H9N2 swine influenza virus infection-induced damage is mediated by TRPM2 channels in mouse pulmonary microvascular endothelial cells.
Apoptosis
Cytokines
H9N2 swine Influenza virus
Mouse pulmonary microvascular endothelial cells
Oxidative stress
TRPM2
Journal
Microbial pathogenesis
ISSN: 1096-1208
Titre abrégé: Microb Pathog
Pays: England
ID NLM: 8606191
Informations de publication
Date de publication:
Nov 2020
Nov 2020
Historique:
received:
27
05
2020
revised:
15
07
2020
accepted:
17
07
2020
pubmed:
25
7
2020
medline:
16
6
2021
entrez:
25
7
2020
Statut:
ppublish
Résumé
Oxidative stress is implicated in the pathogenesis of influenza virus infection. Increasing evidences show that transient receptor potential melastatin 2 (TRPM2), a Ca
Identifiants
pubmed: 32707310
pii: S0882-4010(20)30774-9
doi: 10.1016/j.micpath.2020.104408
pii:
doi:
Substances chimiques
Reactive Oxygen Species
0
TRPM Cation Channels
0
TRPM2 protein, mouse
0
Calcium
SY7Q814VUP
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
104408Informations de copyright
Copyright © 2020. Published by Elsevier Ltd.