Estrogen shields women from COVID-19 complications by reducing ER stress.


Journal

Medical hypotheses
ISSN: 1532-2777
Titre abrégé: Med Hypotheses
Pays: United States
ID NLM: 7505668

Informations de publication

Date de publication:
Oct 2020
Historique:
received: 02 06 2020
revised: 21 06 2020
accepted: 27 07 2020
pubmed: 8 8 2020
medline: 21 10 2020
entrez: 8 8 2020
Statut: ppublish

Résumé

Estrogen hormone acts as a potential key player in providing immunity against certain viral infection. It is found to be associated in providing immunity against acute lungs inflammation and influenza virus by modulating cytokines storm and mediating adaptive immune alterations respectively. Women are less affected by SARS-CoV-2 infection because of the possible influence of estrogen hormone as compared to men. We hypothesized that SARS-CoV-2 causes stress in endoplasmic reticulum (ER) which in turn aggravates the infection, estrogen hormone might play key role in decreasing ER stress by activating estrogen mediated signaling pathways, results in unfolded protein response (UPR). Estrogen governs degradation of phosphotidylinositol 4,5-bisphosphate (PIP

Identifiants

pubmed: 32759016
pii: S0306-9877(20)31612-1
doi: 10.1016/j.mehy.2020.110148
pmc: PMC7390780
pii:
doi:

Substances chimiques

Diglycerides 0
Estrogens 0
Phosphatidylinositol 4,5-Diphosphate 0
Viral Proteins 0
Inositol 1,4,5-Trisphosphate 85166-31-0
Type C Phospholipases EC 3.1.4.-

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

110148

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Auteurs

Sana Shabbir (S)

COMSATS University Islamabad, Pakistan.

Assad Hafeez (A)

Health Services Academy Islamabad, Pakistan.

Muhammad Arshad Rafiq (MA)

COMSATS University Islamabad, Pakistan.

Muhammad Jawad Khan (MJ)

COMSATS University Islamabad, Pakistan. Electronic address: jawadkhan@comsats.edu.pk.

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Classifications MeSH