Heart Failure With Reduced Ejection Fraction Is Characterized by Systemic NEP Downregulation.

ANP, atrial natriuretic peptide ARNI ARNI, angiotensin-receptor neprilysin-inhibitor BNP, B-type natriuretic peptide CMRI+LE, cardiac magnetic resonance and late enhancement HF, heart failure HFrEF, heart failure with reduced ejection fraction LV, left ventricular NEP, neprilysin NT-proBNP, N-terminal pro-B-type natriuretic peptide Q1 to Q3, 25th to 75th percentile RA, right atrial RV, right ventricular biomarker gene expression left atrial, left atrial mRNA, messenger RNA metalloproteinase neprilysin qPCR, real-time polymerase chain reaction translational model of heart failure

Journal

JACC. Basic to translational science
ISSN: 2452-302X
Titre abrégé: JACC Basic Transl Sci
Pays: United States
ID NLM: 101677259

Informations de publication

Date de publication:
Jul 2020
Historique:
received: 30 09 2019
revised: 08 05 2020
accepted: 08 05 2020
entrez: 8 8 2020
pubmed: 8 8 2020
medline: 8 8 2020
Statut: epublish

Résumé

Based on the investigation of neprilysin (NEP) regulation in a translational porcine model of chronic heart failure (HF), this study concluded: 1) that kidneys might play a crucial part in systemic NEP regulation based on 20 to 100 higher NEP content and/or activity compared with any other organ; 2) NEP seems to be downregulated under HF conditions; and 3) that the value of plasma NEP concentrations and activity as biomarkers is questionable. For the first time, these data provide basic knowledge on HF-related pathophysiological alterations of the NEP system and contribute to understanding the mechanism of action of angiotensin-receptor neprilysin-inhibitors, which remains elusive despite broad clinical applications.

Identifiants

pubmed: 32760858
doi: 10.1016/j.jacbts.2020.05.011
pii: S2452-302X(20)30253-9
pmc: PMC7393434
doi:

Types de publication

Journal Article

Langues

eng

Pagination

715-726

Informations de copyright

© 2020 The Authors.

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Auteurs

Imre J Pavo (IJ)

Department of Pediatrics, Medical University of Vienna, Vienna, Austria.

Noemi Pavo (N)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Nina Kastner (N)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Denise Traxler (D)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Dominika Lukovic (D)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Katrin Zlabinger (K)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Andreas Spannbauer (A)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Martin Riesenhuber (M)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

David Lorant (D)

Department of Anesthesiology, Medical University of Vienna, Vienna, Austria.

Philipp E Bartko (PE)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Georg Goliasch (G)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Martin Hülsmann (M)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Johannes Winkler (J)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Mariann Gyöngyösi (M)

Department of Internal Medicine II, Division of Cardiology, Medical University of Vienna, Vienna, Austria.

Classifications MeSH