NADPH oxidase DUOX1 sustains TGF-β1 signalling and promotes lung fibrosis.


Journal

The European respiratory journal
ISSN: 1399-3003
Titre abrégé: Eur Respir J
Pays: England
ID NLM: 8803460

Informations de publication

Date de publication:
01 2021
Historique:
received: 03 10 2019
accepted: 22 07 2020
pubmed: 9 8 2020
medline: 3 7 2021
entrez: 9 8 2020
Statut: epublish

Résumé

Interstitial lung fibroblast activation coupled with extracellular matrix production is a pathological signature of pulmonary fibrosis, and is governed by transforming growth factor (TGF)-β1/Smad signalling. TGF-β1 and oxidative stress cooperate to drive fibrosis. Cells can produce reactive oxygen species through activation and/or induction of NADPH oxidases, such as dual oxidase (DUOX1/2). Since DUOX enzymes, as extracellular hydrogen peroxide (H

Identifiants

pubmed: 32764116
pii: 13993003.01949-2019
doi: 10.1183/13993003.01949-2019
pii:
doi:

Substances chimiques

Transforming Growth Factor beta1 0
Hydrogen Peroxide BBX060AN9V
Dual Oxidases EC 1.11.1.-
NADPH Oxidases EC 1.6.3.-
DUOX1 protein, human EC 1.6.3.1
Duox1 protein, mouse EC 1.6.3.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright ©ERS 2021.

Déclaration de conflit d'intérêts

Conflict of interest: R.A. Louzada has nothing to disclose. Conflict of interest: R. Corre has nothing to disclose. Conflict of interest: R. Ameziane El Hassani has nothing to disclose. Conflict of interest: L. Meziani has nothing to disclose. Conflict of interest: M. Jaillet has nothing to disclose. Conflict of interest: A. Cazes has nothing to disclose. Conflict of interest: B. Crestani reports personal fees for lectures from AstraZeneca, grants, personal fees for advisory board work and non-financial support for meeting attendance from Boehringer Ingelheim and Roche, personal fees for advisory board work and non-financial support for meeting attendance from BMS, personal fees for advisory board work from Sanofi, outside the submitted work. Conflict of interest: E. Deutsch reports grants and personal fees from Roche Genentech, AstraZeneca/Medimmune, Merck Serono and Boehringer, grants from Servier, BMS, MSD and Amazon AWS, personal fees from Amgen and Accuray, outside the submitted work. Conflict of interest: C. Dupuy has nothing to disclose.

Auteurs

Ruy Andrade Louzada (RA)

CNRS UMR 8200, Université Paris-Saclay, Gustave Roussy, Villejuif, France.

Raphaël Corre (R)

CNRS UMR 8200, Université Paris-Saclay, Gustave Roussy, Villejuif, France.

Rabii Ameziane El Hassani (R)

Laboratoire de Biologie des Pathologies Humaines, Université Mohammed V, Faculté des Sciences, Rabat, Morocco.

Lydia Meziani (L)

Inserm U1030, Université Paris-Saclay, Gustave Roussy, Villejuif, France.

Madeleine Jaillet (M)

INSERM U1152, Paris, France.

Aurélie Cazes (A)

Département de Pathologie, Hôpital Bichat, Paris, France.

Bruno Crestani (B)

INSERM U1152, Paris, France.
Université Paris-Diderot, LABEX INFLAMEX, Paris, France.
Assistance Publique-Hôpitaux de Paris, DHU FIRE, Hôpital Bichat, Paris, France.

Eric Deutsch (E)

Inserm U1030, Université Paris-Saclay, Gustave Roussy, Villejuif, France.

Corinne Dupuy (C)

CNRS UMR 8200, Université Paris-Saclay, Gustave Roussy, Villejuif, France corinne.dupuy@gustaveroussy.fr.

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Classifications MeSH