Modeling human age-associated increase in Gadd45γ expression leads to spatial recognition memory impairments in young adult mice.


Journal

Neurobiology of aging
ISSN: 1558-1497
Titre abrégé: Neurobiol Aging
Pays: United States
ID NLM: 8100437

Informations de publication

Date de publication:
10 2020
Historique:
received: 12 01 2020
revised: 22 05 2020
accepted: 27 06 2020
pubmed: 28 7 2020
medline: 30 6 2021
entrez: 26 7 2020
Statut: ppublish

Résumé

Aging is associated with the progressive decay of cognitive function. Hippocampus-dependent processes, such as the formation of spatial memory, are particularly vulnerable to aging. Currently, the molecular mechanisms responsible for age-dependent cognitive decline are largely unknown. Here, we investigated the expression and function of the growth arrest DNA damage gamma (Gadd45γ) during aging and cognition. We report that Gadd45γ expression is increased in the hippocampus of aged humans and that Gadd45γ overexpression in the young adult mouse hippocampus compromises cognition. Moreover, Gadd45γ overexpression in hippocampal neurons disrupted cAMP response element-binding protein signaling and the expression of well-established activity-regulated genes. This work shows that Gadd45γ expression is tightly controlled in the hippocampus and its disruption may be a mechanism contributing to age-related cognitive impairments observed in humans.

Identifiants

pubmed: 32711258
pii: S0197-4580(20)30214-1
doi: 10.1016/j.neurobiolaging.2020.06.021
pii:
doi:

Substances chimiques

Cyclic AMP Response Element-Binding Protein 0
Intracellular Signaling Peptides and Proteins 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

281-286

Informations de copyright

Copyright © 2020 Elsevier Inc. All rights reserved.

Auteurs

David V C Brito (DVC)

Department of Neurobiology, Interdisciplinary Centre for Neurosciences (IZN), Heidelberg University, Heidelberg, Germany.

Kubra Gulmez Karaca (K)

Department of Neurobiology, Interdisciplinary Centre for Neurosciences (IZN), Heidelberg University, Heidelberg, Germany; Department of Cognitive Neuroscience, Donders Institute for Brain, Cognition and Behaviour, Radboud University Medical Center, Nijmegen, the Netherlands.

Janina Kupke (J)

Department of Neurobiology, Interdisciplinary Centre for Neurosciences (IZN), Heidelberg University, Heidelberg, Germany.

Franziska Mudlaff (F)

Department of Neurobiology, Interdisciplinary Centre for Neurosciences (IZN), Heidelberg University, Heidelberg, Germany.

Benjamin Zeuch (B)

Department of Neurobiology, Interdisciplinary Centre for Neurosciences (IZN), Heidelberg University, Heidelberg, Germany; Directors' Research, European Molecular Biology Laboratory (EMBL), Heidelberg, Germany.

Rui Gomes (R)

Instituto de Medicina Molecular João Lobo Antunes, Faculdade de Medicina de Lisboa, Universidade de Lisboa, Lisbon, Portugal.

Luísa V Lopes (LV)

Instituto de Medicina Molecular João Lobo Antunes, Faculdade de Medicina de Lisboa, Universidade de Lisboa, Lisbon, Portugal.

Ana M M Oliveira (AMM)

Department of Neurobiology, Interdisciplinary Centre for Neurosciences (IZN), Heidelberg University, Heidelberg, Germany. Electronic address: oliveira@nbio.uni-heidelberg.de.

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Classifications MeSH