CGI-58: Versatile Regulator of Intracellular Lipid Droplet Homeostasis.


Journal

Advances in experimental medicine and biology
ISSN: 0065-2598
Titre abrégé: Adv Exp Med Biol
Pays: United States
ID NLM: 0121103

Informations de publication

Date de publication:
2020
Historique:
entrez: 25 7 2020
pubmed: 25 7 2020
medline: 29 12 2020
Statut: ppublish

Résumé

Comparative gene identification-58 (CGI-58), also known as α/β-hydrolase domain-containing 5 (ABHD5), is a member of a large family of proteins containing an α/β-hydrolase-fold. CGI-58 is well-known as the co-activator of adipose triglyceride lipase (ATGL), which is a key enzyme initiating cytosolic lipid droplet lipolysis. Mutations in either the human CGI-58 or ATGL gene cause an autosomal recessive neutral lipid storage disease, characterized by the excessive accumulation of triglyceride (TAG)-rich lipid droplets in the cytoplasm of almost all cell types. CGI-58, however, has ATGL-independent functions. Distinct phenotypes associated with CGI-58 deficiency commonly include ichthyosis (scaly dry skin), nonalcoholic steatohepatitis, and hepatic fibrosis. Through regulated interactions with multiple protein families, CGI-58 controls many metabolic and signaling pathways, such as lipid and glucose metabolism, energy balance, insulin signaling, inflammatory responses, and thermogenesis. Recent studies have shown that CGI-58 regulates the pathogenesis of common metabolic diseases in a tissue-specific manner. Future studies are needed to molecularly define ATGL-independent functions of CGI-58, including the newly identified serine protease activity of CGI-58. Elucidation of these versatile functions of CGI-58 may uncover fundamental cellular processes governing lipid and energy homeostasis, which may help develop novel approaches that counter against obesity and its associated metabolic sequelae.

Identifiants

pubmed: 32705602
doi: 10.1007/978-981-15-6082-8_13
pmc: PMC8063591
mid: NIHMS1692474
doi:

Substances chimiques

1-Acylglycerol-3-Phosphate O-Acyltransferase EC 2.3.1.51
ABHD5 protein, human EC 2.3.1.51
Lipase EC 3.1.1.3
PNPLA2 protein, human EC 3.1.1.3

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

197-222

Subventions

Organisme : NIDDK NIH HHS
ID : R01 DK111052
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK116496
Pays : United States

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Auteurs

Liqing Yu (L)

Division of Endocrinology, Diabetes and Nutrition, Department of Medicine, University of Maryland School of Medicine, Baltimore, MD, USA. Lyu@som.umaryland.edu.

Yi Li (Y)

Division of Endocrinology, Diabetes and Nutrition, Department of Medicine, University of Maryland School of Medicine, Baltimore, MD, USA.

Alison Grisé (A)

College of Computer, Math, and Natural Sciences, College of Behavioral and Social Sciences, University of Maryland, College Park, MD, USA.

Huan Wang (H)

Division of Endocrinology, Diabetes and Nutrition, Department of Medicine, University of Maryland School of Medicine, Baltimore, MD, USA.

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