Active Lysogeny in Listeria Monocytogenes Is a Bacteria-Phage Adaptive Response in the Mammalian Environment.


Journal

Cell reports
ISSN: 2211-1247
Titre abrégé: Cell Rep
Pays: United States
ID NLM: 101573691

Informations de publication

Date de publication:
28 07 2020
Historique:
received: 07 02 2020
revised: 05 05 2020
accepted: 02 07 2020
entrez: 30 7 2020
pubmed: 30 7 2020
medline: 11 5 2021
Statut: ppublish

Résumé

Some Listeria monocytogenes (Lm) strains harbor a prophage within the comK gene, which renders it inactive. During Lm infection of macrophage cells, the prophage turns into a molecular switch, promoting comK gene expression and therefore Lm intracellular growth. During this process, the prophage does not produce infective phages or cause bacterial lysis, suggesting it has acquired an adaptive behavior suited to the pathogenic lifestyle of its host. In this study, we demonstrate that this non-classical phage behavior, named active lysogeny, relies on a transcriptional response that is specific to the intracellular niche. While the prophage undergoes lytic induction, the process is arrested midway, preventing the transcription of the late genes. Further, we demonstrate key phage factors, such as LlgA transcription regulator and a DNA replicase, that support the phage adaptive behavior. This study provides molecular insights into the adaptation of phages to their pathogenic hosts, uncovering unusual cooperative interactions.

Identifiants

pubmed: 32726621
pii: S2211-1247(20)30937-2
doi: 10.1016/j.celrep.2020.107956
pmc: PMC7397523
pii:
doi:

Substances chimiques

Bacterial Proteins 0
ComK protein, Listeria monocytogenes 0
Transcription Factors 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

107956

Informations de copyright

Copyright © 2020 The Author(s). Published by Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Interests The authors declare no competing interests.

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Auteurs

Anna Pasechnek (A)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel.

Lev Rabinovich (L)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel.

Olga Stadnyuk (O)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel.

Gil Azulay (G)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel.

Jessica Mioduser (J)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel.

Tal Argov (T)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel.

Ilya Borovok (I)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel.

Nadejda Sigal (N)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel.

Anat A Herskovits (AA)

The School of Molecular Cell Biology and Biotechnology, Faculty of Life Sciences, Tel Aviv University, Ramat Aviv, Tel- Aviv 69978, Israel. Electronic address: anathe@tauex.tau.ac.il.

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