Clinical Correlations of Programmed Cell Death Ligand 1 Status in Liquid and Standard Biopsies in Breast Cancer.


Journal

Clinical chemistry
ISSN: 1530-8561
Titre abrégé: Clin Chem
Pays: England
ID NLM: 9421549

Informations de publication

Date de publication:
01 08 2020
Historique:
received: 31 08 2019
accepted: 01 05 2020
pubmed: 28 7 2020
medline: 30 3 2021
entrez: 27 7 2020
Statut: ppublish

Résumé

Data regarding the prognostic value of programmed cell death ligand 1 (PD-L1) expression on circulating tumor cells (CTCs) are lacking. However, CTCs could represent an alternative approach to serial biopsies, allowing real-time monitoring of cancer phenotype. We evaluated, in a dedicated prospective clinical trial, the clinicopathological correlations and prognostic value of PD-L1(+)-CTCs in 72 patients with metastatic breast cancer (MBC). Eighteen of 56 patients with available archival tissue presented at least one positive (≥1%) PD-L1 tumor sample. Baseline CTCs and PD-L1(+)-CTCs were detected in 57 (79.2%) and 26 (36.1%) patients. No significant correlation was found between PD-L1 tumors and CTC expression. In univariate analysis, triple negative (TN) phenotype, number of metastatic treatments, >2 metastatic sites, ≥5 CTCs and PD-L1(+)-CTCs were significantly associated with progression-free survival, while tissue PD-L1 expression was not. In multivariate analysis, TN phenotype, number of metastatic treatments and of metastatic sites were the only 3 variables independently associated with progression-free survival. Progesterone receptor negativity, TN phenotype, >2 metastatic sites and ≥5 CTCs were significantly associated with overall survival in univariate analysis. In multivariable analysis, TN phenotype and >2 metastatic sites were the only 2 independent variables. Unlike PD-L1(+)-tumor, PD-L1(+)-CTCs correlate to survival in MBC. Reappraisal of the role of PD-L1 expression by tumor tissue and by CTCs under anti-PD-1/PD-L1 treatment is necessary to evaluate its predictive value and potential role as a stratifying factor in strategies and trials for MBC patients with MBC. NCT02866149.

Sections du résumé

BACKGROUND
Data regarding the prognostic value of programmed cell death ligand 1 (PD-L1) expression on circulating tumor cells (CTCs) are lacking. However, CTCs could represent an alternative approach to serial biopsies, allowing real-time monitoring of cancer phenotype.
METHODS
We evaluated, in a dedicated prospective clinical trial, the clinicopathological correlations and prognostic value of PD-L1(+)-CTCs in 72 patients with metastatic breast cancer (MBC).
RESULTS
Eighteen of 56 patients with available archival tissue presented at least one positive (≥1%) PD-L1 tumor sample. Baseline CTCs and PD-L1(+)-CTCs were detected in 57 (79.2%) and 26 (36.1%) patients. No significant correlation was found between PD-L1 tumors and CTC expression. In univariate analysis, triple negative (TN) phenotype, number of metastatic treatments, >2 metastatic sites, ≥5 CTCs and PD-L1(+)-CTCs were significantly associated with progression-free survival, while tissue PD-L1 expression was not. In multivariate analysis, TN phenotype, number of metastatic treatments and of metastatic sites were the only 3 variables independently associated with progression-free survival. Progesterone receptor negativity, TN phenotype, >2 metastatic sites and ≥5 CTCs were significantly associated with overall survival in univariate analysis. In multivariable analysis, TN phenotype and >2 metastatic sites were the only 2 independent variables.
CONCLUSIONS
Unlike PD-L1(+)-tumor, PD-L1(+)-CTCs correlate to survival in MBC. Reappraisal of the role of PD-L1 expression by tumor tissue and by CTCs under anti-PD-1/PD-L1 treatment is necessary to evaluate its predictive value and potential role as a stratifying factor in strategies and trials for MBC patients with MBC.
CLINICAL TRIAL REGISTRATION
NCT02866149.

Identifiants

pubmed: 32712650
pii: 5876554
doi: 10.1093/clinchem/hvaa121
doi:

Substances chimiques

B7-H1 Antigen 0
Biomarkers, Tumor 0
CD274 protein, human 0

Banques de données

ClinicalTrials.gov
['NCT02866149']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1093-1101

Informations de copyright

© American Association for Clinical Chemistry 2020. All rights reserved. For permissions, please email: journals.permissions@oup.com.

Auteurs

William Jacot (W)

Department of Medical Oncology, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.
Institut de Recherche en Cancérologie de Montpellier (IRCM), INSERM U1194, Montpellier University, Montpellier, France.

Martine Mazel (M)

Laboratory of Rare Human Circulating Cells (LCCRH), University Medical Centre of Montpellier, Montpellier University EA2415, Montpellier, France.

Caroline Mollevi (C)

Institut de Recherche en Cancérologie de Montpellier (IRCM), INSERM U1194, Montpellier University, Montpellier, France.
Biometrics Unit, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.

Stéphane Pouderoux (S)

Department of Medical Oncology, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.

Véronique D'Hondt (V)

Department of Medical Oncology, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.
Institut de Recherche en Cancérologie de Montpellier (IRCM), INSERM U1194, Montpellier University, Montpellier, France.

Laure Cayrefourcq (L)

Laboratory of Rare Human Circulating Cells (LCCRH), University Medical Centre of Montpellier, Montpellier University EA2415, Montpellier, France.

Céline Bourgier (C)

Department of Radiation Oncology, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.

Florence Boissiere-Michot (F)

Translational Research Unit, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.

Fella Berrabah (F)

Clinical Research Center, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.

Evelyne Lopez-Crapez (E)

Translational Research Unit, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.

François-Clément Bidard (FC)

Department of Medical Oncology, Institut Curie, PSL Research University, Paris, France.
Versailles Saint Quentin en Yvelines University, Paris, Saclay University, Saint Cloud, Paris, France.
Circulating Tumor Biomarkers Laboratory, Institut Curie, PSL Research University, Paris, France.

Marie Viala (M)

Department of Medical Oncology, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.

Thierry Maudelonde (T)

Laboratory of Rare Human Circulating Cells (LCCRH), University Medical Centre of Montpellier, Montpellier University EA2415, Montpellier, France.

Séverine Guiu (S)

Department of Medical Oncology, Institut du Cancer Montpellier (ICM), Montpellier University, Montpellier, France.
Institut de Recherche en Cancérologie de Montpellier (IRCM), INSERM U1194, Montpellier University, Montpellier, France.

Catherine Alix-Panabières (C)

Laboratory of Rare Human Circulating Cells (LCCRH), University Medical Centre of Montpellier, Montpellier University EA2415, Montpellier, France.

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Classifications MeSH