Shu-Ning Guo (SN)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China.

Guo-Hong Liu (GH)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China.

Yi Ding (Y)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China.

Yu-Rong Chai (YR)

Department of Histology and Embryology, School of Basic Medical Sciences, Zhengzhou University, No. 100 Ke Xue Road, Zhengzhou, 450001, Henan, China. yrchai@zzu.edu.cn.

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